Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice. - Wikidata - wikimedia - TriplyDB

Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice.

Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice.

http://www.wikidata.org/entity/Q41771843

about

Molecular mechanisms of inflammation and tissue injury after major trauma--is complement the "bad guy"?The far-reaching scope of neuroinflammation after traumatic brain injury.Deficiency of complement receptors CR2/CR1 in Cr2⁻/⁻ mice reduces the extent of secondary brain damage after closed head injury.Membrane attack complex inhibitor CD59a protects against focal cerebral ischemia in mice Microglia activation and anti-inflammatory regulation in Alzheimer's disease Longitudinally extensive NMO spinal cord pathology produced by passive transfer of NMO-IgG in mice lacking complement inhibitor CD59.The alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injury Versatility of the complement system in neuroinflammation, neurodegeneration and brain homeostasis A new experimental polytrauma model in rats: molecular characterization of the early inflammatory response.Challenging the role of adaptive immunity in neurotrauma: Rag1(-/-) mice lacking mature B and T cells do not show neuroprotection after closed head injury.Guilty molecules, guilty minds? The conflicting roles of the innate immune response to traumatic brain injury An anticomplement agent that homes to the damaged brain and promotes recovery after traumatic brain injury in mice The alternative complement pathway propagates inflammation and injury in murine ischemic stroke Deletion of the membrane complement inhibitor CD59a drives age and gender-dependent alterations to bone phenotype in mice.Complement activation at the motor end-plates in amyotrophic lateral sclerosis.Dysregulation of the complement cascade in the hSOD1G93A transgenic mouse model of amyotrophic lateral sclerosis.Complement in neuroprotection and neurodegeneration.Complement activation in the injured central nervous system: another dual-edged sword?Innate immunity and neuroinflammation.The role of complement in neurological and neuropsychiatric diseases.C5a/C5aR Pathway Plays a Vital Role in Brain Inflammatory Injury via Initiating Fgl-2 in Intracerebral Hemorrhage.Deficiency of complement component 5 ameliorates glaucoma in DBA/2J mice.The role of the complement system in traumatic brain injury: a review.Neuroimmunology of Traumatic Brain Injury: Time for a Paradigm Shift.Identifying the role of complement in triggering neuroinflammation after traumatic brain injury.

P2860

Q26825724-D656BDD3-44A9-4264-99C5-0F7201099A80 Q30235977-408DD621-DC7E-4ECB-9AC3-62C7CF0150E1 Q33729953-A3611023-9490-47CC-8C10-F1EF9C29572B Q33732180-E45499C5-636A-40D6-B25E-B45E6A6EF56B Q33943873-CBB1BF21-004B-40E5-A719-CCBD36ECF5F0 Q34173376-746720ED-27EB-4A79-9A36-D91585229664 Q34358170-D9852173-3146-4640-A2C1-4A61B332B0D4 Q34471200-788814BB-E345-4B8F-B67C-84A9642AC54E Q35864439-302ED629-52D7-4B57-A234-E8E6152611CC Q35886721-592AD574-9271-4EF7-9313-788EEAB5C1BF Q36026673-6A6F857E-1EFD-4F73-A228-5FF75547BE56 Q36306007-B50780A9-510B-499D-809D-BF9856CF35B0 Q36339354-BE62C14D-CD1E-4895-8482-D704D3E492D2 Q36610276-0717324E-CFDD-46DE-97A1-0BB24FFB07A5 Q36774693-B4BE32A2-12A9-4598-91AA-A8DFE37C184B Q37363769-88BA54F4-F137-4CF2-9B2E-B880816002AD Q37684123-8C241FD9-76E7-411F-A08F-0DEAB93D8DB6 Q38020570-9E35E2E0-93E7-4273-B3F8-75014B05135B Q38120744-D92A8E88-134C-47E8-8021-7B8E1A953739 Q38590250-B9CAA18C-C729-4149-8596-E1A9F90B3D5E Q39317676-5802682F-90B9-4067-AF64-9DCC8A904EAD Q43096209-B64677BC-71D3-48C8-AAB5-BE2C6624E0D9 Q47553098-59485135-9C6C-4429-8259-DEBE9148B304 Q48113339-553C8B1A-3AC9-415B-8625-CDBD325C24EA Q50043627-B67C7E6A-C32B-42D5-B83D-16D18DF30209

P2860

Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice.

http://www.wikidata.org/entity/Q41771843

description

2009 nî lūn-bûn

@nan

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

2009年论文

@zh

2009年论文

@zh-cn

name

Absence of the complement regu ...... raumatic brain injury in mice.

@en

type

label

Absence of the complement regu ...... raumatic brain injury in mice.

@en

prefLabel

Absence of the complement regu ...... raumatic brain injury in mice.

@en

P1433

Q41771843-2FCFA93C-0E76-48CE-BEA5-BBB224CC6ABB

P1476

Q41771843-D65DE266-74B8-42B7-8696-AAECD1ED59DA

P2093

Q41771843-0983AFB3-7628-4A84-8BF4-9A906C0A386C

Q41771843-198993DD-DBDF-4BF7-8F25-A4556A785CC3

Q41771843-2914CC56-9B9C-405C-8813-8CB0870DEC5F

Q41771843-454927DB-4AE9-457C-BCEF-1572EFE97BEA

Q41771843-95A76786-5152-42B4-BC57-480834FC06C1

Q41771843-9D536EC1-10A9-4A1B-A466-9CC7A648CDEB

Q41771843-BC04F36E-BA7B-41BD-9044-CE22AE1CED9C

Q41771843-D2E0A9AB-3DAD-40AB-9730-5B2C88E4B9F3

Q41771843-DD76DD23-2B6A-4F86-A244-B6BB4A01D923

Q41771843-E6B8B5BB-883E-4441-ADD8-99655FDA5C8E

P2860

Q41771843-0DEB6109-CBC1-4B45-8D84-3752B2467F83

Q41771843-10765EAD-DA62-4797-BA0A-D75E21D779FE

Q41771843-19A12B96-7562-45EF-B991-DDF5BD2493AC

Q41771843-2E64749F-0228-4158-9BE4-843E250373B5

Q41771843-30236105-051F-41DB-82D3-9AF8952DB165

Q41771843-3DFA0B8B-BB39-4655-B6C4-3747DDDD2CAA

Q41771843-3EA22575-618D-4947-AF47-5CEECA0CEEBE

Q41771843-4093B802-DBC1-49B6-ABC8-F1D59F5111FD

Q41771843-430264E6-892C-46B7-925C-2113CF8BE80F

Q41771843-494536DA-E6D7-460E-B55C-D4BD72CB8396

P2888

Q41771843-DFE384B3-177B-4415-83F3-3C1E73AA9BA0

P304

Q41771843-BE176E11-5B61-47EA-8922-C8286E111A26

P31

Q41771843-C1DF6D8E-EDD4-4EC9-8895-1CE3BB9CCE6A

P356

Q41771843-EE0CD1CB-6EB3-4C4C-951C-C946336C44C8

P478

Q41771843-15EDCC54-F554-41A6-912E-7CE39A9AA74D

P50

Q41771843-B347166B-2732-42FD-A9DB-13EA7F9E3F16

P577

Q41771843-132BC39B-EEB8-4F2B-9DF5-E0D20130620F

P5875

Q41771843-7D30C135-831D-4112-99FF-48A919E27DAD

P6179

Q41771843-85018E08-A11A-4254-97A5-7CDF6D437B1F

P698

Q41771843-13E82FD2-E7F7-450E-A68C-6966CB5EE1F7

P921

Q41771843-C89BF196-BAC1-4469-AA7F-0265D82F85D1

P932

Q41771843-FB414A2F-FDC1-45C4-BADE-EF61172A1CC8

P356

P1433

Journal of Neuroinflammation

P1476

Absence of the complement regu ...... traumatic brain injury in mice

@en

P2093

Basel M Touban

http://www.w3.org/2001/XMLSchema#string

Christiane Stoll

http://www.w3.org/2001/XMLSchema#string

Claudia Conrad

http://www.w3.org/2001/XMLSchema#string

Iris Leinhase

http://www.w3.org/2001/XMLSchema#string

Ivonne Persigehl

http://www.w3.org/2001/XMLSchema#string

Kathryn Beauchamp

http://www.w3.org/2001/XMLSchema#string

Michael A Flierl

http://www.w3.org/2001/XMLSchema#string

Oliver I Schmidt

http://www.w3.org/2001/XMLSchema#string

Philip F Stahel

http://www.w3.org/2001/XMLSchema#string

Wade R Smith

http://www.w3.org/2001/XMLSchema#string

P2860

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

Regulation of the complement membrane attack pathway.

Inhibition of complement as a therapeutic approach in inflammatory central nervous system (CNS) disease

Complement inhibitors: a resurgent concept in anti-inflammatory therapeutics.

CD59a deficiency exacerbates ischemia-reperfusion injury in mice.

Complement therapeutics; history and current progress.

The alternative pathway of complement in disease: opportunities for therapeutic targeting.

Inhibition of the alternative complement activation pathway in traumatic brain injury by a monoclonal anti-factor B antibody: a randomized placebo-controlled study in mice.

Spontaneous classical pathway activation and deficiency of membrane regulators render human neurons susceptible to complement lysis

Alternative roles for CD59.

P2888

P304

2

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1186/1742-2094-6-2

http://www.w3.org/2001/XMLSchema#string

P478

6

http://www.w3.org/2001/XMLSchema#string

P50

P577

2009-01-08T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

23767895

http://www.w3.org/2001/XMLSchema#string

P6179

1043393909

http://www.w3.org/2001/XMLSchema#string

P698

19133139

http://www.w3.org/2001/XMLSchema#string

P921

traumatic brain injury

P932

2631471

http://www.w3.org/2001/XMLSchema#string