P/Q Ca2+ channel blockade stops spreading depression and related pyramidal neuronal Ca2+ rise in hippocampal organ culture.
about
Abnormal synaptic Ca(2+) homeostasis and morphology in cortical neurons of familial hemiplegic migraine type 1 mutant miceGenetic and hormonal factors modulate spreading depression and transient hemiparesis in mouse models of familial hemiplegic migraine type 1.Spreading depression requires microglia and is decreased by their M2a polarization from environmental enrichment.The neurobiology of migraine.Cortical spreading depression-new insights and persistent questions.Characterization of inhibitory GABA-A receptor activation during spreading depolarization in brain slice.Strategies for study of neuroprotection from cold-preconditioningSynaptic release and extracellular actions of Zn2+ limit propagation of spreading depression and related events in vitro and in vivoThe Role of Negative Charge in the Delivery of Quantum Dots to Neurons.Nanoparticle targeting to neurons in a rat hippocampal slice culture model.Zn2+ influx is critical for some forms of spreading depression in brain slices.Optical current source density analysis in hippocampal organotypic culture shows that spreading depression occurs with uniquely reversing currents.Multiplexed cytokine protein expression profiles from spreading depression in hippocampal organotypic cultures.Contributions of Ca2+ and Zn2+ to spreading depression-like events and neuronal injury.Migraine is a neuronal disease.P/Q-type calcium channel modulators.The changing landscape of voltage-gated calcium channels in neurovascular disorders and in neurodegenerative diseasesRepetitive spreading depression-like events result in cell damage in juvenile hippocampal slice cultures maintained in normoxia.Gabapentin suppresses cortical spreading depression susceptibility.Changes in membrane potential and the intracellular calcium concentration during CSD and OGD in layer V and layer II/III mouse cortical neurons.Spreading depression: from serendipity to targeted therapy in migraine prophylaxis.Intranasally administered IGF-1 inhibits spreading depression in vivo.Novel Therapeutic Targets Against Spreading Depression.Intrinsic cellular and molecular properties of in vivo hippocampal synaptic plasticity are altered in the absence of key synaptic matrix molecules.Sustained NMDA receptor activation by spreading depolarizations can initiate excitotoxic injury in metabolically compromised neurons.
P2860
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P2860
P/Q Ca2+ channel blockade stops spreading depression and related pyramidal neuronal Ca2+ rise in hippocampal organ culture.
description
2004 nî lūn-bûn
@nan
2004 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@ast
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@en
type
label
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@ast
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@en
prefLabel
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@ast
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@en
P2860
P356
P1433
P1476
P/Q Ca2+ channel blockade stop ...... in hippocampal organ culture.
@en
P2093
Phillip E Kunkler
P2860
P304
P356
10.1002/HIPO.10181
P577
2004-01-01T00:00:00Z