Long-term HIF-1α transcriptional activation is essential for heat-acclimation-mediated cross tolerance: mitochondrial target genes.

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Heat Acclimation-Mediated Cross-Tolerance: Origins in within-Life Epigenetics?Heat Acclimatization Protects the Left Ventricle from Increased Diastolic Chamber Stiffness Immediately after Coronary Artery Bypass Surgery: A Lesson from 30 Years of Studies on Heat Acclimation Mediated Cross Tolerance.

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Long-term HIF-1α transcriptional activation is essential for heat-acclimation-mediated cross tolerance: mitochondrial target genes.

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Long-term HIF-1α transcription ...... e: mitochondrial target genes.

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Long-term HIF-1α transcription ...... e: mitochondrial target genes.

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Long-term HIF-1α transcription ...... e: mitochondrial target genes.

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Long-term HIF-1α transcription ...... e: mitochondrial target genes.

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Long-term HIF-1α transcription ...... e: mitochondrial target genes.

@en

Long-term HIF-1α transcription ...... e: mitochondrial target genes.

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Long-term HIF-1α transcription ...... e: mitochondrial target genes.

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Ahmad Mreisat

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Elia Smeir

http://www.w3.org/2001/XMLSchema#string

Gary Gerstenblith

http://www.w3.org/2001/XMLSchema#string

Michael D Stern

http://www.w3.org/2001/XMLSchema#string

Michal Horowitz

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Rivka Alexander-Shani

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P2860

Hypoxia promotes glycogen accumulation through hypoxia inducible factor (HIF)-mediated induction of glycogen synthase 1

HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

HIF-1-mediated expression of pyruvate dehydrogenase kinase: a metabolic switch required for cellular adaptation to hypoxia

HIF-1 mediates adaptation to hypoxia by actively downregulating mitochondrial oxygen consumption

Evolution of the oxygen sensitivity of cytochrome c oxidase subunit 4.

Anoxic contractile failure in rat heart myocytes is caused by failure of intracellular calcium release due to alteration of the action potential

Heat stress proteins and myocardial protection: experimental model or potential clinical tool?

Inhibition of prolyl hydroxylases by dimethyloxaloylglycine after stroke reduces ischemic brain injury and requires hypoxia inducible factor-1α.

Upregulation of the mitochondrial Lon Protease allows adaptation to acute oxidative stress but dysregulation is associated with chronic stress, disease, and aging.

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scholarly article

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10.1152/AJPREGU.00461.2016

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2017-03-08T00:00:00Z

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28274939

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Long-term HIF-1α transcriptional activation is essential for heat-acclimation-mediated cross tolerance: mitochondrial target genes.

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P2860

Long-term HIF-1α transcriptional activation is essential for heat-acclimation-mediated cross tolerance: mitochondrial target genes.

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