Herpes simplex virus 1 open reading frames O and P are not necessary for establishment of latent infection in mice. - Wikidata - wikimedia - TriplyDB

Herpes simplex virus 1 open reading frames O and P are not necessary for establishment of latent infection in mice.

Herpes simplex virus 1 open reading frames O and P are not necessary for establishment of latent infection in mice.

http://www.wikidata.org/entity/Q33604367

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The Role of microRNAs in the Pathogenesis of Herpesvirus Infection Antisense transcription in the human cytomegalovirus transcriptome.An antisense transcript in the human cytomegalovirus UL87 gene region.Neither LAT nor open reading frame P mutations increase expression of spliced or intron-containing ICP0 transcripts in mouse ganglia latently infected with herpes simplex virus Characterization of a novel group of antisense transcripts in human cytomegalovirus UL83 gene region.Characterization of herpes simplex virus 2 primary microRNA Transcript regulation The herpes simplex virus type 1 BgKL variant, unlike the BgOL variant, shows a higher association with orolabial infection than with infections at other sites, supporting the variant-dispersion-replacement hypothesis.The miRNAs of herpes simplex virus (HSV).

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P2860

Herpes simplex virus 1 open reading frames O and P are not necessary for establishment of latent infection in mice.

http://www.wikidata.org/entity/Q33604367

description

2000 nî lūn-bûn

@nan

2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2000年の論文

@ja

2000年論文

@yue

2000年論文

@zh-hant

2000年論文

@zh-hk

2000年論文

@zh-mo

2000年論文

@zh-tw

2000年论文

@wuu

name

Herpes simplex virus 1 open re ...... t of latent infection in mice.

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Herpes simplex virus 1 open re ...... t of latent infection in mice.

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type

label

Herpes simplex virus 1 open re ...... t of latent infection in mice.

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Herpes simplex virus 1 open re ...... t of latent infection in mice.

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Herpes simplex virus 1 open re ...... t of latent infection in mice.

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Herpes simplex virus 1 open re ...... t of latent infection in mice.

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P356

JVI.74.19.9019-9027.2000

P1433

Journal of Virology

P1476

Herpes simplex virus 1 open re ...... t of latent infection in mice.

@en

P2093

M Lagunoff

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P2860

Herpes simplex virus latency-associated transcript is a stable intron

Regulation of herpesvirus macromolecular synthesis. I. Cascade regulation of the synthesis of three groups of viral proteins

Herpes simplex virus type 1 latency-associated transcripts are evidently not essential for latent infection.

Regulation of alpha genes of herpes simplex virus: expression of chimeric genes produced by fusion of thymidine kinase with alpha gene promoters.

A generalized technique for deletion of specific genes in large genomes: alpha gene 22 of herpes simplex virus 1 is not essential for growth.

Regulation of herpesvirus macromolecular synthesis: sequential transition of polypeptide synthesis requires functional viral polypeptides.

Association of the herpes simplex virus regulatory protein ICP4 with specific nucleotide sequences in DNA.

Alpha 4, the major regulatory protein of herpes simplex virus type 1, is stably and specifically associated with promoter-regulatory domains of alpha genes and of selected other viral genes.

Replication, establishment of latency, and induced reactivation of herpes simplex virus gamma 1 34.5 deletion mutants in rodent models.

Quantification of transcripts from the ICP4 and thymidine kinase genes in mouse ganglia latently infected with herpes simplex virus

P304

9019-9027

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scholarly article

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10.1128/JVI.74.19.9019-9027.2000

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19

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P478

74

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Bernard Roizman

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2000-10-01T00:00:00Z

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10982346

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102098

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