Removal of Toxoplasma gondii cysts from the brain by perforin-mediated activity of CD8+ T cells.
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Interferon-gamma- and perforin-mediated immune responses for resistance against Toxoplasma gondii in the brainMicroglia development and functionRecombinant vaccines against T. gondii: comparison between homologous and heterologous vaccination protocols using two viral vectors expressing SAG1Chitinase dependent control of protozoan cyst burden in the brainAnalysis of behavior and trafficking of dendritic cells within the brain during toxoplasmic encephalitisCharacteristics and critical function of CD8+ T cells in the Toxoplasma-infected brain.The protein kinase double-stranded RNA-dependent (PKR) enhances protection against disease cause by a non-viral pathogen.The CD8 T-cell road to immunotherapy of toxoplasmosis.VCAM-1/α4β1 integrin interaction is crucial for prompt recruitment of immune T cells into the brain during the early stage of reactivation of chronic infection with Toxoplasma gondii to prevent toxoplasmic encephalitis.Toxoplasma IgG and IgA, but not IgM, antibody titers increase in sera of immunocompetent mice in association with proliferation of tachyzoites in the brain during the chronic stage of infectionTowards an immunosense vaccine to prevent toxoplasmosis: protective Toxoplasma gondii epitopes restricted by HLA-A*0201CD8 T Cells and Toxoplasma gondii: A New ParadigmEvidence for finely-regulated asynchronous growth of Toxoplasma gondii cysts based on data-driven model selection.MyD88-dependent protective immunity elicited by adenovirus 5 expressing the surface antigen 1 from Toxoplasma gondii is mediated by CD8(+) T lymphocytes.Toxoplasma gondii inhibits granzyme B-mediated apoptosis by the inhibition of granzyme B function in host cellsToxoplasma gondii superinfection and virulence during secondary infection correlate with the exact ROP5/ROP18 allelic combination.Protective efficacy of recombinant canine adenovirus type-2 expressing TgROP18 (CAV-2-ROP18) against acute and chronic Toxoplasma gondii infection in mice.Immune aging, dysmetabolism, and inflammation in neurological diseases.Mechanisms of Toxoplasma gondii persistence and latency.Novel Approaches Reveal that Toxoplasma gondii Bradyzoites within Tissue Cysts Are Dynamic and Replicating Entities In VivoPD-1-mediated attrition of polyfunctional memory CD8+ T cells in chronic toxoplasma infectionToxoplasma gondii antigens recognized by IgG antibodies differ between mice with and without active proliferation of tachyzoites in the brain during the chronic stage of infection.CD73-generated adenosine facilitates Toxoplasma gondii differentiation to long-lived tissue cysts in the central nervous system.CD8(+) T cells remove cysts of Toxoplasma gondii from the brain mostly by recognizing epitopes commonly expressed by or cross-reactive between type II and type III strains of the parasiteDonor CD8+ T cells prevent Toxoplasma gondii de-encystation but fail to rescue the exhausted endogenous CD8+ T cell population.IL-7 and IL-15 do not synergize during CD8 T cell recall response against an obligate intracellular parasite.Toxoplasmosis in the fetus and newborn: an update on prevalence, diagnosis and treatment.Complex immune cell interplay in the gamma interferon response during Toxoplasma gondii infectionDynamic two-photon imaging of the immune response to Toxoplasma gondii infection.CD8+ T cell immunity in an encephalitis model of Toxoplasma gondii infection.Hypometabolism as the ultimate defence in stress response: how the comparative approach helps understanding of medically relevant questions.Molecular and virulence characterization of Toxoplasma gondii strains isolated from humans in Portugal.Determination of a Key Antigen for Immunological Intervention To Target the Latent Stage of Toxoplasma gondii.In vivo effect of anti-TNF agent (etanercept) in reactivation of latent toxoplasmosis.A Bradyzoite is a Bradyzoite is a Bradyzoite?Induction of IL-10-producing regulatory B cells following Toxoplasma gondii infection is important to the cyst formation.Inducible nitric oxide synthase in innate immune cells is important for restricting cyst formation of Toxoplasma gondii in the brain but not required for the protective immune process to remove the cysts.Evaluation of the adjuvant effect of pidotimod on the immune protection induced by UV-attenuated Toxoplasma gondii in mouse models.ToxoplasmosisPD-L1, TIM-3, and CTLA-4 blockade fail to promote resistance to secondary infection with virulent strains of
P2860
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P2860
Removal of Toxoplasma gondii cysts from the brain by perforin-mediated activity of CD8+ T cells.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Removal of Toxoplasma gondii c ...... ated activity of CD8+ T cells.
@ast
Removal of Toxoplasma gondii c ...... ated activity of CD8+ T cells.
@en
type
label
Removal of Toxoplasma gondii c ...... ated activity of CD8+ T cells.
@ast
Removal of Toxoplasma gondii c ...... ated activity of CD8+ T cells.
@en
prefLabel
Removal of Toxoplasma gondii c ...... ated activity of CD8+ T cells.
@ast
Removal of Toxoplasma gondii c ...... ated activity of CD8+ T cells.
@en
P2093
P2860
P1476
Removal of Toxoplasma gondii c ...... iated activity of CD8+ T cells
@en
P2093
Benard S Jortner
Sara A Michie
Sara Perkins
Tomoya Kudo
Xisheng Wang
Yasuhiro Suzuki
P2860
P304
P356
10.2353/AJPATH.2010.090825
P407
P50
P577
2010-02-18T00:00:00Z