Genes involved in oxidative phosphorylation are coordinately upregulated with fasting hyperglycaemia in livers of patients with type 2 diabetes.
about
A network biology workflow to study transcriptomics data of the diabetic liver.Diet-induced gene expression of isolated pancreatic islets from a polygenic mouse model of the metabolic syndrome.Proteome, phosphoproteome, and hydroxyproteome of liver mitochondria in diabetic rats at early pathogenic stages.Do mitochondria care about insulin resistance?Uncovering the complexity of transcriptomes with RNA-Seq.Inverse correlation between serum levels of selenoprotein P and adiponectin in patients with type 2 diabetes.RNA-Seq and human complex diseases: recent accomplishments and future perspectives.Network-based prediction for sources of transcriptional dysregulation using latent pathway identification analysis.Oxidative phosphorylation flexibility in the liver of mice resistant to high-fat diet-induced hepatic steatosis.The role of mitochondria in the pathogenesis of type 2 diabetesProteasome dysfunction mediates obesity-induced endoplasmic reticulum stress and insulin resistance in the liverMitochondrial plasticity in obesity and diabetes mellitus.Statistical properties on semiparametric regression for evaluating pathway effects.Thyroid hormone-related regulation of gene expression in human fatty liver.Nutrition-/diet-induced changes in gene expression in pancreatic β-cells.The multiple universes of estrogen-related receptor α and γ in metabolic control and related diseases.Gene Expression Signatures and the Spectrum of Coronary Artery Disease.Oxidative metabolism genes are not responsive to oxidative stress in rodent Beta cell linesIncreased mitochondrial oxidative phosphorylation in the liver is associated with obesity and insulin resistance.Metformin prevents and reverses inflammation in a non-diabetic mouse model of nonalcoholic steatohepatitis.Obesity upregulates genes involved in oxidative phosphorylation in livers of diabetic patients.Selenoprotein P as a diabetes-associated hepatokine that impairs angiogenesis by inducing VEGF resistance in vascular endothelial cells.Inhibin βE (INHBE) is a possible insulin resistance-associated hepatokine identified by comprehensive gene expression analysis in human liver biopsy samples.The metabolic response to a high-fat diet reveals obesity-prone and -resistant phenotypes in mice with distinct mRNA-seq transcriptome profiles.
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P2860
Genes involved in oxidative phosphorylation are coordinately upregulated with fasting hyperglycaemia in livers of patients with type 2 diabetes.
description
2006 nî lūn-bûn
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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@ast
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@en
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@nl
type
label
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@ast
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@en
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@nl
prefLabel
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@ast
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@en
Genes involved in oxidative ph ...... patients with type 2 diabetes.
@nl
P2093
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P1433
P1476
Genes involved in oxidative ph ...... patients with type 2 diabetes
@en
P2093
N Matsuzawa
T Takamura
T Yamashita
P2860
P2888
P304
P356
10.1007/S00125-006-0489-8
P50
P577
2006-12-23T00:00:00Z
P5875
P6179
1001314529