Mutation of mouse Samd4 causes leanness, myopathy, uncoupled mitochondrial respiration, and dysregulated mTORC1 signaling.
about
Exploring the Functional Disorder and Corresponding Key Transcription Factors in Intraductal Papillary Mucinous Neoplasms Progression.RNA-binding protein SAMD4 regulates skeleton development through translational inhibition of Mig6 expression.Chronic innate immune activation of TBK1 suppresses mTORC1 activity and dysregulates cellular metabolism.CircRNA expression pattern and circRNA-miRNA-mRNA network in the pathogenesis of nonalcoholic steatohepatitis.Smaug variants in neural and non-neuronal cells.Intracellular lipid metabolism impairs β cell compensation during diet-induced obesity.Fully-automated, high-throughput micro-computed tomography analysis of body composition enables therapeutic efficacy monitoring in preclinical models.
P2860
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P2860
Mutation of mouse Samd4 causes leanness, myopathy, uncoupled mitochondrial respiration, and dysregulated mTORC1 signaling.
description
2014 nî lūn-bûn
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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年學術文章
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name
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@ast
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@en
type
label
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@ast
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@en
prefLabel
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@ast
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@en
P2093
P2860
P356
P1476
Mutation of mouse Samd4 causes ...... dysregulated mTORC1 signaling.
@en
P2093
Eva Marie Y Moresco
John M Shelton
Sungyong Won
Wataru Tomisato
William Holland
Xiaohong Li
Xiaoming Zhan
P2860
P304
P356
10.1073/PNAS.1406511111
P407
P577
2014-05-05T00:00:00Z