Ganciclovir-mediated in vivo elimination of myeloid leukemic cells expressing the HSVtk gene induces HSVtk loss variants. - Wikidata - wikimedia - TriplyDB

Ganciclovir-mediated in vivo elimination of myeloid leukemic cells expressing the HSVtk gene induces HSVtk loss variants.

Ganciclovir-mediated in vivo elimination of myeloid leukemic cells expressing the HSVtk gene induces HSVtk loss variants.

http://www.wikidata.org/entity/Q33178788

about

Suicide gene therapy: possible applications in haematopoietic disorders.Selection of HSV-1 TK gene-transfected murine mammary carcinoma cells resistant to (E)-5-(2-bromovinyl)-2'-deoxyuridine (BVDU) and ganciclovir (GCV).

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Ganciclovir-mediated in vivo elimination of myeloid leukemic cells expressing the HSVtk gene induces HSVtk loss variants.

http://www.wikidata.org/entity/Q33178788

description

1999 nî lūn-bûn

@nan

1999 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի հունիսին հրատարակված գիտական հոդված

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1999年の論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年论文

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name

Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

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Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

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type

label

Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

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Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

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prefLabel

Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

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Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

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Ganciclovir-mediated in vivo e ...... e induces HSVtk loss variants.

@en

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Braakman E

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Bron M

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Hagenbeek A

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Hoogerbrugge PM

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Martens AC

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Valerio D

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Vermeulen J

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Vogels R

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P2860

Bystander killing of cancer cells by herpes simplex virus thymidine kinase gene is mediated by connexins

The "bystander effect": association of U-87 cell death with ganciclovir-mediated apoptosis of nearby cells and lack of effect in athymic mice.

Vaccination with irradiated tumor cells engineered to secrete murine granulocyte-macrophage colony-stimulating factor stimulates potent, specific, and long-lasting anti-tumor immunity.

In vitro evidence that metabolic cooperation is responsible for the bystander effect observed with HSV tk retroviral gene therapy.

Metabolism of 5-fluorocytosine to 5-fluorouracil in human colorectal tumor cells transduced with the cytosine deaminase gene: significant antitumor effects when only a small percentage of tumor cells express cytosine deaminase.

Transfer of the bacterial gene for cytosine deaminase to mammalian cells confers lethal sensitivity to 5-fluorocytosine: a negative selection system.

Long-term expression of human adenosine deaminase in rhesus monkeys transplanted with retrovirus-infected bone-marrow cells.

Experimental and clinical studies of cytokine gene-modified tumor cells.

Bystander effects of different enzyme-prodrug systems for cancer gene therapy depend on different pathways for intercellular transfer of toxic metabolites, a factor that will govern clinical choice of appropriate regimes.

Killing Epstein-Barr virus-positive B lymphocytes by gene therapy: comparing the efficacy of cytosine deaminase and herpes simplex virus thymidine kinase.

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1139-1146

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scholarly article

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10.1038/SJ.GT.3300913

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6

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6

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1999-06-01T00:00:00Z

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12845303

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10455417

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