Cutting edge: NKT cells constitutively express IL-23 receptor and RORgammat and rapidly produce IL-17 upon receptor ligation in an IL-6-independent fashion. - Wikidata - wikimedia - TriplyDB

Cutting edge: NKT cells constitutively express IL-23 receptor and RORgammat and rapidly produce IL-17 upon receptor ligation in an IL-6-independent fashion.

Cutting edge: NKT cells constitutively express IL-23 receptor and RORgammat and rapidly produce IL-17 upon receptor ligation in an IL-6-independent fashion.

http://www.wikidata.org/entity/Q34590084

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The role of NKT cells in tumor immunity Interleukin-22 Signaling in the Regulation of Intestinal Health and Disease Regulation of IL-17 in autoimmune diseases by transcriptional factors and microRNAs Regulation of Dendritic Cell Function in Inflammation Immunovirological parameters and cytokines in HIV infection Fine tuning a well-oiled machine: Influence of NK1.1 and NKG2D on NKT cell development and function Positive or negative involvement of heat shock proteins in multiple sclerosis pathogenesis: an overview Stages versus subsets: Invariant Natural Killer T cell lineage differentiation Oropharyngeal Candidiasis in HIV Infection: Analysis of Impaired Mucosal Immune Response to Candida albicans in Mice Expressing the HIV-1 Transgene Cytokine-induced cytokine production by conventional and innate lymphoid cells Diversity of IL-17-producing T lymphocytes Influence of maternal immunization with allergens on the thymic maturation of lymphocytes with regulatory potential in children: a broad field for further exploration Th22 cells in allergic disease TLR4 and NKT cell synergy in immunotherapy against visceral leishmaniasis Invariant NKT cells modulate the suppressive activity of IL-10-secreting neutrophils differentiated with serum amyloid A Identification of Baicalin as an immunoregulatory compound by controlling T(H)17 cell differentiation Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk Galectin-9 prolongs the survival of septic mice by expanding Tim-3-expressing natural killer T cells and PDCA-1+ CD11c+ macrophages NKT cell stimulation with α-galactosylceramide results in a block of Th17 differentiation after intranasal immunization in mice Inflammatory bowel disease NOX2 Activation of Natural Killer T Cells Is Blocked by the Adenosine A2A Receptor to Inhibit Lung Ischemia-Reperfusion Injury IL-17A controls IL-17F production and maintains blood neutrophil counts in mice.IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.TLR signals promote IL-6/IL-17-dependent transplant rejection.A comprehensive ex vivo functional analysis of human NKT cells reveals production of MIP1-α and MIP1-β, a lack of IL-17, and a Th1-bias in males Microbes, the gut and ankylosing spondylitis.Aging, imbalanced inflammation and viral infection Mechanisms of neutrophil accumulation in the lungs against bacteria.Cytokine dependent and independent iNKT cell activation The blessings and curses of intestinal inflammation.Secretion of IFN-gamma but not IL-17 by CD1d-restricted NKT cells enhances rejection of skin grafts expressing epithelial cell-derived antigen.Targeting PI3Kγ activity decreases vascular trauma-induced intimal hyperplasia through modulation of the Th1 response.Distinct and overlapping effector functions of expanded human CD4+, CD8α+ and CD4-CD8α- invariant natural killer T cells Development and function of invariant natural killer T cells producing T(h)2- and T(h)17-cytokines Discovery and mechanism of ustekinumab: a human monoclonal antibody targeting interleukin-12 and interleukin-23 for treatment of immune-mediated disorders.Characterization of autoreactive and bystander IL-17+ T cells induced in immunized C57BL/6 mice.Cytokine requirements for the differentiation and expansion of IL-17A- and IL-22-producing human Vgamma2Vdelta2 T cells.Protective role of interleukin-17 in murine NKT cell-driven acute experimental hepatitis.Loss of effector and anti-inflammatory natural killer T lymphocyte function in pathogenic simian immunodeficiency virus infection Activation of Invariant NKT cells with glycolipid ligand α-galactosylceramide ameliorates glucose-6-phosphate isomerase peptide-induced arthritis

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Cutting edge: NKT cells constitutively express IL-23 receptor and RORgammat and rapidly produce IL-17 upon receptor ligation in an IL-6-independent fashion.

http://www.wikidata.org/entity/Q34590084

description

2008 nî lūn-bûn

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2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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type

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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JIMMUNOL.180.8.5167

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Cutting edge: NKT cells consti ...... n an IL-6-independent fashion.

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Aleksandra V Rachitskaya

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Anna M Hansen

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Dror Luger

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Rafael Villasmil

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Reiko Horai

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Zhuqing Li

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P2860

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Transforming growth factor-beta induces development of the T(H)17 lineage

IL-23 promotes tumour incidence and growth

The biology of NKT cells

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways

IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

Identification of an IL-17-producing NK1.1(neg) iNKT cell population involved in airway neutrophilia.

Treatment with a neutralizing anti-murine interleukin-17 antibody after the onset of collagen-induced arthritis reduces joint inflammation, cartilage destruction, and bone erosion.

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5167-5171

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scholarly article

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10.4049/JIMMUNOL.180.8.5167

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8

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180

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Robert B Nussenblatt

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2008-04-01T00:00:00Z

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269959597

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18390697

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2442579

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