Autoimmune disease classification by inverse association with SNP alleles. - Wikidata - wikimedia - TriplyDB

Autoimmune disease classification by inverse association with SNP alleles.

Autoimmune disease classification by inverse association with SNP alleles.

http://www.wikidata.org/entity/Q33521311

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Extreme evolutionary disparities seen in positive selection across seven complex diseases Sequencing and analysis of a South Asian-Indian personal genome Revealing the genetic basis of multiple sclerosis: are we there yet?Human leukocyte Antigen-DM polymorphisms in autoimmune diseases Knowledge-based analysis of genetic associations of rheumatoid arthritis to inform studies searching for pleiotropic genes: a literature review and network analysis Translational bioinformatics: linking the molecular world to the clinical world Pleiotropy in complex traits: challenges and strategies G-protein signaling modulator-3, a gene linked to autoimmune diseases, regulates monocyte function and its deficiency protects from inflammatory arthritis Leveraging models of cell regulation and GWAS data in integrative network-based association studies The emergence of human-evolutionary medical genomics Novel associations for hypothyroidism include known autoimmune risk loci Pervasive sharing of genetic effects in autoimmune disease.Ancient and recent selective pressures shaped genetic diversity at AIM2-like nucleic acid sensors.Family studies of type 1 diabetes reveal additive and epistatic effects between MGAT1 and three other polymorphisms Generating testable hypotheses for schizophrenia and rheumatoid arthritis pathogenesis by integrating epidemiological, genomic, and protein interaction data Balancing selection is common in the extended MHC region but most alleles with opposite risk profile for autoimmune diseases are neutrally evolving G protein signaling modulator-3: a leukocyte regulator of inflammation in health and disease Evidence for more than one Parkinson's disease-associated variant within the HLA region.A comprehensive analysis of shared loci between systemic lupus erythematosus (SLE) and sixteen autoimmune diseases reveals limited genetic overlap.Principal component analysis characterizes shared pathogenetics from genome-wide association studies.MHC-resident variation affects risks after unrelated donor hematopoietic cell transplantation.Volcano plots in analyzing differential expressions with mRNA microarrays.Integrative analysis of cancer prognosis data with multiple subtypes using regularized gradient descent.Disease risk factors identified through shared genetic architecture and electronic medical records Accounting for eXentricities: analysis of the X chromosome in GWAS reveals X-linked genes implicated in autoimmune diseases.Analysis of PPARGC1B, RUNX3 and TBKBP1 polymorphisms in Chinese Han patients with ankylosing spondylitis: a case-control study.Epistatic interaction between BANK1 and BLK in rheumatoid arthritis: results from a large trans-ethnic meta-analysis.Similarity of markers identified from cancer gene expression studies: observations from GEO PRIMe: a method for characterization and evaluation of pleiotropic regions from multiple genome-wide association studies.Joint GWAS Analysis: Comparing similar GWAS at different genomic resolutions identifies novel pathway associations with six complex diseases.Autoimmune risk loci of IL12RB2, IKZF1, XKR6, TMEM39A and CSK in Chinese patients with systemic lupus erythematosus.Assessment of the genetic basis of rosacea by genome-wide association study Sex differences in disease risk from reported genome-wide association study findings.Associations of Independent IL2RA Gene Variants with Intermediate Uveitis Complex-disease networks of trait-associated single-nucleotide polymorphisms (SNPs) unveiled by information theory.Integrated Analyses of Gene Expression Profiles Digs out Common Markers for Rheumatic Diseases.The autoimmune tautology: an in silico approach Identification of autoimmune gene signatures in autism.Emerging patterns of genetic overlap across autoimmune disorders.Shared HLA Class II in Six Autoimmune Diseases in Latin America: A Meta-Analysis

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Autoimmune disease classification by inverse association with SNP alleles.

http://www.wikidata.org/entity/Q33521311

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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type

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Autoimmune disease classification by inverse association with SNP alleles.

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Marc A Schaub

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Serafim Batzoglou

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William H Robinson

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P2860

Potential etiologic and functional implications of genome-wide association loci for human diseases and traits

The BTNL2 gene and sarcoidosis susceptibility in African Americans and Whites

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants

TRAF1-C5 as a risk locus for rheumatoid arthritis--a genomewide study

PLINK: a tool set for whole-genome association and population-based linkage analyses

Co-occurrence of autoimmune thyroid disease in a multiple sclerosis cohort.

A synonymous SNP of the corneodesmosin gene leads to increased mRNA stability and demonstrates association with psoriasis across diverse ethnic groups

Meta-analysis of genome-wide association data and large-scale replication identifies additional susceptibility loci for type 2 diabetes.

Common regulatory variation impacts gene expression in a cell type-dependent manner

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