TRPC3 channels are necessary for brain-derived neurotrophic factor to activate a nonselective cationic current and to induce dendritic spine formation.
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Altered morphology of hippocampal dentate granule cell presynaptic and postsynaptic terminals following conditional deletion of TrkBInternational Union of Basic and Clinical Pharmacology. LXXVI. Current progress in the mammalian TRP ion channel familyHyperforin modulates dendritic spine morphology in hippocampal pyramidal neurons by activating Ca(2+) -permeable TRPC6 channelsTransmembrane proteoglycans control stretch-activated channels to set cytosolic calcium levels.Lavender oil-potent anxiolytic properties via modulating voltage dependent calcium channelsRat hypocretin/orexin neurons are maintained in a depolarized state by TRPC channelsBrain-derived neurotrophic factor activation of CaM-kinase kinase via transient receptor potential canonical channels induces the translation and synaptic incorporation of GluA1-containing calcium-permeable AMPA receptorsA role of canonical transient receptor potential 5 channel in neuronal differentiation from A2B5 neural progenitor cellsBdnf overexpression in hippocampal neurons prevents dendritic atrophy caused by Rett-associated MECP2 mutationsBrain-derived neurotrophic factor (BDNF)-induced mitochondrial motility arrest and presynaptic docking contribute to BDNF-enhanced synaptic transmission.Targeted delivery of brain-derived neurotrophic factor for the treatment of blindness and deafness.Inhibition of transient receptor potential canonical channels impairs cytokinesis in human malignant gliomas.BDNF evokes release of endogenous cannabinoids at layer 2/3 inhibitory synapses in the neocortexAn ultra-short dopamine pathway regulates basal ganglia output.Constitutively active TRPC3 channels regulate basal ganglia output neurons.BDNF induces calcium elevations associated with IBDNF, a nonselective cationic current mediated by TRPC channels.Activity-dependent release of endogenous BDNF from mossy fibers evokes a TRPC3 current and Ca2+ elevations in CA3 pyramidal neurons.The role of canonical transient receptor potential channels in seizure and excitotoxicityPhysiological Function and Characterization of TRPCs in NeuronsLeptin-induced spine formation requires TrpC channels and the CaM kinase cascade in the hippocampus.A simple role for BDNF in learning and memory?Dendritic spine dysgenesis in Rett syndrome.NMDA receptor-dependent synaptic activation of TRPC channels in olfactory bulb granule cellsA selective histone deacetylase-6 inhibitor improves BDNF trafficking in hippocampal neurons from Mecp2 knockout mice: implications for Rett syndrome.BDNF regulates the expression and distribution of vesicular glutamate transporters in cultured hippocampal neurons.Emerging roles of canonical TRP channels in neuronal function.Differential regulation of TRPC4 in the vasopressin magnocellular system by water deprivation and hepatic cirrhosis in the ratPostsynaptic TRPC1 function contributes to BDNF-induced synaptic potentiation at the developing neuromuscular junctionScreening of Transient Receptor Potential Canonical Channel Activators Identifies Novel Neurotrophic Piperazine Compounds.Distinct subsets of Syt-IV/BDNF vesicles are sorted to axons versus dendrites and recruited to synapses by activityHDAC activity is required for BDNF to increase quantal neurotransmitter release and dendritic spine density in CA1 pyramidal neuronsIntracellular Ca2+ stores and Ca2+ influx are both required for BDNF to rapidly increase quantal vesicular transmitter release.Activity-dependent BDNF release and TRPC signaling is impaired in hippocampal neurons of Mecp2 mutant miceMechanism of earthquake simulation as a prenatal stressor retarding rat offspring development and chinese medicine correcting the retardation: hormones and gene-expression alteration.The actions of BDNF on dendritic spine density and morphology in organotypic slice cultures depend on the presence of serum in culture media.BDNF - a key transducer of antidepressant effects.TRPM2 channels are required for NMDA-induced burst firing and contribute to H(2)O(2)-dependent modulation in substantia nigra pars reticulata GABAergic neuronsRole of the brain-derived neurotrophic factor at glutamatergic synapses.Physiology and pathophysiology of canonical transient receptor potential channelsTRPC3 channels are required for synaptic transmission and motor coordination.
P2860
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P2860
TRPC3 channels are necessary for brain-derived neurotrophic factor to activate a nonselective cationic current and to induce dendritic spine formation.
description
2007 nî lūn-bûn
@nan
2007 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@ast
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@en
type
label
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@ast
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@en
prefLabel
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@ast
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@en
P2860
P1476
TRPC3 channels are necessary f ...... uce dendritic spine formation.
@en
P2093
Michelle D Amaral
P2860
P304
P356
10.1523/JNEUROSCI.5499-06.2007
P407
P577
2007-05-01T00:00:00Z