The biology of nitric oxide and other reactive intermediates in systemic lupus erythematosus.
about
Nitric oxide and peroxynitrite in health and diseaseMechanisms of tissue injury in lupus nephritisThe dendritic cell response to classic, emerging, and homeostatic danger signals. Implications for autoimmunityThe biology of reactive intermediates in systemic lupus erythematosus.Oxidative stress, T cell DNA methylation, and lupus.Mollaret meningitis may be caused by reactivation of latent cerebral toxoplasmosis.Accelerated vascular disease in systemic lupus erythematosus: role of macrophage.Systemic lupus erythematosus serum deposits C4d on red blood cells, decreases red blood cell membrane deformability, and promotes nitric oxide production.CD4(+) T cells epigenetically modified by oxidative stress cause lupus-like autoimmunity in mice.Lack of nitric oxide synthases increases lipoprotein immune complex deposition in the aorta and elevates plasma sphingolipid levels in lupusEndothelial nitric oxide synthase inhibits the development of autoimmune-mediated vasculitis in mice.Heroin-induced conditioned immunomodulation requires expression of IL-1β in the dorsal hippocampus.Histone H2AX phosphorylation as a measure of DNA double-strand breaks and a marker of environmental stress and disease activity in lupus.Inducible nitric oxide synthase inhibitor SD-3651 reduces proteinuria in MRL/lpr mice deficient in the NOS2 gene.Whole-genome transcription and DNA methylation analysis of peripheral blood mononuclear cells identified aberrant gene regulation pathways in systemic lupus erythematosus.Dissecting complex epigenetic alterations in human lupus.Potential Benefits of PeroxynitriteAssociation of serum nitrate and nitrite levels with longitudinal assessments of disease activity and damage in systemic lupus erythematosus and lupus nephritis.Role of peroxynitrite-modified biomolecules in the etiopathogenesis of systemic lupus erythematosus.Understanding mechanisms of hypertension in systemic lupus erythematosus.Immunobiology of Nitric Oxide and Regulation of Inducible Nitric Oxide Synthase.T cell PKCδ kinase inactivation induces lupus-like autoimmunity in mice.Oxidative stress and homocysteine metabolism in patients with lupus nephritis.Analysis of endothelial nitric oxide synthase (eNOS) G894T polymorphism and semen parameters in a Chinese Han population.Nitration of H2B histone elicits an immune response in experimental animals.The interaction between environmental triggers and epigenetics in autoimmunity.Association of eNOS gene polymorphisms and systemic lupus erythematosus in southeast Iran.Expression of oxidized protein tyrosine phosphatase and γH2AX predicts poor survival of gastric carcinoma patients
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P2860
The biology of nitric oxide and other reactive intermediates in systemic lupus erythematosus.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
The biology of nitric oxide an ...... systemic lupus erythematosus.
@ast
The biology of nitric oxide an ...... systemic lupus erythematosus.
@en
type
label
The biology of nitric oxide an ...... systemic lupus erythematosus.
@ast
The biology of nitric oxide an ...... systemic lupus erythematosus.
@en
prefLabel
The biology of nitric oxide an ...... systemic lupus erythematosus.
@ast
The biology of nitric oxide an ...... systemic lupus erythematosus.
@en
P2860
P1433
P1476
The biology of nitric oxide an ...... systemic lupus erythematosus.
@en
P2093
Gary S Gilkeson
Jim C Oates
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P304
P356
10.1016/J.CLIM.2006.06.001
P577
2006-07-24T00:00:00Z