Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation - Wikidata - wikimedia - TriplyDB

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

http://www.wikidata.org/entity/Q33250260

about

Molecular mechanisms of inflammation and tissue injury after major trauma--is complement the "bad guy"?B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.Membrane attack complex inhibitor CD59a protects against focal cerebral ischemia in mice Experimental traumatic brain injury The alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injury Inhibition of the alternative complement activation pathway in traumatic brain injury by a monoclonal anti-factor B antibody: a randomized placebo-controlled study in mice.Challenging the role of adaptive immunity in neurotrauma: Rag1(-/-) mice lacking mature B and T cells do not show neuroprotection after closed head injury.Guilty molecules, guilty minds? The conflicting roles of the innate immune response to traumatic brain injury Alteration of complement hemolytic activity in different trauma and sepsis models.An anticomplement agent that homes to the damaged brain and promotes recovery after traumatic brain injury in mice The alternative complement pathway propagates inflammation and injury in murine ischemic stroke Osteoblast-specific overexpression of complement receptor C5aR1 impairs fracture healing.Therapeutic inhibition of the alternative complement pathway attenuates chronic EAE.Complement activation at the motor end-plates in amyotrophic lateral sclerosis.Functions of the complement components C3 and C5 during sepsis Complement-Mediated Regulation of Metabolism and Basic Cellular Processes Protective effects of decay-accelerating factor on blast-induced neurotrauma in rats.Complement activation in the injured central nervous system: another dual-edged sword?Role of complement in multiorgan failure.Targeted modulation of the neuroinflammatory response after spinal cord injury: the ongoing quest for the "holy grail".Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice.Gene expression profiling of aging in multiple mouse strains: identification of aging biomarkers and impact of dietary antioxidants.The role of the complement system in traumatic brain injury: a review.Neuroimmunology of Traumatic Brain Injury: Time for a Paradigm Shift.Is plasma C3 and C4 levels useful in young cerebral ischemic stroke patients? Associations with prognosis at 3 months.Identifying the role of complement in triggering neuroinflammation after traumatic brain injury.

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P2860

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

http://www.wikidata.org/entity/Q33250260

description

2006 nî lūn-bûn

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2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2006 թվականի հուլիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Reduced neuronal cell death af ...... thway of complement activation

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Reduced neuronal cell death af ...... thway of complement activation

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Reduced neuronal cell death af ...... thway of complement activation

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BMC Neuroscience

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Reduced neuronal cell death af ...... thway of complement activation

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Denise Harhausen

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Iris Leinhase

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Joshua M Thurman

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Malte Pietzcker

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Mohy E Taha

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Oliver I Schmidt

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Philip F Stahel

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V Michael Holers

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Wade R Smith

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P2860

Bench-to-bedside review: Apoptosis/programmed cell death triggered by traumatic brain injury.

TNF-alpha-mediated expression of the receptor for anaphylatoxin C5a on neurons in experimental Listeria meningoencephalitis

Abrogation of the alternative complement pathway by targeted deletion of murine factor B

Progesterone differentially regulates pro- and anti-apoptotic gene expression in cerebral cortex following traumatic brain injury in rats

Complement C3 and C5 play critical roles in traumatic brain cryoinjury: blocking effects on neutrophil extravasation by C5a receptor antagonist.

Neuronal expression of a functional receptor for the C5a complement activation fragment.

Regulation of the complement membrane attack pathway.

Complement anaphylatoxin receptors on neurons: new tricks for old receptors?

Inhibition of complement as a therapeutic approach in inflammatory central nervous system (CNS) disease

Gender differences in acute CNS trauma and stroke: neuroprotective effects of estrogen and progesterone.

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10.1186/1471-2202-7-55

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Denise Harhausen

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6942373

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