Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling. - Wikidata - wikimedia - TriplyDB

Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling.

Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling.

http://www.wikidata.org/entity/Q33641422

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Ambivalent role of the innate immune response in rabies virus pathogenesis Subversion of the Immune Response by Rabies Virus Resistance to Rhabdoviridae Infection and Subversion of Antiviral Responses Keeping it in check: chronic viral infection and antiviral immunity in the brain Innate immune interactions within the central nervous system modulate pathogenesis of viral infections Interplay between innate immunity and negative-strand RNA viruses: towards a rational model RelAp43, a member of the NF-κB family involved in innate immune response against Lyssavirus infection IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling Early activation of teleost B cells in response to rhabdovirus infection.The inability of wild-type rabies virus to activate dendritic cells is dependent on the glycoprotein and correlates with its low level of the de novo-synthesized leader RNA.In vivo ligands of MDA5 and RIG-I in measles virus-infected cells.Protective vaccine-induced CD4(+) T cell-independent B cell responses against rabies infection.Critical Role of K1685 and K1829 in the Large Protein of Rabies Virus in Viral Pathogenicity and Immune Evasion Type I Interferon response in olfactory bulb, the site of tick-borne flavivirus accumulation, is primarily regulated by IPS-1.Abortively Infected Astrocytes Appear To Represent the Main Source of Interferon Beta in the Virus-Infected Brain.hsp70 and a novel axis of type I interferon-dependent antiviral immunity in the measles virus-infected brain.Profile of Cytokines and Chemokines Triggered by Wild-Type Strains of Rabies Virus in Mice Genome-Wide Transcriptional Profiling Reveals Two Distinct Outcomes in Central Nervous System Infections of Rabies Virus.B cell infection and activation by rabies virus-based vaccines.The importance of immune evasion in the pathogenesis of rabies virus.Virus-heat shock protein interaction and a novel axis for innate antiviral immunity.RIG-I like receptors and their signaling crosstalk in the regulation of antiviral immunity.Sensing of RNA viruses: a review of innate immune receptors involved in recognizing RNA virus invasion.Immunity to fish rhabdoviruses Dynamics of virus-receptor interactions in virus binding, signaling, and endocytosis Immunological aspects of rabies: a literature review.Wild-type rabies virus phosphoprotein is associated with viral sensitivity to type I interferon treatment.Systems Biomedicine of Rabies Delineates the Affected Signaling Pathways.The type I interferon response bridles rabies virus infection and reduces pathogenicity.Pattern recognition receptor MDA5 modulates CD8+ T cell-dependent clearance of West Nile virus from the central nervous system.Rabies virus is recognized by the NLRP3 inflammasome and activates interleukin-1β release in murine dendritic cells Type 1 Interferons Induce Changes in Core Metabolism that Are Critical for Immune Function.Characterization and immune response expression of the Rig-I-like receptor mda5 in common carp Cyprinus carpio.Dendritic cells infected by recombinant rabies virus vaccine vector expressing HIV-1 Gag are immunogenic even in the presence of vector-specific immunity.MicroRNA and mRNA Dysregulation in Astrocytes Infected with Zika Virus.Novel insect-specific Eilat virus-based chimeric vaccine candidates provide durable, mono- and multi-valent, single dose protection against lethal alphavirus challenge.Lab-Attenuated Rabies Virus Causes Abortive Infection and Induces Cytokine Expression in Astrocytes by Activating Mitochondrial Antiviral-Signaling Protein Signaling Pathway.Analysis of expression profiles of long noncoding RNAs and mRNAs in brains of mice infected by rabies virus by RNA sequencing

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Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling.

http://www.wikidata.org/entity/Q33641422

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Rabies virus infection induces ...... ion relies on IFNAR signaling.

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Celestine N Wanjalla

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Christoph Wirblich

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Elizabeth J Faul

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Matthias J Schnell

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Mehul S Suthar

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P2860

IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction

Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

Virus-dependent phosphorylation of the IRF-3 transcription factor regulates nuclear translocation, transactivation potential, and proteasome-mediated degradation

Antiviral actions of interferons

Recognition of single-stranded RNA viruses by Toll-like receptor 7

Establishment and Maintenance of the Innate Antiviral Response to West Nile Virus Involves both RIG-I and MDA5 Signaling through IPS-1

Melanoma Differentiation-Associated Gene 5 (MDA5) Is Involved in the Innate Immune Response to Paramyxoviridae Infection In Vivo

IPS-1 Is Essential for the Control of West Nile Virus Infection and Immunity

Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

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