Formation of stable transcription complexes as assayed by analysis of individual templates.
about
Stochasticity in transcriptional regulation: origins, consequences, and mathematical representationsCell signaling can direct either binary or graded transcriptional responsesRegulatory T cells: mechanisms of differentiation and functionMetazoan rDNA enhancer acts by making more genes transcriptionally active.Quantitative modeling of stochastic systems in molecular biology by using stochastic Petri nets.Modeling stochastic gene expression: implications for haploinsufficiency.Conservation of the primary structure, organization, and function of the human and mouse beta-globin locus-activating regions.Memories of lost enhancersThe nucleosome remodeling complex, Snf/Swi, is required for the maintenance of transcription in vivo and is partially redundant with the histone acetyltransferase, Gcn5.Enhancement of beta-globin locus control region-mediated transactivation by mitogen-activated protein kinases through stochastic and graded mechanismsSequence requirements for myosin gene expression and regulation in Caenorhabditis elegansTarget gene specificity of E2F and pocket protein family members in living cells.Enhancers increase the probability but not the level of gene expression.A core region of the mafK gene IN promoter directs neurone-specific transcription in vivo.Renin gene expression: the switch and the fingers.Structure and regulation of the salivary gland secretion protein gene Sgs-1 of Drosophila melanogaster.DNA replication facilitates the action of transcriptional enhancers in transient expression assays.Communication over a large distance: enhancers and insulators.Transcription occurs in pulses in muscle fibersCombinatorial control of positive and negative, upstream and intragenic regulatory DNA domains of the mouse alpha 1-foetoprotein geneEnhancer-activated plasmid transcription complexes contain constrained supercoiling.An upstream activator of transcription coordinately increases the level and epigenetic stability of gene expressionSynergism between hypersensitive sites confers long-range gene activation by the beta-globin locus control region.Cooperation between elements of an organ-specific transcriptional enhancer in animalsA globin enhancer acts by increasing the proportion of erythrocytes expressing a linked transgene.Regulation of the myeloid-cell-expressed human gp91-phox gene as studied by transfer of yeast artificial chromosome clones into embryonic stem cells: suppression of a variegated cellular pattern of expression requires a full complement of distant ciIn vitro definition of the yeast RNA polymerase I enhancer.The Xenopus ribosomal DNA 60- and 81-base-pair repeats are position-dependent enhancers that function at the establishment of the preinitiation complex: analysis in vivo and in an enhancer-responsive in vitro system.Graded transcriptional response to different concentrations of a single transactivator.Direct observation of frequency modulated transcription in single cells using light activation.Methylation in the preinitiation domain suppresses gene transcription by an indirect mechanism.Xenopus transcription factors: key molecules in the developmental regulation of differential gene expression.Chromatin structure and gene expressionThe chicken beta-globin 5'HS4 boundary element blocks enhancer-mediated suppression of silencing.Variegated expression of the endogenous immunoglobulin heavy-chain gene in the absence of the intronic locus control region.Activated levels of rRNA synthesis in fission yeast are driven by an intergenic rDNA region positioned over 2500 nucleotides upstream of the initiation site.The developmental activation of the chicken lysozyme locus in transgenic mice requires the interaction of a subset of enhancer elements with the promoterPosition effects in mice carrying a lacZ transgene in cis with the beta-globin LCR can be explained by a graded model.Stochastic, stage-specific mechanisms account for the variegation of a human globin transgeneTwo synthetic Sp1-binding sites functionally substitute for the 21-base-pair repeat region to activate simian virus 40 growth in CV-1 cells.
P2860
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P2860
Formation of stable transcription complexes as assayed by analysis of individual templates.
description
1988 nî lūn-bûn
@nan
1988 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Formation of stable transcript ...... lysis of individual templates.
@ast
Formation of stable transcript ...... lysis of individual templates.
@en
type
label
Formation of stable transcript ...... lysis of individual templates.
@ast
Formation of stable transcript ...... lysis of individual templates.
@en
prefLabel
Formation of stable transcript ...... lysis of individual templates.
@ast
Formation of stable transcript ...... lysis of individual templates.
@en
P2860
P356
P1476
Formation of stable transcript ...... lysis of individual templates.
@en
P2093
Weintraub H
P2860
P304
P356
10.1073/PNAS.85.16.5819
P407
P577
1988-08-01T00:00:00Z