Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers. - Wikidata - wikimedia - TriplyDB

Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers.

Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers.

http://www.wikidata.org/entity/Q34598849

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Interferon-alpha, immune activation and immune dysfunction in treated HIV infection CD8 T cell persistence in treated HIV infection Does EBV alter the pathogenesis of malaria?Immune Activation Influences SAMHD1 Expression and Vpx-mediated SAMHD1 Degradation during Chronic HIV-1 Infection Recent Insights into the HIV/AIDS Pandemic.Murine Cytomegalovirus Disrupts Splenic Dendritic Cell Subsets via Type I Interferon-Dependent and -Independent Mechanisms.The dynamic changes of interferon lambdas related genes and proteins in JAK/STAT pathway in both acute and chronic HIV-1 infected patients.Type I interferon in the pathogenesis of lupus.Innate and adaptive immune responses in male and female reproductive tracts in homeostasis and following HIV infection.Targeting of type I interferon in systemic autoimmune diseases Interferon signature in the blood in inflammatory common variable immune deficiency Blockade of interferon Beta, but not interferon alpha, signaling controls persistent viral infection Interferon-α inhibits CD4 T cell responses to interleukin-7 and interleukin-2 and selectively interferes with Akt signaling.A Computational Model of Inhibition of HIV-1 by Interferon-Alpha Inflammation Perturbs the IL-7 Axis, Promoting Senescence and Exhaustion that Broadly Characterize Immune Failure in Treated HIV Infection.Sex differences in HIV-1-mediated immunopathology.Networking at the level of host immunity: immune cell interactions during persistent viral infections Interferons and HIV Infection: The Good, the Bad, and the Ugly Immune activation and collateral damage in AIDS pathogenesis.APOBEC3 proteins can copackage and comutate HIV-1 genomes.Applications and limitations of inflammatory biomarkers for studies on neurocognitive impairment in HIV infection.Blocking type I interferon signaling enhances T cell recovery and reduces HIV-1 reservoirs.Effects of alpha interferon treatment on intrinsic anti-HIV-1 immunity in vivo.The Envelope Gene of Transmitted HIV-1 Resists a Late Interferon Gamma-Induced Block.Influence of common mucosal co-factors on HIV infection in the female genital tract.Advances in understanding the role of type I interferons in systemic lupus erythematosus.Cerebrospinal fluid interferon alpha levels correlate with neurocognitive impairment in ambulatory HIV-Infected individuals.Cellular RNA Helicase DDX1 Is Involved in Transmissible Gastroenteritis Virus nsp14-Induced Interferon-Beta Production Type I interferon responses are impaired in latently HIV infected cells.HIV-TB co-infection: mechanisms that drive reactivation of Mycobacterium tuberculosis in HIV infection.Current views on interferon therapy for HIV.Transcriptomic meta-analysis identifies gene expression characteristics in various samples of HIV-infected patients with nonprogressive disease.Type I interferons suppress viral replication but contribute to T cell depletion and dysfunction during chronic HIV-1 infection.The interferon paradox: can inhibiting an antiviral mechanism advance an HIV cure?Gene therapy with plasmids encoding IFN-β or IFN-α14 confers long-term resistance to HIV-1 in humanized mice.Responsiveness to IL-7 but not to IFN-α is diminished in CD4+ T cells from treated HIV infected patients who experience poor CD4+ T-cell recovery Interferon Alpha Subtype-Specific Suppression of HIV-1 Infection In Vivo.High immune activation and abnormal expression of cytokines contribute to death of SHIV89.6-infected Chinese rhesus macaques.HIV-1 single-stranded RNA induces CXCL13 secretion in human monocytes via TLR7 activation and plasmacytoid dendritic cell-derived type I IFN.Plasma Cytokine Predictors of Tuberculosis Recurrence in Antiretroviral-Treated Human Immunodeficiency Virus-infected Individuals from Durban, South Africa.

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Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers.

http://www.wikidata.org/entity/Q34598849

description

2013 nî lūn-bûn

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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի փետրվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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name

Interferon-α is the primary pl ...... ctivation and disease markers.

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Interferon-α is the primary pl ...... ctivation and disease markers.

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Interferon-α is the primary pl ...... ctivation and disease markers.

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Interferon-α is the primary pl ...... ctivation and disease markers.

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Interferon-α is the primary pl ...... ctivation and disease markers.

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Interferon-α is the primary pl ...... ctivation and disease markers.

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Interferon-α is the primary pl ...... ctivation and disease markers.

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P2860

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

The nature of the principal type 1 interferon-producing cells in human blood

Lack of mucosal immune reconstitution during prolonged treatment of acute and early HIV-1 infection.

The dynamics of CD4+ T-cell depletion in HIV disease.

Downregulation of robust acute type I interferon responses distinguishes nonpathogenic simian immunodeficiency virus (SIV) infection of natural hosts from pathogenic SIV infection of rhesus macaques.

T cell depletion in HIV-1 infection: how CD4+ T cells go out of stock.

Endocytosis of HIV-1 activates plasmacytoid dendritic cells via Toll-like receptor-viral RNA interactions

Interferon-alpha administration enhances CD8+ T cell activation in HIV infection

Differential expression of IFN-alpha and TRAIL/DR5 in lymphoid tissue of progressor versus nonprogressor HIV-1-infected patients.

Natural type I interferon-producing cells as a link between innate and adaptive immunity.

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