Persistence of human papillomavirus infection: keys to malignant progression - Wikidata - wikimedia - TriplyDB

Persistence of human papillomavirus infection: keys to malignant progression

Persistence of human papillomavirus infection: keys to malignant progression

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Human papillomavirus induced transformation in cervical and head and neck cancers Attenuated Recombinant Influenza A Virus Expressing HPV16 E6 and E7 as a Novel Therapeutic Vaccine Approach Cervical Cancer Screening after Perimenopause: How Is Human Papillomavirus Test Performed?Control of human papillomavirus gene expression by alternative splicing The acetyltransferase Tip60 is a critical regulator of the differentiation-dependent amplification of human papillomaviruses.Degradation of Human PDZ-Proteins by Human Alphapapillomaviruses Represents an Evolutionary Adaptation to a Novel Cellular Niche Human papillomavirus and head and neck carcinomas: focus on evidence in the babel of published data The antiviral restriction factors IFITM1, 2 and 3 do not inhibit infection of human papillomavirus, cytomegalovirus and adenovirus.Interactions between polymorphisms in the 3'untranslated region of the cyclin dependent kinase 6 gene and the human papillomavirus infection, and risk of cervical precancerous lesions.Therapeutic options for treatment of human papillomavirus-associated cancers - novel immunologic vaccines: ADXS11-001.Differential methylation of the HPV 16 upstream regulatory region during epithelial differentiation and neoplastic transformation.The cellular bromodomain protein Brd4 has multiple functions in E2-mediated papillomavirus transcription activation Identification of protein biomarkers for cervical cancer using human cervicovaginal fluid Molecular mapping of high-grade cervical intraepithelial neoplasia shows etiological dominance of HPV16.High-risk human papillomavirus E6 protein promotes reprogramming of Fanconi anemia patient cells through repression of p53 but does not allow for sustained growth of induced pluripotent stem cells.Functional interaction between human papillomavirus type 16 E6 and E7 oncoproteins and cigarette smoke components in lung epithelial cells.Polymorphisms in immune mediators associate with risk of cervical cancer Design of a highly effective therapeutic HPV16 E6/E7-specific DNA vaccine: optimization by different ways of sequence rearrangements (shuffling).Presence of human papilloma virus in a series of breast carcinoma from Argentina.Productive infection of bovine papillomavirus type 2 in the urothelial cells of naturally occurring urinary bladder tumors in cattle and water buffaloes.Cidofovir selectivity is based on the different response of normal and cancer cells to DNA damage.Multiplex Identification of Human Papillomavirus 16 DNA Integration Sites in Cervical Carcinomas.RPS19 and TYMS SNPs and Prevalent High Risk Human Papilloma Virus Infection in Nigerian Women Assessing the detection of human papillomavirus late mRNA in liquid base cytology samples for risk stratification of cervical disease.APOBEC3A functions as a restriction factor of human papillomavirus DNA damage repair genes controlling human papillomavirus (HPV) episome levels under conditions of stability and extreme instability HIV associated high-risk HPV infection among Nigerian women.Link between chronic inflammation and human papillomavirus-induced carcinogenesis (Review).Genetic variation of human papillomavirus type 16 in individual clinical specimens revealed by deep sequencing.Suppression of STAT-1 expression by human papillomaviruses is necessary for differentiation-dependent genome amplification and plasmid maintenance.Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.The human papillomavirus type 16 E5 oncoprotein translocates calpactin I to the perinuclear region.HPV-Associated Head and Neck Cancer: Unique Features of Epidemiology and Clinical Management.Stratification of HPV-induced cervical pathology using the virally encoded molecular marker E4 in combination with p16 or MCM Bridging Links between Long Noncoding RNA HOTAIR and HPV Oncoprotein E7 in Cervical Cancer Pathogenesis.RNA Binding Proteins that Control Human Papillomavirus Gene Expression.Heterogeneous Nuclear Ribonucleoprotein C Proteins Interact with the Human Papillomavirus Type 16 (HPV16) Early 3'-Untranslated Region and Alleviate Suppression of HPV16 Late L1 mRNA Splicing.Productive Lifecycle of Human Papillomaviruses that Depends Upon Squamous Epithelial Differentiation.Serial type-specific human papillomavirus (HPV) load measurement allows differentiation between regressing cervical lesions and serial virion productive transient infections.The fanconi anemia pathway limits human papillomavirus replication

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P2860

Persistence of human papillomavirus infection: keys to malignant progression

http://www.wikidata.org/entity/Q34690139

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2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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Persistence of human papillomavirus infection: keys to malignant progression

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J.TIM.2010.10.002

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Trends in Microbiology

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Persistence of human papillomavirus infection: keys to malignant progression

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Jason Bodily

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Laimonis A Laimins

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P2860

The Hallmarks of Cancer

Association of the human papillomavirus type 16 E7 oncoprotein with the 600-kDa retinoblastoma protein-associated factor, p600

Antiviral actions of interferons

Quadrivalent Human Papillomavirus Vaccine: Recommendations of the Advisory Committee on Immunization Practices (ACIP)

Human papillomaviruses activate the ATM DNA damage pathway for viral genome amplification upon differentiation

Human papillomavirus and anal intraepithelial neoplasia.

HPV-16 RNA processing.

Human Immunodeficiency Virus and Human Papilloma Virus - why HPV-induced lesions do not spontaneously resolve and why therapeutic vaccination can be successful.

Role of cytokines in epidermal Langerhans cell migration.

Temporal usage of multiple promoters during the life cycle of human papillomavirus type 31b.

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33-39

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scholarly article

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10.1016/J.TIM.2010.10.002

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1

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47678346

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21050765

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3059725

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