Mu-opioid receptors transiently activate the Akt-nNOS pathway to produce sustained potentiation of PKC-mediated NMDAR-CaMKII signaling.
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Zinc and the modulation of redox homeostasisNitric oxide synthase dysfunction contributes to impaired cerebroarteriolar reactivity in experimental cerebral malariaProtein kinase D interacts with neuronal nitric oxide synthase and phosphorylates the activatory residue serine 1412CaMKIIα may modulate fentanyl-induced hyperalgesia via a CeLC-PAG-RVM-spinal cord descending facilitative pain pathway in ratsThe neural mobilization technique modulates the expression of endogenous opioids in the periaqueductal gray and improves muscle strength and mobility in rats with neuropathic painBrain-derived neurotrophic factor exerts neuroprotective actions against amyloid β-induced apoptosis in neuroblastoma cells.Analgesic tone conferred by constitutively active mu opioid receptors in mice lacking β-arrestin 2Estradiol induces partial desensitization of serotonin 1A receptor signaling in the paraventricular nucleus of the hypothalamus and alters expression and interaction of RGSZ1 and GαzChronic oxycodone induces integrated stress response in rat brain.Nitric oxide and zinc-mediated protein assemblies involved in mu opioid receptor signaling.Nitric Oxide and Hydrogen Sulfide Regulation of Ischemic Vascular Remodeling.Psychostimulant-Induced Endoplasmic Reticulum Stress and Neurodegeneration.Opioids Resistance in Chronic Pain Management.Tanshinone IIA protects PC12 cells from β-amyloid(25-35)-induced apoptosis via PI3K/Akt signaling pathway.Calmodulin kinase IV-dependent CREB activation is required for neuroprotection via NMDA receptor-PSD95 disruption.The mu-opioid receptor and the NMDA receptor associate in PAG neurons: implications in pain control.Spinal Neuronal NOS Signaling Contributes to Morphine Cardioprotection in Ischemia Reperfusion Injury in Rats.Morphine induces hyperalgesia without involvement of μ-opioid receptor or morphine-3-glucuronide.NO-released zinc supports the simultaneous binding of Raf-1 and PKCγ cysteine-rich domains to HINT1 protein at the mu-opioid receptor.Stimulation of peripheral kappa opioid receptors inhibits inflammatory hyperalgesia via activation of the PI3Kγ/AKT/nNOS/NO signaling pathway.The histidine triad nucleotide-binding protein 1 supports mu-opioid receptor-glutamate NMDA receptor cross-regulation.cAMP-dependent post-translational modification of neuronal nitric oxide synthase neuroprotects penile erection in rats.
P2860
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P2860
Mu-opioid receptors transiently activate the Akt-nNOS pathway to produce sustained potentiation of PKC-mediated NMDAR-CaMKII signaling.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@ast
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@en
type
label
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@ast
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@en
prefLabel
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@ast
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@en
P2860
P50
P1433
P1476
Mu-opioid receptors transientl ...... diated NMDAR-CaMKII signaling.
@en
P2093
Pilar Sánchez-Blázquez
P2860
P304
P356
10.1371/JOURNAL.PONE.0011278
P407
P577
2010-06-23T00:00:00Z