Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
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The Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged SwordInflammasomes and Their Role in Innate Immunity of Sexually Transmitted InfectionsInnate immunity in ocular Chlamydia trachomatis infection: contribution of IL8 and CSF2 gene variants to risk of trachomatous scarring in GambiansHost chemokine and cytokine response in the endocervix within the first developmental cycle of Chlamydia muridarum.Does inhibition of tumor necrosis factor alpha affect chlamydial genital tract infection in mice and guinea pigs?A MyD88-dependent early IL-17 production protects mice against airway infection with the obligate intracellular pathogen Chlamydia muridarum.Manipulation of Host Cholesterol by Obligate Intracellular Bacteria.Neither interleukin-6 nor inducible nitric oxide synthase is required for clearance of Chlamydia trachomatis from the murine genital tract epithelium.Neutrophils aid in protection of the vaginal mucosae of immune mice against challenge with herpes simplex virus type 2.Role of polymorphonuclear neutrophils in a murine model of Chlamydia psittaci-induced abortion.Lack of long-lasting hydrosalpinx in A/J mice correlates with rapid but transient chlamydial ascension and neutrophil recruitment in the oviduct following intravaginal inoculation with Chlamydia muridarum.Neutrophils play a protective nonphagocytic role in systemic Mycobacterium tuberculosis infection of micePolymorphonuclear neutrophils are necessary for the recruitment of CD8(+) T cells in the liver in a pregnant mouse model of Chlamydophila abortus (Chlamydia psittaci serotype 1) infection.In situ analysis of the evolution of the primary immune response in murine Chlamydia trachomatis genital tract infection.Early local cytokine profiles in strains of mice with different outcomes from chlamydial genital tract infectionNeutrophil depletion during Toxoplasma gondii infection leads to impaired immunity and lethal systemic pathology.Chlamydiae and polymorphonuclear leukocytes: unlikely allies in the spread of chlamydial infectionThe contribution of interleukin-12/interferon-gamma axis in protection against neonatal pulmonary Chlamydia muridarum challengeHuman conjunctival transcriptome analysis reveals the prominence of innate defense in Chlamydia trachomatis infectionEssential role for neutrophils in pathogenesis and adaptive immunity in Chlamydia caviae ocular infectionsThe hypothetical protein CT813 is localized in the Chlamydia trachomatis inclusion membrane and is immunogenic in women urogenitally infected with C. trachomatis.A chlamydial type III-secreted effector protein (Tarp) is predominantly recognized by antibodies from humans infected with Chlamydia trachomatis and induces protective immunity against upper genital tract pathologies in mice.Protective immunity against mouse upper genital tract pathology correlates with high IFNγ but low IL-17 T cell and anti-secretion protein antibody responses induced by replicating chlamydial organisms in the airway.Plac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tractCaspase-1 contributes to Chlamydia trachomatis-induced upper urogenital tract inflammatory pathologies without affecting the course of infection.Chlamydial infection and spatial ascension of the female genital tract: a novel hybrid cellular automata and continuum mathematical model.The infecting dose of Chlamydia muridarum modulates the innate immune response and ascending infection.Genital Chlamydia trachomatis: understanding the roles of innate and adaptive immunity in vaccine research.Chlamydia trachomatis Cellular Exit Alters Interactions with Host Dendritic CellsChlamydia trachomatis growth inhibition and restoration of LDL-receptor level in HepG2 cells treated with mevastatin.Genetic profiling of dendritic cells exposed to live- or ultraviolet-irradiated Chlamydia muridarum reveals marked differences in CXC chemokine profiles.Intranasal inoculation of Chlamydia trachomatis mouse pneumonitis agent induces significant neutrophil infiltration which is not efficient in controlling the infection in mice.Rapid, transient phosphatidylserine externalization induced in host cells by infection with Chlamydia spp.A role for interleukin-6 in host defense against murine Chlamydia trachomatis infection.Nonspecific immune responses and mechanisms of resistance to Eimeria papillata infections in mice.Chlamydia Spreading from the Genital Tract to the Gastrointestinal Tract - A Two-Hit Hypothesis.
P2860
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P2860
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
description
1996 nî lūn-bûn
@nan
1996 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@ast
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@en
type
label
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@ast
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@en
prefLabel
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@ast
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@en
P2093
P2860
P1476
Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.
@en
P2093
E M Peterson
L M de la Maza
N Barteneva
P2860
P304
P407
P577
1996-11-01T00:00:00Z