Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
about
Mitochondrial structure, function and dynamics are temporally controlled by c-MycHIV-1 Tat Activates Neuronal Ryanodine Receptors with Rapid Induction of the Unfolded Protein Response and Mitochondrial HyperpolarizationBeing right on Q: shaping eukaryotic evolutionAn interferon signature in the peripheral blood of dermatomyositis patients is associated with disease activityThe dendritic cell response to classic, emerging, and homeostatic danger signals. Implications for autoimmunitySurvival features of EBV-stabilized cells from centenarians: morpho-functional and transcriptomic analysesRETRACTED: Functional activation of proline-rich tyrosine kinase2 (PYK2) in peripheral blood mononuclear cells from patients with systemic lupus erythematosus.Systems biology of lupus: mapping the impact of genomic and environmental factors on gene expression signatures, cellular signaling, metabolic pathways, hormonal and cytokine imbalance, and selecting targets for treatment.Rapamycin reduces disease activity and normalizes T cell activation-induced calcium fluxing in patients with systemic lupus erythematosus.Infection in systemic lupus erythematosus: friend or foe?Pathogenic mechanisms in systemic lupus erythematosus.Emerging new pathways of pathogenesis and targets for treatment in systemic lupus erythematosus and Sjogren's syndrome.Oxidative stress in the pathology and treatment of systemic lupus erythematosusHRES-1/Rab4-mediated depletion of Drp1 impairs mitochondrial homeostasis and represents a target for treatment in SLE.T-cell and B-cell signaling biomarkers and treatment targets in lupus.Mitochondrial dysregulation and glycolytic insufficiency functionally impair CD8 T cells infiltrating human renal cell carcinoma.Characterization of the metabolic phenotype of rapamycin-treated CD8+ T cells with augmented ability to generate long-lasting memory cellsEstrogen receptor signaling and its relationship to cytokines in systemic lupus erythematosus.Altered mitochondrial membrane potential, mass, and morphology in the mononuclear cells of humans with type 2 diabetes.ERK signaling leads to mitochondrial dysfunction in extracellular zinc-induced neurotoxicity.Central role of nitric oxide in the pathogenesis of rheumatoid arthritis and systemic lupus erythematosus.Mitochondrial DNA damage is associated with damage accrual and disease duration in patients with systemic lupus erythematosusMechanistic target of rapamycin activation triggers IL-4 production and necrotic death of double-negative T cells in patients with systemic lupus erythematosus.Quantification of uncoupling protein 2 reveals its main expression in immune cells and selective up-regulation during T-cell proliferationMitochondria regulate platelet metamorphosis induced by opsonized zymosan A--activation and long-term commitment to cell death.Activation of mammalian target of rapamycin controls the loss of TCRzeta in lupus T cells through HRES-1/Rab4-regulated lysosomal degradationMitochondrial membrane potential probes and the proton gradient: a practical usage guideThe role of nitric oxide in abnormal T cell signal transduction in systemic lupus erythematosusViolacein induces cell death by triggering mitochondrial membrane hyperpolarization in vitro.Association of mtDNA M/N haplogroups with systemic lupus erythematosus: a case-control study of Han Chinese women.Nitric oxide, mitochondrial hyperpolarization, and T cell activation.Murine lupus susceptibility locus Sle1c2 mediates CD4+ T cell activation and maps to estrogen-related receptor γ.Metabolic regulation of organelle homeostasis in lupus T cellsLeukocyte mitochondrial DNA alteration in systemic lupus erythematosus and its relevance to the susceptibility to lupus nephritisROS as signalling molecules in T cells--evidence for abnormal redox signalling in the autoimmune disease, rheumatoid arthritis.Glucose Oxidation Is Critical for CD4+ T Cell Activation in a Mouse Model of Systemic Lupus Erythematosus.The biology of nitric oxide and other reactive intermediates in systemic lupus erythematosus.Association of common mitochondrial DNA variants with multiple sclerosis and systemic lupus erythematosus.Fungal Innate Immunity Induced by Bacterial Microbe-Associated Molecular Patterns (MAMPs).Molecular mimicry and immunomodulation by the HRES-1 endogenous retrovirus in SLE.
P2860
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P2860
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
description
2004 nî lūn-bûn
@nan
2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@ast
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@en
type
label
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@ast
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@en
prefLabel
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@ast
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@en
P2093
P2860
P356
P1433
P1476
Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.
@en
P2093
Agnes Koncz
Gyorgy Nagy
Katalin Banki
Peter Gergely
P2860
P304
P356
10.1016/J.IT.2004.05.001
P50
P577
2004-07-01T00:00:00Z