Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis - Wikidata - wikimedia - TriplyDB

Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis

Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis

http://www.wikidata.org/entity/Q33642456

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Developmental exposure to estrogen alters differentiation and epigenetic programming in a human fetal prostate xenograft model Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses Lymphoma and lung cancer in offspring born to pregnant mice dosed with dibenzo[a,l]pyrene: the importance of in utero vs. lactational exposure Endocrine-disrupting chemicals: an Endocrine Society scientific statement Early-life Exposure to Endocrine Disrupting Chemicals and Later-life Health Outcomes: An Epigenetic Bridge?Xenotransplantation models to study the effects of toxicants on human fetal tissues Environmental endocrine disruptors: Effects on the human male reproductive system DNA methylation changes in whole blood is associated with exposure to the environmental contaminants, mercury, lead, cadmium and bisphenol A, in women undergoing ovarian stimulation for IVF Endocrine-disrupting chemicals: associated disorders and mechanisms of action Disruption of androgen receptor signaling in males by environmental chemicals The bisphenol A experience: a primer for the analysis of environmental effects on mammalian reproduction Endocrine disruptors and prostate cancer risk Actions of estrogens and endocrine disrupting chemicals on human prostate stem/progenitor cells and prostate cancer risk Toxicogenomic and phenotypic analyses of bisphenol-A early-life exposure toxicity in zebrafish Stat3 is a candidate epigenetic biomarker of perinatal Bisphenol A exposure associated with murine hepatic tumors with implications for human health Estrogens in Male Physiology The role of Bisphenol A in shaping the brain, epigenome and behavior.Bisphenol-A (BPA), BPA glucuronide, and BPA sulfate in midgestation umbilical cord serum in a northern and central California population.Dose-dependent incidence of hepatic tumors in adult mice following perinatal exposure to bisphenol A Epigenetic transgenerational actions of environmental factors in disease etiology Maternal dioxin exposure combined with a diet high in fat increases mammary cancer incidence in mice.Epigenetic changes with dietary soy in cynomolgus monkeys Epigenomic disruption: the effects of early developmental exposures.Environmental toxicants and the developing immune system: a missing link in the global battle against infectious disease?Human excretion of bisphenol A: blood, urine, and sweat (BUS) study.Epigenetic transgenerational actions of endocrine disruptors Influence of environmental exposure on human epigenetic regulation Atypical fetal prostate development is associated with ipsilateral hypoplasia of the wolffian ducts in the ACI rat.A human fetal prostate xenograft model of developmental estrogenization.Preimplantation exposure to bisphenol A (BPA) affects embryo transport, preimplantation embryo development, and uterine receptivity in mice.The biological basis and clinical significance of hormonal imprinting, an epigenetic process Multigenerational and transgenerational effects of endocrine disrupting chemicals: A role for altered epigenetic regulation?Epigenetic responses following maternal dietary exposure to physiologically relevant levels of bisphenol A.A review of the carcinogenic potential of bisphenol A.Differential Effects of Estradiol and Bisphenol A on SET8 and SIRT1 Expression in Ovarian Cancer Cells.Male reprotoxicity and endocrine disruption.The epigenome as a potential mediator of cancer and disease prevention in prenatal development.Bisphenol A modifies the regulation exerted by testosterone on 5 α -reductase isozymes in ventral prostate of adult rats.Estrogen signaling is not required for prostatic bud patterning or for its disruption by 2,3,7,8-tetrachlorodibenzo-p-dioxin.Prenatal and pubertal testosterone exposure imprint permanent modifications in the prostate that predispose to the development of lesions in old Mongolian gerbils.

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P2860

Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis

http://www.wikidata.org/entity/Q33642456

description

2008 nî lūn-bûn

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2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2008 թվականի փետրվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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J.1742-7843.2007.00166.X

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Basic and Clinical Pharmacology and Toxicology

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Perinatal exposure to oestradi ...... sceptibility to carcinogenesis

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Gail S Prins

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Jessica Belmonte

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Shuk-Mei Ho

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Wan-Yee Tang

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P2860

Estrogenic chemicals in plastic and oral contraceptives disrupt development of the fetal mouse prostate and urethra.

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

Developmental increases in rat hepatic microsomal UDP-glucuronosyltransferase activities toward xenoestrogens and decreases during pregnancy.

The decline of androgen levels in elderly men and its clinical and therapeutic implications

Exposure to bisphenol A advances puberty

Epigenetic transgenerational actions of endocrine disruptors and male fertility

Cancer statistics, 2006

Determination of bisphenol A concentrations in human biological fluids reveals significant early prenatal exposure

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