about
Multiple-integrations of HPV16 genome and altered transcription of viral oncogenes and cellular genes are associated with the development of cervical cancerMultiple ASF/SF2 sites in the human papillomavirus type 16 (HPV-16) E4-coding region promote splicing to the most commonly used 3'-splice site on the HPV-16 genome.Alternative splicing of human papillomavirus type-16 E6/E6* early mRNA is coupled to EGF signaling via Erk1/2 activation.Analysis of transcripts from predicted open reading frames of Musca domestica salivary gland hypertrophy virus.Intron definition and a branch site adenosine at nt 385 control RNA splicing of HPV16 E6*I and E7 expressionE6-associated transcription patterns in human papilloma virus 16-positive cervical tissuesPersistence of human papillomavirus infection: keys to malignant progressionEight nucleotide substitutions inhibit splicing to HPV-16 3'-splice site SA3358 and reduce the efficiency by which HPV-16 increases the life span of primary human keratinocytes.Converging strategies in expression of human complex retrovirusesProductive Lifecycle of Human Papillomaviruses that Depends Upon Squamous Epithelial Differentiation.Characterization of Mus musculus papillomavirus 1 infection in situ reveals an unusual pattern of late gene expression and capsid protein localization.Functional interplay between viral and cellular SR proteins in control of post-transcriptional gene regulation.Suppression of HPV-16 late L1 5'-splice site SD3632 by binding of hnRNP D proteins and hnRNP A2/B1 to upstream AUAGUA RNA motifs.Codon usage roles in human papillomavirus.Fine tuning of the temporal expression of HTLV-1 and HTLV-2.Physical and methylation status of human papillomavirus 16 in asymptomatic cervical infections changes with malignant transformation.An L2 SUMO interacting motif is important for PML localization and infection of human papillomavirus type 16.The E8--E2 gene product of human papillomavirus type 16 represses early transcription and replication but is dispensable for viral plasmid persistence in keratinocytes.HPV-16 E2 contributes to induction of HPV-16 late gene expression by inhibiting early polyadenylation.Molecular and phylogenetic analysis of the HPV 16 E4 gene in cervical lesions from women in Greece.
P2860
Q31172479-B45AEC8F-B4C7-44B5-93AF-2CA55DCCD99CQ33592653-18276FF5-D13A-43F2-9410-C3C043BAE289Q33859710-B0EBF4DD-F822-47BE-BAF7-2FBF286DD356Q33920506-246115E6-15D6-47C0-9127-71445465B4A9Q34441421-42F559B2-A30C-4660-A3E1-70121C0A0D4BQ34582740-71EC1B70-627C-4792-8C32-611F1A65816FQ34690139-CDD0454D-8D2F-496E-8C8B-7E91B52C3DB2Q34988558-505DA9FA-64A1-4DC3-9F95-CE2991063B15Q35261679-5478EADA-EC47-4ACB-BDFF-E4720E19FDAEQ35908359-4A632C9C-57A8-41C9-B369-EC419598D6C3Q37336676-56A5D206-6AAD-465E-AFCF-2F74328BD1C5Q37394282-310C15DE-B594-4D6F-AABE-604ED4CA6E3DQ37528882-1958C312-2FBF-4B47-9AA5-E75A35E9FAC0Q37949297-9AC5E1FD-DDF5-4BCC-A311-5D3120A10CC0Q38137077-58C4480F-E2B9-4278-B3AA-172CA672403DQ38922687-6F92178B-2D9C-49E5-B9C1-DF91D178E114Q39032783-A90AF04E-4384-47E3-9FCC-427487EE1BA3Q41994813-B9BAD1ED-6367-430E-AB73-009FDFAE9991Q42241871-3165DE71-6429-45A3-B8DB-FF8E5192C06FQ48048969-A4DCFDC0-1640-4009-A52D-E44919D287CA
P2860
description
2008 nî lūn-bûn
@nan
2008 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
HPV-16 RNA processing.
@ast
HPV-16 RNA processing.
@en
HPV-16 RNA processing.
@nl
type
label
HPV-16 RNA processing.
@ast
HPV-16 RNA processing.
@en
HPV-16 RNA processing.
@nl
prefLabel
HPV-16 RNA processing.
@ast
HPV-16 RNA processing.
@en
HPV-16 RNA processing.
@nl
P356
P1476
HPV-16 RNA processing.
@en
P2093
Stefan Schwartz
P304
P356
10.2741/3123
P577
2008-05-01T00:00:00Z