Degradation of host sphingomyelin is essential for Leishmania virulence.
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Role of Inositol Phosphosphingolipid Phospholipase C1, the Yeast Homolog of Neutral Sphingomyelinases in DNA Damage Response and DiseasesSphingolipid degradation in Leishmania (Leishmania) amazonensisSterol biosynthesis is required for heat resistance but not extracellular survival in leishmaniaDevelopmentally regulated sphingolipid degradation in Leishmania majorPhospholipid and sphingolipid metabolism in Leishmania.Hydroxyurea treatment inhibits proliferation of Cryptococcus neoformans in miceCharacterization of inositol phospho-sphingolipid-phospholipase C 1 (Isc1) in Cryptococcus neoformans reveals unique biochemical features.Identification of a sphingolipid-specific phospholipase D activity associated with the generation of phytoceramide-1-phosphate in cabbage leaves.Two major inositol transporters and their role in cryptococcal virulenceCharacterization of metabolically quiescent Leishmania parasites in murine lesions using heavy water labeling.Sphingolipid degradation by Leishmania major is required for its resistance to acidic pH in the mammalian host.Sphingolipid and ceramide homeostasis: potential therapeutic targets.Inositolphosphoceramide metabolism in Trypanosoma cruzi as compared with other trypanosomatids.Systems analysis of host-parasite interactions.Leishmania parasites possess a platelet-activating factor acetylhydrolase important for virulence.Plasmenylethanolamine synthesis in Leishmania major.Lipid metabolism in Trypanosoma bruceiSphingosine kinase A is a pleiotropic and essential enzyme for Leishmania survival and virulence.Subversion and Utilization of Host Innate Defense by Leishmania amazonensis.Endocytosis and Sphingolipid Scavenging in Leishmania mexicana Amastigotes.Changes in Lipid and Fatty Acid Composition During Intramacrophagic Transformation of Leishmania donovani Complex Promastigotes into Amastigotes.Lipidomics and anti-trypanosomatid chemotherapy.Comparative sphingolipidomics of disease-causing trypanosomatids reveal unique lifecycle- and taxonomy-specific lipid chemistries.Lipidomic alterations of in vitro macrophage infection by L. infantum and L. amazonensis.A complex interplay between sphingolipid and sterol metabolism revealed by perturbations to the Leishmania metabolome caused by miltefosine.The glycosomal alkyl-dihydroxyacetonephosphate synthase TbADS is essential for the synthesis of ether glycerophospholipids in procyclic trypanosomes.Expression, immunolocalization, and serological reactivity of a novel sphingomyelin phosphodiesterase-like protein, an excretory/secretory antigen from Clonorchis sinensis.
P2860
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P2860
Degradation of host sphingomyelin is essential for Leishmania virulence.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Degradation of host sphingomyelin is essential for Leishmania virulence.
@ast
Degradation of host sphingomyelin is essential for Leishmania virulence.
@en
Degradation of host sphingomyelin is essential for Leishmania virulence.
@nl
type
label
Degradation of host sphingomyelin is essential for Leishmania virulence.
@ast
Degradation of host sphingomyelin is essential for Leishmania virulence.
@en
Degradation of host sphingomyelin is essential for Leishmania virulence.
@nl
prefLabel
Degradation of host sphingomyelin is essential for Leishmania virulence.
@ast
Degradation of host sphingomyelin is essential for Leishmania virulence.
@en
Degradation of host sphingomyelin is essential for Leishmania virulence.
@nl
P2093
P2860
P921
P1433
P1476
Degradation of host sphingomyelin is essential for Leishmania virulence
@en
P2093
Fong-Fu Hsu
Lynn Soong
Mattie C Wilson
Phillip Key
Yingwei Wang
P2860
P304
P356
10.1371/JOURNAL.PPAT.1000692
P577
2009-12-11T00:00:00Z