Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4 - Wikidata - wikimedia - TriplyDB

Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

http://www.wikidata.org/entity/Q24648201

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Developmental exposure to estrogen alters differentiation and epigenetic programming in a human fetal prostate xenograft model Exposure to bisphenol A correlates with early-onset prostate cancer and promotes centrosome amplification and anchorage-independent growth in vitro Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutations Epigenetic mechanism underlying the development of polycystic ovary syndrome (PCOS)-like phenotypes in prenatally androgenized rhesus monkeys Perinatal bisphenol A exposure promotes dose-dependent alterations of the mouse methylome Cancer is a preventable disease that requires major lifestyle changes Incorporating genetics and genomics in risk assessment for inhaled manganese: from data to policy Basic exploratory research versus guideline-compliant studies used for hazard evaluation and risk assessment: bisphenol A as a case study Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure No effect of route of exposure (oral; subcutaneous injection) on plasma bisphenol A throughout 24h after administration in neonatal female mice Disruption of adult expression of sexually selected traits by developmental exposure to bisphenol A.Xenoestrogen-induced epigenetic repression of microRNA-9-3 in breast epithelial cells Environmental and occupational causes of cancer: new evidence 2005-2007 Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption Endocrine-disrupting chemicals: an Endocrine Society scientific statement Why public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol A Developmental exposure to bisphenol A increases prostate cancer susceptibility in adult rats: epigenetic mode of action is implicated Oral exposure to bisphenol a increases dimethylbenzanthracene-induced mammary cancer in rats Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life Estradiol and Bisphenol A stimulate androgen receptor and estrogen receptor gene expression in fetal mouse prostate mesenchyme cells Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure In vivo effects of bisphenol A in laboratory rodent studies Historical review of the causes of cancer Molecular Mechanisms of Action of BPA Environmental Epigenetics: Crossroad between Public Health, Lifestyle, and Cancer Prevention The estrogenic content of rodent diets, bedding, cages, and water bottles and its effect on bisphenol A studies Peripheral chemoreception and arterial pressure responses to intermittent hypoxia Early-life Exposure to Endocrine Disrupting Chemicals and Later-life Health Outcomes: An Epigenetic Bridge?Epigenetic effects of environmental chemicals bisphenol A and phthalates Directed Differentiation of Human Embryonic Stem Cells into Prostate Organoids In Vitro and its Perturbation by Low-Dose Bisphenol A Exposure Prenatal Exposure to DEHP Affects Spermatogenesis and Sperm DNA Methylation in a Strain-Dependent Manner Toxicity testing in the 21st century: a vision and a strategy Environmental endocrine disruptors: Effects on the human male reproductive system Environmental immune disruptors, inflammation and cancer risk Environmental causes of cancer: endocrine disruptors as carcinogens EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals DNA methylation changes in whole blood is associated with exposure to the environmental contaminants, mercury, lead, cadmium and bisphenol A, in women undergoing ovarian stimulation for IVF Effects of High-Butterfat Diet on Embryo Implantation in Female Rats Exposed to Bisphenol A

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Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

http://www.wikidata.org/entity/Q24648201

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2006 nî lūn-bûn

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2006 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2006年の論文

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Cancer Research

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Gail S Prins

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Jessica Belmonte de Frausto

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Shuk-Mei Ho

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Wan-Yee Tang

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P2860

Relative binding affinity-serum modified access (RBA-SMA) assay predicts the relative in vivo bioactivity of the xenoestrogens bisphenol A and octylphenol

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

Cancer and developmental exposure to endocrine disruptors

The decline of androgen levels in elderly men and its clinical and therapeutic implications

Exposure to bisphenol A advances puberty

Epigenetic transgenerational actions of endocrine disruptors and male fertility

MethPrimer: designing primers for methylation PCRs

Prostate enlargement in mice due to fetal exposure to low doses of estradiol or diethylstilbestrol and opposite effects at high doses

BiQ Analyzer: visualization and quality control for DNA methylation data from bisulfite sequencing.

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