Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML. - Wikidata - wikimedia - TriplyDB

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

http://www.wikidata.org/entity/Q33703030

about

c-Jun N-terminal Kinase (JNK) Signaling as a Therapeutic Target for Alzheimer's Disease Aberrant nuclear factor-kappa B activity in acute myeloid leukemia: from molecular pathogenesis to therapeutic target Protective effects of astaxanthin on ConA-induced autoimmune hepatitis by the JNK/p-JNK pathway-mediated inhibition of autophagy and apoptosis.DUSP4 deficiency caused by promoter hypermethylation drives JNK signaling and tumor cell survival in diffuse large B cell lymphoma Mesenchymal stromal cells derived from acute myeloid leukemia bone marrow exhibit aberrant cytogenetics and cytokine elaboration.Inflamed macrophage microvesicles induce insulin resistance in human adipocytes.A Simple and Efficient Method for Preparing Cell Slides and Staining without Using Cytocentrifuge and Cytoclips.The Interleukin-2-mTORc1 Kinase Axis Defines the Signaling, Differentiation, and Metabolism of T Helper 1 and Follicular B Helper T Cells.Lineage-specific and single-cell chromatin accessibility charts human hematopoiesis and leukemia evolution.Sensitizing leukemia stem cells to NF-κB inhibitor treatment in vivo by inactivation of both TNF and IL-1 signaling.Targeting novel signaling pathways for resistant acute myeloid leukemia.Biology and Clinical Relevance of Acute Myeloid Leukemia Stem Cells.Toll-like receptor signaling in hematopoietic homeostasis and the pathogenesis of hematologic diseases.Therapeutic targeting of necroptosis by Smac mimetic bypasses apoptosis resistance in acute myeloid leukemia cells.Mechanism by which nuclear factor-kappa beta (NF-kB) regulates ovine fetal pulmonary vascular smooth muscle cell proliferation.Lack of Oestrogenic Inhibition of the Nuclear Factor-κB Pathway in Somatolactotroph Tumour Cells.Mass cytometry analysis reveals hyperactive NF Kappa B signaling in myelofibrosis and secondary acute myeloid leukemia.Sensitizing acute myeloid leukemia cells to induced differentiation by inhibiting the RIP1/RIP3 pathway.Fanconi Anemia Mesenchymal Stromal Cells-Derived Glycerophospholipids Skew Hematopoietic Stem Cell Differentiation Through Toll-Like Receptor Signaling.Validation of a novel rinse and filtration method for efficient processing of fresh produce samples for microbiological indicator enumeration.Identification of PGAM5 as a Mammalian Protein Histidine Phosphatase that Plays a Central Role to Negatively Regulate CD4(+) T Cells.Inflammatory Signaling Pathways in Preleukemic and Leukemic Stem Cells.A machine learning approach to integrate big data for precision medicine in acute myeloid leukemia.JNK1 as a signaling node in VDR-BRAF induction of cell death in AML.Therapeutic Targeting of MLL Degradation Pathways in MLL-Rearranged Leukemia.Identification of DNA methylation prognostic signature of acute myelocytic leukemia.

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P2860

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

http://www.wikidata.org/entity/Q33703030

description

2014 nî lūn-bûn

@nan

2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@ast

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@en

type

label

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@ast

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@en

prefLabel

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@ast

Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@en

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Journal of Experimental Medicine

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Co-inhibition of NF-κB and JNK is synergistic in TNF-expressing human AML.

@en

P2093

Andrew Volk

http://www.w3.org/2001/XMLSchema#string

Dewen You

http://www.w3.org/2001/XMLSchema#string

Jianjun Chen

http://www.w3.org/2001/XMLSchema#string

Jianke Zhang

http://www.w3.org/2001/XMLSchema#string

Jing Li

http://www.w3.org/2001/XMLSchema#string

Jiwang Zhang

http://www.w3.org/2001/XMLSchema#string

Jun Zhang

http://www.w3.org/2001/XMLSchema#string

Junping Xin

http://www.w3.org/2001/XMLSchema#string

Paul C Kuo

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Peter Breslin

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P2860

Tumor necrosis factor-alpha-induced IKK phosphorylation of NF-kappaB p65 on serine 536 is mediated through the TRAF2, TRAF5, and TAK1 signaling pathway

cIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms

TNF-alpha induces two distinct caspase-8 activation pathways

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

The functional contrariety of JNK

Cancer-related inflammation

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

Gadd45 beta mediates the NF-kappa B suppression of JNK signalling by targeting MKK7/JNKK2

JNK-induced apoptosis, compensatory growth, and cancer stem cells

Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis

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1093-1108

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10.1084/JEM.20130990

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24842373

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