Does inhibition of tumor necrosis factor alpha affect chlamydial genital tract infection in mice and guinea pigs?
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Role for inducible nitric oxide synthase in protection from chronic Chlamydia trachomatis urogenital disease in mice and its regulation by oxygen free radicalsTwo different homing pathways involving integrin β7 and E-selectin significantly influence trafficking of CD4 cells to the genital tract following Chlamydia muridarum infection.Early local cytokine profiles in strains of mice with different outcomes from chlamydial genital tract infectionMouse strain-dependent chemokine regulation of the genital tract T helper cell type 1 immune response.Expression of matrix metalloproteinases subsequent to urogenital Chlamydia muridarum infection of miceChemokine expression patterns differ within anatomically distinct regions of the genital tract during Chlamydia trachomatis infection.Differences in innate immune responses (in vitro) to HeLa cells infected with nondisseminating serovar E and disseminating serovar L2 of Chlamydia trachomatis.Immunity to murine chlamydial genital infection.Host nectin-1 is required for efficient Chlamydia trachomatis serovar E development.Cellular immunity and Chlamydia genital infection: induction, recruitment, and effector mechanisms.Tumor necrosis factor alpha production from CD8+ T cells mediates oviduct pathological sequelae following primary genital Chlamydia muridarum infectionVaginal chlamydial clearance following primary or secondary infection in mice occurs independently of TNF-α.In infertile women, cells from Chlamydia trachomatis infected sites release higher levels of interferon-gamma, interleukin-10 and tumor necrosis factor-alpha upon heat-shock-protein stimulation than fertile womenHost immune responses to chlamydial inclusion membrane proteins B and C in Chlamydia trachomatis infected women with or without fertility disordersThe guinea pig as a model of infectious diseases.The Predominant CD4+ Th1 Cytokine Elicited to Chlamydia trachomatis Infection in Women Is Tumor Necrosis Factor Alpha and Not Interferon Gamma.The TNF-α-308G/A Polymorphism is Not Associated with Ocular Chlamydia trachomatis Infection in Han Chinese Children.Differences in innate immune responses correlate with differences in murine susceptibility to Chlamydia muridarum pulmonary infection.Immunization with chlamydial plasmid protein pORF5 DNA vaccine induces protective immunity against genital chlamydial infection in mice.Cytokine expression pattern in the genital tract of Chlamydia trachomatis positive infertile women - implication for T-cell responses.Role of Bcl-2 family members in caspase-independent apoptosis during Chlamydia infection.Toll-like receptor 3 (TLR3) promotes the resolution of Chlamydia muridarum genital tract infection in congenic C57BL/6N mice.
P2860
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P2860
Does inhibition of tumor necrosis factor alpha affect chlamydial genital tract infection in mice and guinea pigs?
description
2000 nî lūn-bûn
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2000 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի սեպտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
@zh-hk
2000年論文
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2000年論文
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2000年论文
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Does inhibition of tumor necro ...... ction in mice and guinea pigs?
@ast
Does inhibition of tumor necro ...... ction in mice and guinea pigs?
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type
label
Does inhibition of tumor necro ...... ction in mice and guinea pigs?
@ast
Does inhibition of tumor necro ...... ction in mice and guinea pigs?
@en
prefLabel
Does inhibition of tumor necro ...... ction in mice and guinea pigs?
@ast
Does inhibition of tumor necro ...... ction in mice and guinea pigs?
@en
P2093
P2860
P1476
Does inhibition of tumor necro ...... ction in mice and guinea pigs?
@en
P2093
P2860
P304
P356
10.1128/IAI.68.9.5299-5305.2000
P407
P577
2000-09-01T00:00:00Z