Unfolded protein response in fuchs endothelial corneal dystrophy: a unifying pathogenic pathway? - Wikidata - wikimedia - TriplyDB

Unfolded protein response in fuchs endothelial corneal dystrophy: a unifying pathogenic pathway?

Unfolded protein response in fuchs endothelial corneal dystrophy: a unifying pathogenic pathway?

http://www.wikidata.org/entity/Q33617806

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Characteristics of corneal dystrophies: a review from clinical, histological and genetic perspectives Fuchs endothelial corneal dystrophy: current perspectives The divergence, actions, roles, and relatives of sodium-coupled bicarbonate transporters N-Acetylcysteine increases corneal endothelial cell survival in a mouse model of Fuchs endothelial corneal dystrophy Alterations in the aqueous humor proteome in patients with Fuchs endothelial corneal dystrophy.A common trinucleotide repeat expansion within the transcription factor 4 (TCF4, E2-2) gene predicts Fuchs corneal dystrophy.A cellular model for the investigation of Fuchs' endothelial corneal dystrophy.On the origin of 'guttae'Molecular bases of corneal endothelial dystrophies.An alpha 2 collagen VIII transgenic knock-in mouse model of Fuchs endothelial corneal dystrophy shows early endothelial cell unfolded protein response and apoptosis Impact of Corneal Endothelial Dysfunctions on Intraocular Oxygen Levels in Human Eyes Decline in DJ-1 and decreased nuclear translocation of Nrf2 in Fuchs endothelial corneal dystrophy.Screening and Characterization of Drugs That Protect Corneal Endothelial Cells Against Unfolded Protein Response and Oxidative Stress Endothelial Cdkn1a (p21) overexpression and accelerated senescence in a mouse model of Fuchs endothelial corneal dystrophy Tissue microarray analysis of cyclin-dependent kinase inhibitors p21 and p16 in Fuchs dystrophy Keratin 12 missense mutation induces the unfolded protein response and apoptosis in Meesmann epithelial corneal dystrophy.Endothelial cell whole genome expression analysis in a mouse model of early-onset Fuchs' endothelial corneal dystrophy.The genetics of Fuchs' corneal dystrophy L450W and Q455K Col8a2 knock-in mouse models of Fuchs endothelial corneal dystrophy show distinct phenotypes and evidence for altered autophagy.In Vivo Imaging of Corneal Endothelial Dystrophy in Boston Terriers: A Spontaneous, Canine Model for Fuchs' Endothelial Corneal Dystrophy.Lithium treatment increases endothelial cell survival and autophagy in a mouse model of Fuchs endothelial corneal dystrophy.Association and familial segregation of CTG18.1 trinucleotide repeat expansion of TCF4 gene in Fuchs' endothelial corneal dystrophy.Endothelial cell microRNA expression in human late-onset Fuchs' dystrophy.Fuchs endothelial corneal dystrophy in patients with myotonic dystrophy: a case series Gender specific issues in hereditary ocular disorders.Activation of mitophagy leads to decline in Mfn2 and loss of mitochondrial mass in Fuchs endothelial corneal dystrophy.MicroRNA-29b Overexpression Decreases Extracellular Matrix mRNA and Protein Production in Human Corneal Endothelial Cells.Involvement of ZEB1 and Snail1 in excessive production of extracellular matrix in Fuchs endothelial corneal dystrophy.What does the future hold for the treatment of Fuchs endothelial dystrophy; will 'keratoplasty' still be a valid procedure?Corneal Backscatters as an Objective Index for Assessing Fuchs' Endothelial Corneal Dystrophy: A Pilot Study.Loss of ion transporters and increased unfolded protein response in Fuchs' dystrophy.Activation of TGF-β signaling induces cell death via the unfolded protein response in Fuchs endothelial corneal dystrophy.The unfolded protein response in human corneal endothelial cells following hypothermic storage: implications of a novel stress pathway Fuchs endothelial corneal dystrophy: a neurodegenerative disorder?Changes in lipidomic profile of aqueous humour in Fuchs endothelial dystrophy.Repeat-Associated Non-ATG (RAN) Translation in Fuchs' Endothelial Corneal Dystrophy.Perspective of Future Potent Therapies for Fuchs Endothelial Corneal Dystrophy Changes in Surface Tension of Aqueous Humor in Anterior Segment Ocular Pathologies

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P2860

Unfolded protein response in fuchs endothelial corneal dystrophy: a unifying pathogenic pathway?

http://www.wikidata.org/entity/Q33617806

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Unfolded protein response in f ...... a unifying pathogenic pathway?

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Unfolded protein response in f ...... a unifying pathogenic pathway?

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Unfolded protein response in f ...... a unifying pathogenic pathway?

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Unfolded protein response in f ...... a unifying pathogenic pathway?

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Unfolded protein response in f ...... a unifying pathogenic pathway?

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J.AJO.2009.09.009

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American Journal of Ophthalmology

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Unfolded protein response in f ...... a unifying pathogenic pathway?

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Albert S Jun

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Arielle R Spitze

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Caroline L Speck

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Christoph Engler

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Clare Kelliher

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P2860

An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12

Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Abeta-induced cell death

Immunohistochemistry and electron microscopy of early-onset fuchs corneal dystrophy in three cases with the same L450W COL8A2 mutation

Signal integration in the endoplasmic reticulum unfolded protein response

Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

Mediators of endoplasmic reticulum stress-induced apoptosis

IRE1 signaling affects cell fate during the unfolded protein response

The presence of malfolded proteins in the endoplasmic reticulum signals the induction of glucose-regulated proteins

Overexpression of myocilin in the Drosophila eye activates the unfolded protein response: implications for glaucoma.

Missense mutations in COL8A2, the gene encoding the alpha2 chain of type VIII collagen, cause two forms of corneal endothelial dystrophy.

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Charles G Eberhart

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