Hypoxia-inducible factor-dependent signaling between triple-negative breast cancer cells and mesenchymal stem cells promotes macrophage recruitment. - Wikidata - wikimedia - TriplyDB

Hypoxia-inducible factor-dependent signaling between triple-negative breast cancer cells and mesenchymal stem cells promotes macrophage recruitment.

Hypoxia-inducible factor-dependent signaling between triple-negative breast cancer cells and mesenchymal stem cells promotes macrophage recruitment.

http://www.wikidata.org/entity/Q33665254

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Hypoxic control of metastasis A Review of Systemic Treatment in Metastatic Triple-Negative Breast Cancer Recent Advances in Discovering the Role of CCL5 in Metastatic Breast Cancer Scavenger Receptors: Emerging Roles in Cancer Biology and Immunology Radio-resistant mesenchymal stem cells: mechanisms of resistance and potential implications for the clinic Hypoxia: a key player in antitumor immune response. A Review in the Theme: Cellular Responses to Hypoxia Whole Genome Amplification of Day 3 or Day 5 Human Embryos Biopsies Provides a Suitable DNA Template for PCR-Based Techniques for Genotyping, a Complement of Preimplantation Genetic Testing.New targeted therapies for breast cancer: A focus on tumor microenvironmental signals and chemoresistant breast cancers Hypoxia-inducible factor 1 mediates TAZ expression and nuclear localization to induce the breast cancer stem cell phenotype.Gene-expression molecular subtyping of triple-negative breast cancer tumours: importance of immune response.Hypoxia-inducible factors in cancer stem cells and inflammation.HIF-1 at the crossroads of hypoxia, inflammation, and cancer.Immunological mechanisms of the antitumor effects of supplemental oxygenation.HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells.Basal-like breast cancer: molecular profiles, clinical features and survival outcomes The hypoxic tumor microenvironment: A driving force for breast cancer progression.A bladder cancer microenvironment simulation system based on a microfluidic co-culture model CXCR3 as a molecular target in breast cancer metastasis: inhibition of tumor cell migration and promotion of host anti-tumor immunity.Alterations of tumor microenvironment by carbon monoxide impedes lung cancer growth.Inflammation and Cancer: Extra- and Intracellular Determinants of Tumor-Associated Macrophages as Tumor Promoters.TNFα-activated mesenchymal stromal cells promote breast cancer metastasis by recruiting CXCR2+ neutrophils Nanocarrier-mediated drugs targeting cancer stem cells: an emerging delivery approach.Cancer stem cells and tumor-associated macrophages: a roadmap for multitargeting strategies.Tumour-associated mesenchymal stem/stromal cells: emerging therapeutic targets.Interactions between mesenchymal stem cells and the immune system.The Therapeutic Potential of Targeting Tumor Microenvironment in Breast Cancer: Rational Strategies and Recent Progress.Hypoxia: Signaling the Metastatic Cascade.Radiofrequency ablation-increased CXCL10 is associated with earlier recurrence of hepatocellular carcinoma by promoting stemness.Geminin overexpression-dependent recruitment and crosstalk with mesenchymal stem cells enhance aggressiveness in triple negative breast cancers.The Linkage between Breast Cancer, Hypoxia, and Adipose Tissue.Immunization against HIF-1α Inhibits the Growth of Basal Mammary Tumors and Targets Mammary Stem Cells In Vivo.Tumor-educated mesenchymal stem cells promote pro-metastatic phenotype.The Immunomodulatory Effects of Mesenchymal Stem Cell Polarization within the Tumor Microenvironment Niche.Robust method for identification of prognostic gene signatures from gene expression profiles.A niche that triggers aggressiveness within BRCA1-IRIS overexpressing triple negative tumors is supported by reciprocal interactions with the microenvironment.Pathologic Involvement of Glutamatergic Striatal Inputs From the Cortices in TAR DNA-Binding Protein 43 kDa-Related Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis.Chemotherapy induces enrichment of CD47+/CD73+/PDL1+ immune evasive triple-negative breast cancer cells.Anti-angiogenic treatment promotes triple-negative breast cancer invasion via vasculogenic mimicry.Inflammatory Human Umbilical Cord-Derived Mesenchymal Stem Cells Promote Stem Cell-Like Characteristics of Cancer Cells in an IL-1β-Dependent Manner.The role of tumor microenvironment in resistance to anti-angiogenic therapy.

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Hypoxia-inducible factor-dependent signaling between triple-negative breast cancer cells and mesenchymal stem cells promotes macrophage recruitment.

http://www.wikidata.org/entity/Q33665254

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年學術文章

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name

Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Proceedings of the National Academy of Sciences of the United States of America

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Hypoxia-inducible factor-depen ...... omotes macrophage recruitment.

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Daniele M Gilkes

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Naoharu Takano

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Pallavi Chaturvedi

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P2860

Hypoxia-inducible factors mediate coordinated RhoA-ROCK1 expression and signaling in breast cancer cells

Comprehensive molecular portraits of human breast tumours

Levels of hypoxia-inducible factor-1alpha independently predict prognosis in patients with lymph node negative breast carcinoma

Critical role for lysyl oxidase in mesenchymal stem cell-driven breast cancer malignancy

Hypoxia--a key regulatory factor in tumour growth

Microenvironmental regulation of metastasis

Mesenchymal stem cells within tumour stroma promote breast cancer metastasis

Tumour-educated macrophages promote tumour progression and metastasis

Colony-stimulating factor 1 promotes progression of mammary tumors to malignancy

CCL2 recruits inflammatory monocytes to facilitate breast-tumour metastasis

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