Lack of mucosal immune reconstitution during prolonged treatment of acute and early HIV-1 infection. - Wikidata - wikimedia - TriplyDB

Lack of mucosal immune reconstitution during prolonged treatment of acute and early HIV-1 infection.

Lack of mucosal immune reconstitution during prolonged treatment of acute and early HIV-1 infection.

http://www.wikidata.org/entity/Q33265482

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Decreases in colonic and systemic inflammation in chronic HIV infection after IL-7 administration Should we treat acute HIV infection?Redefining the viral reservoirs that prevent HIV-1 eradication HIV therapeutic vaccines: moving towards a functional cure CD8 T cell persistence in treated HIV infection A Small Molecule, Which Competes with MAdCAM-1, Activates Integrin α4β7 and Fails to Prevent Mucosal Transmission of SHIV-SF162P3 The Expression of Functional Vpx during Pathogenic SIVmac Infections of Rhesus Macaques Suppresses SAMHD1 in CD4+ Memory T Cells Comparative Analysis of Immune Activation Markers of CD8(+) T Cells in Lymph Nodes of Different Origins in SIV-Infected Chinese Rhesus Macaques Treatment with IL-7 prevents the decline of circulating CD4+ T cells during the acute phase of SIV infection in rhesus macaques Perspectives for immunotherapy: which applications might achieve an HIV functional cure?Cross-sectional detection of acute HIV infection: timing of transmission, inflammation and antiretroviral therapy Recombinant Mycobacterium bovis BCG as an HIV vaccine vector.Preliminary in vivo efficacy studies of a recombinant rhesus anti-alpha(4)beta(7) monoclonal antibody.[The Competence Network for HIV/AIDS. Data, Samples, Facts].Impact of antiretroviral therapy on intestinal lymphoid tissues in HIV infection Resisting immune exhaustion in HIV-1 infection.Magnitude and complexity of rectal mucosa HIV-1-specific CD8+ T-cell responses during chronic infection reflect clinical status Systemic effects of inflammation on health during chronic HIV infection CD25 preselective anti-HIV vectors for improved HIV gene therapy.The detection and management of early HIV infection: a clinical and public health emergency.Impact of highly active antiretroviral therapy initiation on CD4(+) T-cell repopulation in duodenal and rectal mucosa.CCR6 ligands inhibit HIV by inducing APOBEC3G.Cycling of gut mucosal CD4+ T cells decreases after prolonged anti-retroviral therapy and is associated with plasma LPS levels.Human Beta Defensin 2 Selectively Inhibits HIV-1 in Highly Permissive CCR6⁺CD4⁺ T Cells.The virologic and immunologic effects of cyclosporine as an adjunct to antiretroviral therapy in patients treated during acute and early HIV-1 infection.Unraveling the relationship between microbial translocation and systemic immune activation in HIV infection Residual immune dysregulation syndrome in treated HIV infection.Planning for pre-exposure prophylaxis to prevent HIV transmission: challenges and opportunities.Reconstitution of CD4 T cells in bronchoalveolar lavage fluid after initiation of highly active antiretroviral therapy.Mucosal immunity in human and simian immunodeficiency lentivirus infections Immune and genetic gardening of the intestinal microbiome.The use of BLT humanized mice to investigate the immune reconstitution of the gastrointestinal tract.Persistence of genital tract T cell responses in HIV-infected women on highly active antiretroviral therapy.Attenuation of immune activation in an open-label clinical trial for HIV-AIDS using a polyherbal formulation.Loss and dysregulation of Th17 cells during HIV infection.Immune activation and HIV persistence: implications for curative approaches to HIV infection.Differences in HIV burden and immune activation within the gut of HIV-positive patients receiving suppressive antiretroviral therapy Reconstitution of intestinal CD4 and Th17 T cells in antiretroviral therapy suppressed HIV-infected subjects: implication for residual immune activation from the results of a clinical trial.Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers.HIV infection and the gastrointestinal immune system

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Lack of mucosal immune reconstitution during prolonged treatment of acute and early HIV-1 infection.

http://www.wikidata.org/entity/Q33265482

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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type

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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Lack of mucosal immune reconst ...... ute and early HIV-1 infection.

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Andrea Low

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Daniel Boden

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Hiroshi Mohri

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Klara Tenner-Racz

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Martin Markowitz

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Patrick Jean-Pierre

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Paul Racz

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Saurabh Mehandru

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Victoria Manuelli

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P2860

CD4+ T cell depletion during all stages of HIV disease occurs predominantly in the gastrointestinal tract

Primary HIV-1 infection is associated with preferential depletion of CD4+ T lymphocytes from effector sites in the gastrointestinal tract

IL-15 induces CD4 effector memory T cell production and tissue emigration in nonhuman primates

Two subsets of memory T lymphocytes with distinct homing potentials and effector functions

Declining morbidity and mortality among patients with advanced human immunodeficiency virus infection. HIV Outpatient Study Investigators

Rapid turnover of plasma virions and CD4 lymphocytes in HIV-1 infection

Massive infection and loss of memory CD4+ T cells in multiple tissues during acute SIV infection

Lymphocyte homing and homeostasis

Selective generation of gut tropic T cells in gut-associated lymphoid tissue (GALT): requirement for GALT dendritic cells and adjuvant.

Activated memory CD4(+) T helper cells repopulate the intestine early following antiretroviral therapy of simian immunodeficiency virus-infected rhesus macaques but exhibit a decreased potential to produce interleukin-2

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