Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis - Wikidata - wikimedia - TriplyDB

Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis

Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis

http://www.wikidata.org/entity/Q33492757

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The significance and regulatory mechanisms of innate immune cells in the development of sepsis Modulation of Dendritic-Epithelial Cell Responses against Sphingomonas Paucimobilis by Dietary Fibers Early alterations of B cells in patients with septic shock.An unbalanced PD-L1/CD86 ratio in CD14(++)CD16(+) monocytes is correlated with HCV viremia during chronic HCV infection.Neutrophils activate alveolar macrophages by producing caspase-6-mediated cleavage of IL-1 receptor-associated kinase-M Role of circulating lymphocytes in patients with sepsis.TRIF is required for TLR4 mediated adjuvant effects on T cell clonal expansion.FoxP3⁺ regulatory T cells attenuate experimental necrotizing enterocolitis Effects of exercise on weight loss and monocytes in obese mice Monocyte unresponsiveness and impaired IL1β, TNFα and IL7 production are associated with a poor outcome in Malawian adults with pulmonary tuberculosis Characterization of the inflammatory cell infiltrate and expression of costimulatory molecules in chronic echinococcus granulosus infection of the human liver IRAK-M modulates expression of IL-10 and cell surface markers CD80 and MHC II after bacterial re-stimulation of tolerized dendritic cells A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis.The pathogenesis of sepsis.Macrophage A2A adenosinergic receptor modulates oxygen-induced augmentation of murine lung injury.Colonic epithelial response to injury requires Myd88 signaling in myeloid cells CD80+ and CD86+ B cells as biomarkers and possible therapeutic targets in HTLV-1 associated myelopathy/tropical spastic paraparesis and multiple sclerosis.Sepsis: multiple abnormalities, heterogeneous responses, and evolving understanding.Diverse macrophage populations mediate acute lung inflammation and resolution.Anti-inflammatory effects of silver-polyvinyl pyrrolidone (Ag-PVP) nanoparticles in mouse macrophages infected with live Chlamydia trachomatis.IRAK-M regulation and function in host defense and immune homeostasis.Immunoregulatory profile of monocytes from cutaneous leishmaniasis patients and association with lesion size.Cutting Edge: 2B4-Mediated Coinhibition of CD4+ T Cells Underlies Mortality in Experimental Sepsis.Febrile response induced by cecal ligation and puncture (CLP) in rats: involvement of prostaglandin E2 and cytokines.Functional polymorphisms in CD86 gene are associated with susceptibility to pneumonia-induced sepsis.Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction.Effect of clarithromycin in inflammatory markers of patients with ventilator-associated pneumonia and sepsis caused by Gram-negative bacteria: results from a randomized clinical study CD80 Regulates Th17 Cell Differentiation in Coxsackie Virus B3-Induced Acute Myocarditis.CD80/CD86 signaling contributes to the proinflammatory response of Staphylococcus aureus in the airway.Recombinant human lactoferrin modulates human PBMC derived macrophage responses to BCG and LPS.Targeting the CD80/CD86 costimulatory pathway with CTLA4-Ig directs microglia toward a repair phenotype and promotes axonal outgrowth.Integrative omics to detect bacteremia in patients with febrile neutropenia.Fluid resuscitation-associated increased mortality and inflammatory cytokine expression in murine polymicrobial sepsis

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Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis

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description

2009 nî lūn-bûn

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2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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Genome-level expression profiles in pediatric septic shock indicate a role for altered zinc homeostasis in poor outcome.

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