Anoxic contractile failure in rat heart myocytes is caused by failure of intracellular calcium release due to alteration of the action potential
about
Hypoxia/reoxygenation cardiac injury and regeneration in zebrafish adult heartMyocardial ischemia and increased heart work modulate the phosphorylation state of eukaryotic elongation factor-2Hypoxia, not the frequency of sleep apnea, induces acute hemodynamic stress in patients with chronic heart failure.The Driving Force of the Na/Ca-Exchanger during Metabolic Inhibition.Effects of K+ channel blockers on the action potential of hypoxic rabbit myocardiumRole of adenosine A1 and A3 receptors in regulation of cardiomyocyte homeostasis after mitochondrial respiratory chain injury.Effect of metabolic inhibition on couplon behavior in rabbit ventricular myocytesExcitation-contraction coupling in heart failure.Current methodologies for the study of pineal morphophysiology.Simultaneous recording of ATP-sensitive K+ current and intracellular Ca2+ in anoxic rat ventricular myocytes. Effects of glibenclamide.Weak electromagnetic fields alter Ca(2+) handling and protect against hypoxia-mediated damage in primary newborn rat myotube cultures.The electrophysiology of ischemia and cardioplegia: implications for myocardial protection.Effects of pretreatment with caffeine or ryanodine on the myocardial response to simulated ischaemiaInvolvement of uracil nucleotides in protection of cardiomyocytes from hypoxic stress.Regulation of intracellular free Mg2+ and contraction in single adult mammalian cardiac myocytes.Continuous fluorimetric assessment of the changes in cytoplasmic calcium concentration during exposure of rat isolated myocardium to conditions of simulated ischaemia.Exercise training alters an anoxia-induced, glibenclamide-sensitive current in rat ventricular cardiocytes.Graded response of K+ current, membrane potential, and [Ca2+]i to hypoxia in pulmonary arterial smooth muscle.MK 801 attenuates c-Fos and c-Jun expression after in vitro ischemia in rat neuronal cell cultures but not in PC 12 cells.Early effects of metabolic inhibition on intracellular Ca2+ in toad pacemaker cells: involvement of Ca2+ stores.Na(+) current through KATP channels: consequences for Na(+) and K(+) fluxes during early myocardial ischemia.A model of graded calcium release and L-type Ca2+ channel inactivation in cardiac muscle.SUR2A C-terminal fragments reduce KATP currents and ischaemic tolerance of rat cardiac myocytes.ATP-sensitive K+ channels in heart muscle cells first open and subsequently close at maintained anoxia.β-Adrenergic Inhibition Prevents Action Potential and Calcium Handling Changes during Regional Myocardial Ischemia.Regulation of Ca2+ signaling by acute hypoxia and acidosis in rat neonatal cardiomyocytes.Long-term HIF-1α transcriptional activation is essential for heat-acclimation-mediated cross tolerance: mitochondrial target genes.
P2860
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P2860
Anoxic contractile failure in rat heart myocytes is caused by failure of intracellular calcium release due to alteration of the action potential
description
1988 nî lūn-bûn
@nan
1988 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Anoxic contractile failure in ...... ration of the action potential
@ast
Anoxic contractile failure in ...... ration of the action potential
@en
type
label
Anoxic contractile failure in ...... ration of the action potential
@ast
Anoxic contractile failure in ...... ration of the action potential
@en
prefLabel
Anoxic contractile failure in ...... ration of the action potential
@ast
Anoxic contractile failure in ...... ration of the action potential
@en
P2093
P2860
P356
P1476
Anoxic contractile failure in ...... ration of the action potential
@en
P2093
C G Nichols
E G Lakatta
H S Silverman
M C Capogrossi
R A Josephson
S R Houser
W J Lederer
P2860
P304
P356
10.1073/PNAS.85.18.6954
P407
P577
1988-09-01T00:00:00Z