High CO2 levels impair alveolar epithelial function independently of pH. - Wikidata - wikimedia - TriplyDB

High CO2 levels impair alveolar epithelial function independently of pH.

High CO2 levels impair alveolar epithelial function independently of pH.

http://www.wikidata.org/entity/Q33307536

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Effect of elevated carbon dioxide on bronchial epithelial innate immune receptor response to organic dust from swine confinement barns The response to high CO2 levels requires the neuropeptide secretion component HID-1 to promote pumping inhibition Endothelin-1 impairs alveolar epithelial function via endothelial ETB receptor AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis Insulin regulates alveolar epithelial function by inducing Na+/K+-ATPase translocation to the plasma membrane in a process mediated by the action of Akt Carbonic anhydrase inhibitor attenuates ischemia-reperfusion induced acute lung injury.Gas Exchange Disturbances Regulate Alveolar Fluid Clearance during Acute Lung Injury.Resolution of pulmonary edema. Thirty years of progress.Bench-to-bedside review: carbon dioxide.Elevated CO2 selectively inhibits interleukin-6 and tumor necrosis factor expression and decreases phagocytosis in the macrophage.Extracellular signal-regulated kinase (ERK) participates in the hypercapnia-induced Na,K-ATPase downregulation.Evolutionary conserved role of c-Jun-N-terminal kinase in CO2-induced epithelial dysfunction.Role of endothelin-1 in acute lung injury.Hypoxia leads to Na,K-ATPase downregulation via Ca(2+) release-activated Ca(2+) channels and AMPK activation Elevated CO(2) levels cause mitochondrial dysfunction and impair cell proliferation.Hypercapnia Inhibits Autophagy and Bacterial Killing in Human Macrophages by Increasing Expression of Bcl-2 and Bcl-xL.Pharyngeal pumping inhibition and avoidance by acute exposure to high CO2 levels are both regulated by the BAG neurons via different molecular pathways.Ubiquitination and proteolysis in acute lung injury Sensing, physiological effects and molecular response to elevated CO2 levels in eukaryotes Hypercapnia: a nonpermissive environment for the lung.High CO2 Leads to Na,K-ATPase Endocytosis via c-Jun Amino-Terminal Kinase-Induced LMO7b Phosphorylation.Permissive hypercapnia for severe acute respiratory distress syndrome in immunocompromised children: A single center experience.Protein kinase A-Iα regulates Na,K-ATPase endocytosis in alveolar epithelial cells exposed to high CO(2) concentrations.Elevated CO2 levels affect development, motility, and fertility and extend life span in Caenorhabditis elegans.Alpha1-AMP-activated protein kinase regulates hypoxia-induced Na,K-ATPase endocytosis via direct phosphorylation of protein kinase C zeta.CO2-induced ion and fluid transport in human retinal pigment epithelium.Elevated CO2 suppresses specific Drosophila innate immune responses and resistance to bacterial infection Ubiquitination participates in the lysosomal degradation of Na,K-ATPase in steady-state conditions Carbon dioxide-sensing in organisms and its implications for human disease.Alveolar Fluid Clearance in Pathologically Relevant Conditions: In Vitro and In Vivo Models of Acute Respiratory Distress Syndrome.Novel concepts of acute lung injury and alveolar-capillary barrier dysfunction.Severe hypercapnia and outcome of mechanically ventilated patients with moderate or severe acute respiratory distress syndrome.Update on the role of extracorporeal CO₂ removal as an adjunct to mechanical ventilation in ARDS.Hypercapnia Impairs ENaC Cell Surface Stability by Promoting Phosphorylation, Polyubiquitination and Endocytosis of β-ENaC in a Human Alveolar Epithelial Cell Line.Hypercapnia Accelerates Adipogenesis: A Novel Role of High CO2 in Exacerbating Obesity.Focused Screening Identifies Evoxine as a Small Molecule That Counteracts CO2-Induced Immune Suppression.Hypercapnia modulates cAMP signalling and cystic fibrosis transmembrane conductance regulator-dependent anion and fluid secretion in airway epithelia.Effects of hypercapnia on the lung.Linking Ventilator Injury-Induced Leak across the Blood-Gas Barrier to Derangements in Murine Lung Function Lung Edema Clearance: Relevance to Patients with Lung Injury.

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High CO2 levels impair alveolar epithelial function independently of pH.

http://www.wikidata.org/entity/Q33307536

description

2007 nî lūn-bûn

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2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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name

High CO2 levels impair alveolar epithelial function independently of pH.

@ast

High CO2 levels impair alveolar epithelial function independently of pH.

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High CO2 levels impair alveolar epithelial function independently of pH.

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type

label

High CO2 levels impair alveolar epithelial function independently of pH.

@ast

High CO2 levels impair alveolar epithelial function independently of pH.

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High CO2 levels impair alveolar epithelial function independently of pH.

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prefLabel

High CO2 levels impair alveolar epithelial function independently of pH.

@ast

High CO2 levels impair alveolar epithelial function independently of pH.

@en

High CO2 levels impair alveolar epithelial function independently of pH.

@nl

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High CO2 levels impair alveolar epithelial function independently of pH.

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Arturo Briva

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Daniel Batlle

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Emilia Lecuona

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Humberto E Trejo

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István Vadász

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Jacob I Sznajder

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Jiwang Chen

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Vidas Dumasius

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Cleavage of structural proteins during the assembly of the head of bacteriophage T4

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Ventilation with lower tidal volumes as compared with traditional tidal volumes for acute lung injury and the acute respiratory distress syndrome. The Acute Respiratory Distress Syndrome Network

The acute respiratory distress syndrome

Smoke/burn injury-induced respiratory failure elicits apoptosis in ovine lungs and cultured lung cells, ameliorated with arteriovenous CO2 removal.

Dopamine-induced exocytosis of Na,K-ATPase is dependent on activation of protein kinase C-epsilon and -delta.

Carbon dioxide and the critically ill--too little of a good thing?

Evidence from renal proximal tubules that HCO3- and solute reabsorption are acutely regulated not by pH but by basolateral HCO3- and CO2.

Effect of a protective-ventilation strategy on mortality in the acute respiratory distress syndrome.

Lung epithelial fluid transport and the resolution of pulmonary edema.

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István Vadász

Pavlos Myrianthefs

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