Antisickling property of fetal hemoglobin enhances nitric oxide bioavailability and ameliorates organ oxidative stress in transgenic-knockout sickle mice.
about
Triggers, inhibitors, mechanisms, and significance of eryptosis: the suicidal erythrocyte deathGenetic and biochemical markers of hydroxyurea therapeutic response in sickle cell anemia.MP4CO, a pegylated hemoglobin saturated with carbon monoxide, is a modulator of HO-1, inflammation, and vaso-occlusion in transgenic sickle mice.Reconstructing sickle cell disease: a data-based analysis of the "hyperhemolysis paradigm" for pulmonary hypertension from the perspective of evidence-based medicine.2D DIGE based proteomics study of erythrocyte cytosol in sickle cell disease: altered proteostasis and oxidative stress.NT-pro brain natriuretic peptide levels and the risk of death in the cooperative study of sickle cell disease.Comparing segmented ASL perfusion of vascular territories using manual versus semiautomated techniques in children with sickle cell anemiaTargeting NADPH oxidase decreases oxidative stress in the transgenic sickle cell mouse penisHydroxyurea with AKT2 inhibition decreases vaso-occlusive events in sickle cell disease miceAntisickling fetal hemoglobin reduces hypoxia-inducible factor-1α expression in normoxic sickle mice: microvascular implications.Erythrocyte NADPH oxidase activity modulated by Rac GTPases, PKC, and plasma cytokines contributes to oxidative stress in sickle cell disease.ARQ 092, an orally-available, selective AKT inhibitor, attenuates neutrophil-platelet interactions in sickle cell disease.Oxidative stress in sickle cell disease; pathophysiology and potential implications for disease management.Therapeutic potential of manipulating suicidal erythrocyte death.Mechanistic studies of anti-hyperpigmentary compounds: elucidating their inhibitory and regulatory actionsOxidative status in sickle cell anemia.Association of Inflammatory Biomarker C-Reactive Protein, Lipid Peroxidation and Antioxidant Capacity Marker with HbF Level in Sickle Cell Disease Patients from Chattisgarh.Oxidative stress status, clinical outcome, and β-globin gene cluster haplotypes in pediatric patients with sickle cell disease.Analysis of cytokine immune response profile in response to inflammatory stimuli in mice with genetic defects in fetal and adult hemoglobin chain expression.Dimethyl fumarate increases fetal hemoglobin, provides heme detoxification, and corrects anemia in sickle cell disease.
P2860
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P2860
Antisickling property of fetal hemoglobin enhances nitric oxide bioavailability and ameliorates organ oxidative stress in transgenic-knockout sickle mice.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@ast
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@en
type
label
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@ast
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@en
prefLabel
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@ast
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@en
P2093
P2860
P1476
Antisickling property of fetal ...... ansgenic-knockout sickle mice.
@en
P2093
Dhananjay K Kaul
Mary E Fabry
Trisha Dasgupta
P2860
P304
P356
10.1152/AJPREGU.00611.2009
P577
2009-12-09T00:00:00Z