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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

http://www.wikidata.org/entity/Q33402916

about

Identification of novel inhibitors of dietary lipid absorption using zebrafish Postprandial lipemia enhances the capacity of large HDL2 particles to mediate free cholesterol efflux via SR-BI and ABCG1 pathways in type IIB hyperlipidemia.Deletion of intestinal epithelial insulin receptor attenuates high-fat diet-induced elevations in cholesterol and stem, enteroendocrine, and Paneth cell mRNAs.Micellar lipid composition affects micelle interaction with class B scavenger receptor extracellular loops.Gut microbiota-derived lipopolysaccharide uptake and trafficking to adipose tissue: implications for inflammation and obesity.Intestinal SR-BI does not impact cholesterol absorption or transintestinal cholesterol efflux in mice.Autophagosomes contribute to intracellular lipid distribution in enterocytes.Activation of intestinal peroxisome proliferator-activated receptor-α increases high-density lipoprotein production.Gut-liver interaction in triglyceride-rich lipoprotein metabolism.Dietary 1-monoolein decreases postprandial GIP release by reducing jejunal transport of glucose and fatty acid in rodents.Scavenger receptor class B type I is a plasma membrane cholesterol sensor.Vitamin E Bioavailability: Mechanisms of Intestinal Absorption in the Spotlight.Cholesterol trafficking and raft-like membrane domain composition mediate scavenger receptor class B type 1-dependent lipid sensing in intestinal epithelial cells.Intestinal scavenger receptor class B type I as a novel regulator of chylomicron production in healthy and diet-induced obese states.Intestinal SR-BI is upregulated in insulin-resistant states and is associated with overproduction of intestinal apoB48-containing lipoproteins.

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

http://www.wikidata.org/entity/Q33402916

description

2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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JOURNAL.PONE.0004278

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Sensing of dietary lipids by enterocytes: a new role for SR-BI/CLA-1

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Carine Cueille

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Danielle Chateau

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François Delers

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Jean Chambaz

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Monique Rousset

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Olivier Béaslas

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P2860

Targeted disruption of the PDZK1 gene in mice causes tissue-specific depletion of the high density lipoprotein receptor scavenger receptor class B type I and altered lipoprotein metabolism

Serum amyloid A binding to CLA-1 (CD36 and LIMPII analogous-1) mediates serum amyloid A protein-induced activation of ERK1/2 and p38 mitogen-activated protein kinases

Down-regulation of intestinal scavenger receptor class B, type I (SR-BI) expression in rodents under conditions of deficient bile delivery to the intestine

Murine SR-BI, a high density lipoprotein receptor that mediates selective lipid uptake, is N-glycosylated and fatty acylated and colocalizes with plasma membrane caveolae

High-density lipoprotein promotes endothelial cell migration and reendothelialization via scavenger receptor-B type I

Niemann-Pick C1 Like 1 protein is critical for intestinal cholesterol absorption

Insulin and angiotensin II induce the translocation of scavenger receptor class B, type I from intracellular sites to the plasma membrane of adipocytes.

Hepatic cholesterol and bile acid metabolism and intestinal cholesterol absorption in scavenger receptor class B type I-deficient mice.

Proteomic characterization of lipid rafts markers from the rat intestinal brush border.

Cholesterol binding, efflux, and a PDZ-interacting domain of scavenger receptor-BI mediate HDL-initiated signaling.

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1

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Véronique Carrière

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