Susceptibility to chronic infection with Toxoplasma gondii does not correlate with susceptibility to acute infection in mice.
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Mice infected with low-virulence strains of Toxoplasma gondii lose their innate aversion to cat urine, even after extensive parasite clearanceStructures of Toxoplasma gondii tachyzoites, bradyzoites, and sporozoites and biology and development of tissue cysts.Removal of Toxoplasma gondii cysts from the brain by perforin-mediated activity of CD8+ T cells.VCAM-1/α4β1 integrin interaction is crucial for prompt recruitment of immune T cells into the brain during the early stage of reactivation of chronic infection with Toxoplasma gondii to prevent toxoplasmic encephalitis.Experimental ocular toxoplasmosis in genetically susceptible and resistant mice.Expression of herpes simplex virus thymidine kinase in Toxoplasma gondii attenuates tachyzoite virulence in miceRequirement of non-T cells that produce gamma interferon for prevention of reactivation of Toxoplasma gondii infection in the brainPredominant interferon-γ-mediated expression of CXCL9, CXCL10, and CCL5 proteins in the brain during chronic infection with Toxoplasma gondii in BALB/c mice resistant to development of toxoplasmic encephalitis.Toxoplasma IgG and IgA, but not IgM, antibody titers increase in sera of immunocompetent mice in association with proliferation of tachyzoites in the brain during the chronic stage of infectionAntibody against interleukin-6 reduces inflammation and numbers of cysts in brains of mice with toxoplasmic encephalitis.Production of gamma interferon by natural killer cells from Toxoplasma gondii-infected SCID mice: regulation by interleukin-10, interleukin-12, and tumor necrosis factor alpha.Infection with Toxoplasma gondii reduces established and developing Th2 responses induced by Nippostrongylus brasiliensis infection.CXCL9 is important for recruiting immune T cells into the brain and inducing an accumulation of the T cells to the areas of tachyzoite proliferation to prevent reactivation of chronic cerebral infection with Toxoplasma gondii.Intranasal immunization with SAG1 protein of Toxoplasma gondii in association with cholera toxin dramatically reduces development of cerebral cysts after oral infection.Acute virulence in mice is associated with markers on chromosome VIII in Toxoplasma gondiiGamma interferon induces Fas-dependent apoptosis of Peyer's patch T cells in mice following peroral infection with Toxoplasma gondii.Toxoplasma encephalitis in congenic B10 and BALB mice: impact of genetic factors on the immune response.Kinetics and time dependence of the differential agglutination of acetone [AC]- and formalin [HS]-fixed Toxoplasma tachyzoites by serum of mice with experimental toxoplasmosisMitogen- and antigen-specific proliferation of T cells in murine toxoplasmosis is inhibited by reactive nitrogen intermediates.A comparative study between excretory/secretory and autoclaved vaccines against RH strain of Toxoplasma gondii in murine models.Toxoplasma gondii antigens recognized by IgG antibodies differ between mice with and without active proliferation of tachyzoites in the brain during the chronic stage of infection.Association of CD4+ T cell-dependent, interferon-gamma-mediated necrosis of the small intestine with genetic susceptibility of mice to peroral infection with Toxoplasma gondiiRole of cytokines and major histocompatibility complex restriction in mouse resistance to infection with a natural recombinant strain (type I-III) of Toxoplasma gondiiThe MIC3 gene of Toxoplasma gondii is a novel potent vaccine candidate against toxoplasmosisGamma interferon production, but not perforin-mediated cytolytic activity, of T cells is required for prevention of toxoplasmic encephalitis in BALB/c mice genetically resistant to the disease.IL-2 produced by CD8+ immune T cells can augment their IFN-γ production independently from their proliferation in the secondary response to an intracellular pathogen.12/15-lipoxygenase-dependent myeloid production of interleukin-12 is essential for resistance to chronic toxoplasmosis.Molecular markers of susceptibility to ocular toxoplasmosis, host and guest behaving badly.Toxoplasma gondii: Entry, association, and physiological influence on the central nervous system.
P2860
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P2860
Susceptibility to chronic infection with Toxoplasma gondii does not correlate with susceptibility to acute infection in mice.
description
1993 nî lūn-bûn
@nan
1993 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Susceptibility to chronic infe ...... ty to acute infection in mice.
@ast
Susceptibility to chronic infe ...... ty to acute infection in mice.
@en
type
label
Susceptibility to chronic infe ...... ty to acute infection in mice.
@ast
Susceptibility to chronic infe ...... ty to acute infection in mice.
@en
prefLabel
Susceptibility to chronic infe ...... ty to acute infection in mice.
@ast
Susceptibility to chronic infe ...... ty to acute infection in mice.
@en
P2093
P2860
P1476
Susceptibility to chronic infe ...... ty to acute infection in mice.
@en
P2093
F K Conley
J S Remington
M A Orellana
P2860
P304
P407
P577
1993-06-01T00:00:00Z