Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease. - Wikidata - wikimedia - TriplyDB

Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease.

Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease.

http://www.wikidata.org/entity/Q33953052

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Interventions for treating pulmonary hypertension in people with sickle cell disease or thalassaemia Towards a unifying, systems biology understanding of large-scale cellular death and destruction caused by poorly liganded iron: Parkinson's, Huntington's, Alzheimer's, prions, bactericides, chemical toxicology and others as examples Therapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinol The role of the arginine metabolome in pain: implications for sickle cell disease A new paradigm for XOR-catalyzed reactive species generation in the endothelium Oxidative stress in β-thalassaemia and sickle cell disease Molecular pathophysiology of priapism: emerging targets Interplay between oxidant species and energy metabolism Iron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseases Oxidases and peroxidases in cardiovascular and lung disease: new concepts in reactive oxygen species signaling Red blood cells induce hypoxic lung inflammation Sickle Cells Abolish Melanoma Tumorigenesis in Hemoglobin SS Knockin Mice and Augment the Tumoricidal Effect of Oncolytic Virus In Vivo Adenosine A2A receptors induced on iNKT and NK cells reduce pulmonary inflammation and injury in mice with sickle cell disease.Recent trends in pulmonary arterial hypertension Endothelin receptor antagonism prevents hypoxia-induced mortality and morbidity in a mouse model of sickle-cell disease.Nitric oxide-dependent generation of reactive species in sickle cell disease. Actin tyrosine induces defective cytoskeletal polymerization.Proteomic identification of altered apolipoprotein patterns in pulmonary hypertension and vasculopathy of sickle cell disease Platelet bioenergetic screen in sickle cell patients reveals mitochondrial complex V inhibition, which contributes to platelet activation.Functional consequences of actin nitration: in vitro and in disease states.Oxidative modification of von Willebrand factor by neutrophil oxidants inhibits its cleavage by ADAMTS13.Heme degradation and vascular injury.Antisickling property of fetal hemoglobin enhances nitric oxide bioavailability and ameliorates organ oxidative stress in transgenic-knockout sickle mice.Cellular, pharmacological, and biophysical evaluation of explanted lungs from a patient with sickle cell disease and severe pulmonary arterial hypertension Reaping of nitric oxide by sickle cell disease.Genetic deletion of apolipoprotein A-I increases airway hyperresponsiveness, inflammation, and collagen deposition in the lung.Urinary albumin excretion is associated with pulmonary hypertension in sickle cell disease: potential role of soluble fms-like tyrosine kinase-1.Hydroxyurea-induced expression of glutathione peroxidase 1 in red blood cells of individuals with sickle cell anemia Superoxide dismutase mimics: chemistry, pharmacology, and therapeutic potential Endogenous nitric oxide synthase inhibitors in sickle cell disease: abnormal levels and correlations with pulmonary hypertension, desaturation, haemolysis, organ dysfunction and death Effect of fetal hemoglobin on microvascular regulation in sickle transgenic-knockout mice.Dysregulated arginine metabolism, hemolysis-associated pulmonary hypertension, and mortality in sickle cell disease Rational design of superoxide dismutase (SOD) mimics: the evaluation of the therapeutic potential of new cationic Mn porphyrins with linear and cyclic substituents.Protective effect of arginine on oxidative stress in transgenic sickle mouse models.Sickle cell disease increases high mobility group box 1: a novel mechanism of inflammation.Arginine therapy of transgenic-knockout sickle mice improves microvascular function by reducing non-nitric oxide vasodilators, hemolysis, and oxidative stress.Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.Vasculopathy in sickle cell disease: Biology, pathophysiology, genetics, translational medicine, and new research directions Deconstructing endothelial dysfunction: soluble guanylyl cyclase oxidation and the NO resistance syndrome.Vasculopathy and pulmonary hypertension in sickle cell disease.Anion exchange HPLC isolation of high-density lipoprotein (HDL) and on-line estimation of proinflammatory HDL

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Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease.

http://www.wikidata.org/entity/Q33953052

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Proceedings of the National Academy of Sciences of the United States of America

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Oxygen radical inhibition of n ...... nction in sickle cell disease.

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A Tousson

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B A Freeman

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P2860

Crystal structures of bovine milk xanthine dehydrogenase and xanthine oxidase: structure-based mechanism of conversion

Proteolytic conversion of xanthine dehydrogenase from the NAD-dependent type to the O2-dependent type. Amino acid sequence of rat liver xanthine dehydrogenase and identification of the cleavage sites of the enzyme protein during irreversible convers

Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

Formation and properties of peroxynitrite as studied by laser flash photolysis, high-pressure stopped-flow technique, and pulse radiolysis.

Metalloporphyrin class of therapeutic catalytic antioxidants.

In vivo blood flow abnormalities in the transgenic knockout sickle cell mouse.

Reactive species in sickle cell disease.

Potentiated adherence of sickle erythrocytes to endothelium infected by virus.

Regulation of xanthine dehydrogenase and xanthine oxidase activity and gene expression in cultured rat pulmonary endothelial cells

Peroxynitrite induces both vasodilatation and impaired vascular relaxation in the isolated perfused rat heart

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10.1073/PNAS.221292098

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