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CpG methylation controls reactivation of HIV from latency.

CpG methylation controls reactivation of HIV from latency.

http://www.wikidata.org/entity/Q33495565

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Epigenetic control of HIV-1 post integration latency: implications for therapy Impact of Chromatin on HIV Replication Redefining the viral reservoirs that prevent HIV-1 eradication HIV-1 functional cure: will the dream come true?Transcriptional control of HIV latency: cellular signaling pathways, epigenetics, happenstance and the hope for a cure The immunological synapse: the gateway to the HIV reservoir Recent developments in human immunodeficiency virus-1 latency research Phosphorylation of CDK9 at Ser175 enhances HIV transcription and is a marker of activated P-TEFb in CD4(+) T lymphocytes Epigenetic alterations in the brain associated with HIV-1 infection and methamphetamine dependence Novel genetic locus implicated for HIV-1 acquisition with putative regulatory links to HIV replication and infectivity: a genome-wide association study Therapy for persistent HIV.Eradication therapies for HIV Infection: time to begin again.Infection with influenza A viruses causes changes in promoter DNA methylation of inflammatory genes.Negative elongation factor is required for the maintenance of proviral latency but does not induce promoter-proximal pausing of RNA polymerase II on the HIV long terminal repeat.Epigenetic analysis of HIV-1 proviral genomes from infected individuals: predominance of unmethylated CpG's.Mechanisms of HIV Transcriptional Regulation by Drugs of Abuse.Combinatorial latency reactivation for HIV-1 subtypes and variants Involvement of histone H3 lysine 9 (H3K9) methyltransferase G9a in the maintenance of HIV-1 latency and its reactivation by BIX01294.The Human Immunodeficiency Virus 1 ASP RNA promotes viral latency by recruiting the Polycomb Repressor Complex 2 and promoting nucleosome assembly Activation of HIV transcription by the viral Tat protein requires a demethylation step mediated by lysine-specific demethylase 1 (LSD1/KDM1).Combination of biological screening in a cellular model of viral latency and virtual screening identifies novel compounds that reactivate HIV-1 A flexible model of HIV-1 latency permitting evaluation of many primary CD4 T-cell reservoirs.Cell line-dependent variability in HIV activation employing DNMT inhibitors Transcriptional provirus silencing as a crosstalk of de novo DNA methylation and epigenomic features at the integration site Ectopic DNMT3L triggers assembly of a repressive complex for retroviral silencing in somatic cells.Snapshots: chromatin control of viral infection.The molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit.(Some) cellular mechanisms influencing the transcription of human endogenous retrovirus, HERV-Fc1.Relationship between LTR methylation and gag expression of HIV-1 in human spermatozoa and sperm-derived embryos Comparative analysis of measures of viral reservoirs in HIV-1 eradication studies Towards an HIV cure: a global scientific strategy Short chain fatty acids potently induce latent HIV-1 in T-cells by activating P-TEFb and multiple histone modifications.Interaction between endogenous bacterial flora and latent HIV infection.Histone deacetylase inhibitors and HIV latency.Epigenetic heterogeneity in HIV-1 latency establishment.Role of DNA methylation in expression and transmission of porcine endogenous retroviruses.Viral-host interactions that control HIV-1 transcriptional elongation Genome-wide DNA methylation patterns in CD4+ T cells from patients with systemic lupus erythematosus.T-cell receptor signaling enhances transcriptional elongation from latent HIV proviruses by activating P-TEFb through an ERK-dependent pathway.Screening for noise in gene expression identifies drug synergies

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CpG methylation controls reactivation of HIV from latency.

http://www.wikidata.org/entity/Q33495565

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

CpG methylation controls reactivation of HIV from latency.

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CpG methylation controls reactivation of HIV from latency.

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type

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CpG methylation controls reactivation of HIV from latency.

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CpG methylation controls reactivation of HIV from latency.

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prefLabel

CpG methylation controls reactivation of HIV from latency.

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CpG methylation controls reactivation of HIV from latency.

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JOURNAL.PPAT.1000554

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CpG methylation controls reactivation of HIV from latency.

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Allan Guiguen

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Daniel Olive

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Françoise Gondois-Rey

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Jana Blazkova

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Jiri Hejnar

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Katerina Trejbalova

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Patrick Philibert

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Philippe Halfon

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P2860

Emerging concepts in the immunopathogenesis of AIDS

Recruitment of chromatin-modifying enzymes by CTIP2 promotes HIV-1 transcriptional silencing

Requirement for SWI/SNF chromatin-remodeling complex in Tat-mediated activation of the HIV-1 promoter

The SWI/SNF chromatin-remodeling complex is a cofactor for Tat transactivation of the HIV promoter

Counterregulation of chromatin deacetylation and histone deacetylase occupancy at the integrated promoter of human immunodeficiency virus type 1 (HIV-1) by the HIV-1 repressor YY1 and HIV-1 activator Tat

Residual human immunodeficiency virus type 1 viremia in some patients on antiretroviral therapy is dominated by a small number of invariant clones rarely found in circulating CD4+ T cells

Dual role of prostratin in inhibition of infection and reactivation of human immunodeficiency virus from latency in primary blood lymphocytes and lymphoid tissue

CREB-binding protein/p300 are transcriptional coactivators of p65

Silencing of human T-cell leukemia virus type I gene transcription by epigenetic mechanisms

Identification of a reservoir for HIV-1 in patients on highly active antiretroviral therapy

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e1000554

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scholarly article

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10.1371/JOURNAL.PPAT.1000554

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8

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5

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Carine Van Lint

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2009-08-21T00:00:00Z

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26757312

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19696893

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2722084

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