Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice. - Wikidata - wikimedia - TriplyDB

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

http://www.wikidata.org/entity/Q33276940

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Antigen-antibody dissociation in Alzheimer disease: a novel approach to diagnosis Autoantibodies in Alzheimer's disease: potential biomarkers, pathogenic roles, and therapeutic implications Spink2 modulates apoptotic susceptibility and is a candidate gene in the Rgcs1 QTL that affects retinal ganglion cell death after optic nerve damage Silencing of Fem1cR3 gene expression in the DBA/2J mouse precedes retinal ganglion cell death and is associated with histone deacetylase activity Neuroprotective natural antibodies to assemblies of amyloidogenic peptides decrease with normal aging and advancing Alzheimer's disease.Reducing AD-like pathology in 3xTg-AD mouse model by DNA epitope vaccine - a novel immunotherapeutic strategy.Rgcs1, a dominant QTL that affects retinal ganglion cell death after optic nerve crush in mice.Differential response of C57BL/6J mouse and DBA/2J mouse to optic nerve crush Laser-induced ocular hypertension in albino CD-1 mice.Retinal cell responses to elevated intraocular pressure: a gene array comparison between the whole retina and retinal ganglion cell layer.Immunological origin and functional properties of catalytic autoantibodies to amyloid beta peptide.Histone H4 deacetylation plays a critical role in early gene silencing during neuronal apoptosis Rodent models of glaucoma.Early gene expression changes in the retinal ganglion cell layer of a rat glaucoma model A single nucleotide polymorphism in the Bax gene promoter affects transcription and influences retinal ganglion cell death Anti-amyloidogenic activity of IgGs contained in normal plasma Optic nerve crush induces spatial and temporal gene expression patterns in retina and optic nerve of BALB/cJ mice.DBA/2J mice are susceptible to diabetic nephropathy and diabetic exacerbation of IOP elevation.Histone deacetylase 3 (HDAC3) plays an important role in retinal ganglion cell death after acute optic nerve injury.Human anti-Aβ IgGs target conformational epitopes on synthetic dimer assemblies and the AD brain-derived peptide.Tumor necrosis factor alpha has an early protective effect on retinal ganglion cells after optic nerve crush.Mouse models of retinal ganglion cell death and glaucoma.Accelerated retinal ganglion cell death in mice deficient in the Sigma-1 receptor Optic neuritis and retinal ganglion cell loss in a chronic murine model of multiple sclerosis.ELISA measurement of specific non-antigen-bound antibodies to Aβ1-42 monomer and soluble oligomers in sera from Alzheimer's disease, mild cognitively impaired, and noncognitively impaired subjects IgG Conformer's Binding to Amyloidogenic Aggregates.Dose-dependent treatment of optic nerve crush by exogenous systemic mutant erythropoietin The Bcl-2 family member BIM has multiple glaucoma-relevant functions in DBA/2J mice Retinal Ganglion Cell Loss is Delayed Following Optic Nerve Crush in NLRP3 Knockout Mice.Mutant human myocilin induces strain specific differences in ocular hypertension and optic nerve damage in mice.Nuclear atrophy of retinal ganglion cells precedes the bax-dependent stage of apoptosis.Retinal glial responses to optic nerve crush are attenuated in Bax-deficient mice and modulated by purinergic signaling pathways.Evaluation of the percentage of ganglion cells in the ganglion cell layer of the rodent retina Hydrocortisone stimulates neurite outgrowth from mouse retinal explants by modulating macroglial activity Suppression of amyloid deposition leads to long-term reductions in Alzheimer's pathologies in Tg2576 mice.Epigenetic intervention with a BET inhibitor ameliorates acute retinal ganglion cell death in mice.Potential sources of interference on Abeta immunoassays in biological samples CNS repair and axon regeneration: Using genetic variation to determine mechanisms.Characterizing the "POAGome": A bioinformatics-driven approach to primary open-angle glaucoma.JUN is important for ocular hypertension-induced retinal ganglion cell degeneration.

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P2860

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

http://www.wikidata.org/entity/Q33276940

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

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Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

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Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

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type

label

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

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Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

@en

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

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prefLabel

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

@ast

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

@en

Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

@nl

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Dominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.

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Cassandra L Schlamp

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Robert W Nickells

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Sheila J Semaan

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Yan Li

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Overcoming antigen masking of anti-amyloidbeta antibodies reveals breaking of B cell tolerance by virus-like particles in amyloidbeta immunized amyloid precursor protein transgenic mice

Susceptibility to neurodegeneration in a glaucoma is modified by Bax gene dosage

Mutation analysis of the neurofilament M gene in Parkinson's disease

Suppression of developmental retinal cell death but not of photoreceptor degeneration in Bax-deficient mice

BAX is required for neuronal death after trophic factor deprivation and during development

Mitochondria and apoptosis

Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse

Factors for glaucoma progression and the effect of treatment: the early manifest glaucoma trial.

Deformation of the lamina cribrosa and anterior scleral canal wall in early experimental glaucoma.

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