A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3. - Wikidata - wikimedia - TriplyDB

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

http://www.wikidata.org/entity/Q33585268

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Mitochondrial Stat3, the Need for Design Thinking Genetic and epigenetic studies of atopic dermatitis Diminution of signal transducer and activator of transcription 3 signaling inhibits vascular permeability and anaphylaxis STAT3 restrains RANK- and TLR4-mediated signalling by suppressing expression of the E2 ubiquitin-conjugating enzyme Ubc13.Immunological loss-of-function due to genetic gain-of-function in humans: autosomal dominance of the third kind Mitochondrial Ca²⁺ and membrane potential, an alternative pathway for Interleukin 6 to regulate CD4 cell effector function Mechanisms of Jak/STAT signaling in immunity and disease The STAT3 HIES mutation is a gain-of-function mutation that activates genes via AGG-element carrying promoters The Ying and Yang of STAT3 in Human Disease Stat3/Cdc25a-dependent cell proliferation promotes embryonic axis extension during zebrafish gastrulation Multi-ancestry genome-wide association study of 21,000 cases and 95,000 controls identifies new risk loci for atopic dermatitis The JAK-STAT pathway: impact on human disease and therapeutic intervention STAT3 signaling in immunity.The Potential and Limits of Hematopoietic Stem Cell Transplantation for the Treatment of Autosomal Dominant Hyper-IgE Syndrome.A biallelic mutation in IL6ST encoding the GP130 co-receptor causes immunodeficiency and craniosynostosis.PD-L1 up-regulation restrains Th17 cell differentiation in STAT3 loss- and STAT1 gain-of-function patients.Protein stabilization improves STAT3 function in autosomal dominant hyper-IgE syndrome.CD8+ T cells produce a dialyzable antigen-specific activator of dendritic cells.An Inhibitory Role for the Transcription Factor Stat3 in Controlling IL-4 and Bcl6 Expression in Follicular Helper T Cells.Targeting JAK2 reduces GVHD and xenograft rejection through regulation of T cell differentiation.Interleukin-6/Stat3 signaling has an essential role in the host antimicrobial response to urinary tract infection.

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A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

http://www.wikidata.org/entity/Q33585268

description

2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մարտին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@ast

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@en

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@nl

type

label

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@ast

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

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A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

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prefLabel

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@ast

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@en

A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

@nl

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A mouse model of HIES reveals pro- and anti-inflammatory functions of STAT3.

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Alex Kotlyar

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Elizabeth A Wohlfert

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Fiona Powrie

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Golnaz Vahedi

http://www.w3.org/2001/XMLSchema#string

Hong-Wei Sun

http://www.w3.org/2001/XMLSchema#string

John J O'Shea

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Joshua D Milner

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Kan Jiang

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Lionel Feigenbaum

http://www.w3.org/2001/XMLSchema#string

Rafael Casellas

http://www.w3.org/2001/XMLSchema#string

P2860

Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells

Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality

Transforming growth factor-beta induces development of the T(H)17 lineage

STAT3 mutations in the hyper-IgE syndrome

Extreme hyperimmunoglobulinemia E and undue susceptibility to infection

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

Impaired T(H)17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation

Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

Jaks and STATs: biological implications

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Scott M Steward-Tharp

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