Laser-induced endothelial cell activation supports fibrin formation.
about
Imaging coagulation reactions in vivoEndothelium-derived but not platelet-derived protein disulfide isomerase is required for thrombus formation in vivo.Active site-labeled prothrombin inhibits prothrombinase in vitro and thrombosis in vivo.Protein disulfide isomerase inhibitors constitute a new class of antithrombotic agents.Protein disulfide isomerase capture during thrombus formation in vivo depends on the presence of β3 integrinsNew insights into the spatiotemporal localization of prothrombinase in vivo.Platelets are required for enhanced activation of the endothelium and fibrinogen in a mouse thrombosis model of APS.Agonist-induced platelet procoagulant activity requires shear and a Rac1-dependent signaling mechanism.CEACAM2 negatively regulates hemi (ITAM-bearing) GPVI and CLEC-2 pathways and thrombus growth in vitro and in vivo.Neutrophils recruit and activate human endothelial colony-forming cells at the site of vessel injury via P-selectin glycoprotein ligand-1 and L-selectin.Procoagulant activity in hemostasis and thrombosis: Virchow's triad revisited.Thrombotic Role of Blood and Endothelial Cells in Uremia through Phosphatidylserine Exposure and Microparticle ReleaseEffect of zymogen domains and active site occupation on activation of prothrombin by von Willebrand factor-binding protein.A systems approach to hemostasis: 4. How hemostatic thrombi limit the loss of plasma-borne molecules from the microvasculature.Tissue factor-bearing microparticles and thrombus formation.Tissue factor pathway inhibitor: structure-function.Formation of the clot.Revisited role of microparticles in arterial and venous thrombosis.Encryption and decryption of tissue factor.Spatiotemporal regulation of coagulation and platelet activation during the hemostatic response in vivo.Inhibition of Protein Disulfide Isomerase in Thrombosis.A vascular injury model using focal heat-induced activation of endothelial cells.P2X7 receptor signaling contributes to tissue factor-dependent thrombosis in mice.Mice with a deficiency in CLEC-2 are protected against deep vein thrombosis.The endothelial protein C receptor enhances hemostasis of FVIIa administration in hemophilic mice in vivo.P-Selectin and ICAM-1 synergy in mediating THP-1 monocyte adhesion in hemodynamic flow is length dependent.Inhibition of platelet activation prevents the P-selectin and integrin-dependent accumulation of cancer cell microparticles and reduces tumor growth and metastasis in vivo.
P2860
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P2860
Laser-induced endothelial cell activation supports fibrin formation.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Laser-induced endothelial cell activation supports fibrin formation.
@ast
Laser-induced endothelial cell activation supports fibrin formation.
@en
type
label
Laser-induced endothelial cell activation supports fibrin formation.
@ast
Laser-induced endothelial cell activation supports fibrin formation.
@en
prefLabel
Laser-induced endothelial cell activation supports fibrin formation.
@ast
Laser-induced endothelial cell activation supports fibrin formation.
@en
P2093
P2860
P1433
P1476
Laser-induced endothelial cell activation supports fibrin formation.
@en
P2093
Barbara C Furie
Ben T Atkinson
Bruce Furie
Prathima Nandivada
Reema Jasuja
P2860
P304
P356
10.1182/BLOOD-2010-05-283986
P407
P50
P577
2010-07-30T00:00:00Z