Recruitment of fanconi anemia and breast cancer proteins to DNA damage sites is differentially governed by replication
about
Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replicationINO80 chromatin remodeling complex promotes the removal of UV lesions by the nucleotide excision repair pathwayA histone-fold complex and FANCM form a conserved DNA-remodeling complex to maintain genome stabilityFanconi anemia (FA) binding protein FAAP20 stabilizes FA complementation group A (FANCA) and participates in interstrand cross-link repairA ubiquitin-binding protein, FAAP20, links RNF8-mediated ubiquitination to the Fanconi anemia DNA repair networkSusceptibility pathways in Fanconi's anemia and breast cancerHigh mobility group protein 1: A collaborator in nucleosome dynamics and estrogen-responsive gene expressionStructure of the FANCI-FANCD2 Complex: Insights into the Fanconi Anemia DNA Repair PathwayFanconi anaemia and the repair of Watson and Crick DNA crosslinksMammalian nucleotide excision repair proteins and interstrand crosslink repairAdvances in understanding the complex mechanisms of DNA interstrand cross-link repairHuman variation databasesThe FANCM/FAAP24 complex is required for the DNA interstrand crosslink-induced checkpoint responseCtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity.Mutagenic repair of DNA interstrand crosslinks.The BRCA1-interacting protein Abraxas is required for genomic stability and tumor suppression.Using synthetic DNA interstrand crosslinks to elucidate repair pathways and identify new therapeutic targets for cancer chemotherapy.Orchestrating the nucleases involved in DNA interstrand cross-link (ICL) repair.Repair of cisplatin-induced DNA interstrand crosslinks by a replication-independent pathway involving transcription-coupled repair and translesion synthesisDNA double strand breaks but not interstrand crosslinks prevent progress through meiosis in fully grown mouse oocytes.Fanconi anemia group J helicase and MRE11 nuclease interact to facilitate the DNA damage response.The Fanconi anemia pathway and ICL repair: implications for cancer therapy.BRCA1 is required for postreplication repair after UV-induced DNA damageAlcohol metabolism in human cells causes DNA damage and activates the Fanconi anemia-breast cancer susceptibility (FA-BRCA) DNA damage response network.Rescue of replication failure by Fanconi anaemia proteins.Single Molecule Analysis of Laser Localized Interstrand Crosslinks.Personalized therapy for urothelial cancer: review of the clinical evidence.UHRF1 contributes to DNA damage repair as a lesion recognition factor and nuclease scaffold.FANCM and FAAP24 maintain genome stability via cooperative as well as unique functionsFANCJ at the FORK.Construction of plasmids containing site-specific DNA interstrand cross-links for biochemical and cell biological studies.DNA crosslinking damage and cancer - a tale of friend and foe.Ubiquitylation and the Fanconi anemia pathway.BRCA1 and its toolbox for the maintenance of genome integrityRepair of DNA interstrand cross-links during S phase of the mammalian cell cycle.The Fanconi anemia pathway in replication stress and DNA crosslink repair.REV1 and DNA polymerase zeta in DNA interstrand crosslink repair.The roles of DNA polymerase ζ and the Y family DNA polymerases in promoting or preventing genome instability.The yin and yang of repair mechanisms in DNA structure-induced genetic instability.The differences between ICL repair during and outside of S phase
P2860
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P2860
Recruitment of fanconi anemia and breast cancer proteins to DNA damage sites is differentially governed by replication
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Recruitment of fanconi anemia ...... tially governed by replication
@ast
Recruitment of fanconi anemia ...... tially governed by replication
@en
type
label
Recruitment of fanconi anemia ...... tially governed by replication
@ast
Recruitment of fanconi anemia ...... tially governed by replication
@en
prefLabel
Recruitment of fanconi anemia ...... tially governed by replication
@ast
Recruitment of fanconi anemia ...... tially governed by replication
@en
P2093
P2860
P1433
P1476
Recruitment of fanconi anemia ...... tially governed by replication
@en
P2093
Johan P de Winter
Maria Tomasz
Stephen J Elledge
Weidong Wang
Woo-Hyun Chung
Yongjiang Li
P2860
P304
P356
10.1016/J.MOLCEL.2009.06.034
P577
2009-09-01T00:00:00Z