IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
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The Innate Immune System in Acute and Chronic WoundsDifferential effect of Platelet Endothelial Cell Adhesion Molecule-1 (PECAM-1) on leukocyte infiltration during contact hypersensitivity responses.Chitinase-like proteins promote IL-17-mediated neutrophilia in a tradeoff between nematode killing and host damage.Vitamin D up-regulates the vitamin D receptor by protecting it from proteasomal degradation in human CD4+ T cells.Midline 1 controls polarization and migration of murine cytotoxic T cells.NKG2D-dependent activation of dendritic epidermal T cells in contact hypersensitivity.Dermal γδ T Cells Do Not Freely Re-Circulate Out of Skin and Produce IL-17 to Promote Neutrophil Infiltration during Primary Contact HypersensitivityOzone-induced IL-17A and neutrophilic airway inflammation is orchestrated by the caspase-1-IL-1 cascade.Development of interleukin-17-producing Vγ2+ γδ T cells is reduced by ICOS signaling in the thymus.Multiple Receptor-Ligand Interactions Direct Tissue-Resident γδ T Cell Activation.Emerging Skin T-Cell Functions in Response to Environmental InsultsImmunological, chemical and clinical aspects of exposure to mixtures of contact allergens.CD3bright signals on γδ T cells identify IL-17A-producing Vγ6Vδ1+ T cells.γδ T cells in homeostasis and host defence of epithelial barrier tissues.Arginase1 Deficiency in Monocytes/Macrophages Upregulates Inducible Nitric Oxide Synthase To Promote Cutaneous Contact Hypersensitivity.Occludin Expression in Epidermal γδ T Cells in Response to Epidermal Stress Causes Them To Migrate into Draining Lymph Nodes.Rapid allergen-induced interleukin-17 and interferon-γ secretion by skin-resident memory CD8+ T cells.Vγ4 T Cells Inhibit the Pro-healing Functions of Dendritic Epidermal T Cells to Delay Skin Wound Closure Through IL-17A.Functions of Vγ4 T Cells and Dendritic Epidermal T Cells on Skin Wound Healing.Recent advances in understanding and managing contact dermatitis.Detection of local inflammation induced by repeated exposure to contact allergens by use of IVIS SpectrumCT analysesEpidermal filaggrin deficiency mediates increased systemic T-helper 17 immune response
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P2860
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
description
2014 nî lūn-bûn
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2014 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մարտին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@ast
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@en
type
label
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@ast
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@en
prefLabel
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@ast
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@en
P2093
P2860
P50
P356
P1476
IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.
@en
P2093
Amanda S MacLeod
Deborah A Witherden
Morten M Nielsen
Niels Ødum
Paola Lovato
P2860
P304
P356
10.4049/JIMMUNOL.1301689
P407
P50
P577
2014-03-05T00:00:00Z