Influenza virus-induced glucocorticoids compromise innate host defense against a secondary bacterial infection.
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Bacterial-viral load and the immune response in stable and exacerbated COPD: significance and therapeutic prospectsBiocompatible materials for continuous glucose monitoring devicesInnocent until proven guilty: mechanisms and roles of Streptococcus-Candida interactions in oral health and diseaseRegulating the adaptive immune response to respiratory virus infectionLimited brain metabolism changes differentiate between the progression and clearance of rabies virusRhinovirus attenuates non-typeable Hemophilus influenzae-stimulated IL-8 responses via TLR2-dependent degradation of IRAK-1Effects of Interferons and Viruses on MetabolismPotential role of HPA axis and sympathetic nervous responses in depletion of B cells induced by H9N2 avian influenza virus infectionButyrophilin-like 1 encodes an enterocyte protein that selectively regulates functional interactions with T lymphocytesTime and dose-dependent risk of pneumococcal pneumonia following influenza: a model for within-host interaction between influenza and Streptococcus pneumoniaeProtecting against post-influenza bacterial pneumonia by increasing phagocyte recruitment and ROS production.Difference in immune response in vaccinated and unvaccinated Swedish individuals after the 2009 influenza pandemicUnusual dengue virus 3 epidemic in Nicaragua, 2009Kinetics of coinfection with influenza A virus and Streptococcus pneumoniae.Clinical Characteristics of Influenza-Associated Pneumonia of Adults: Clinical Features and Factors Contributing to Severity and Mortality.Microneedle patch delivery of influenza vaccine during pregnancy enhances maternal immune responses promoting survival and long-lasting passive immunity to offspring.Influenza virus infection induces the nuclear relocalization of the Hsp90 co-chaperone p23 and inhibits the glucocorticoid receptor response.Dynamic modulation of thymic microRNAs in response to stress.Influenza A virus infection of human primary dendritic cells impairs their ability to cross-present antigen to CD8 T cells.Microbial-immune cross-talk and regulation of the immune system.A single E627K mutation in the PB2 protein of H9N2 avian influenza virus increases virulence by inducing higher glucocorticoids (GCs) level.Transcriptional specialization of human dendritic cell subsets in response to microbial vaccines.Budesonide suppresses pulmonary antibacterial host defense by down-regulating cathelicidin-related antimicrobial peptide in allergic inflammation mice and in lung epithelial cells.Transcriptional adaptation of pneumococci and human pharyngeal cells in the presence of a virus infection.Tissue expression of steroid hormone receptors is associated with differential immune responsiveness.Influenza virus infection is associated with increased risk of death amongst patients hospitalized with confirmed pulmonary tuberculosis in South Africa, 2010-2011.Viral infection augments Nod1/2 signaling to potentiate lethality associated with secondary bacterial infections.Systemic responses during local viral infections: type I IFNs sound the alarm.Nitric oxide increases susceptibility of Toll-like receptor-activated macrophages to spreading Listeria monocytogenesExposure to common respiratory bacteria alters the airway epithelial response to subsequent viral infection.Bacterial colonization dampens influenza-mediated acute lung injury via induction of M2 alveolar macrophages.CD4+ T cells are trigger and target of the glucocorticoid response that prevents lethal immunopathology in toxoplasma infectionSome unmet challenges in the immunology of viral infections.The two faces of heterologous immunity: protection or immunopathology.Streptococcal co-infection augments Candida pathogenicity by amplifying the mucosal inflammatory response.Chronic Toxoplasma gondii Infection Exacerbates Secondary Polymicrobial Sepsis.Respective roles of TLR, RIG-I and NLRP3 in influenza virus infection and immunity: impact on vaccine design.The complication of coinfection.Interactions between Streptococcus pneumoniae and influenza virus: a mutually beneficial relationship?The impact of infection and tissue damage in solid-organ transplantation.
P2860
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P2860
Influenza virus-induced glucocorticoids compromise innate host defense against a secondary bacterial infection.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
Influenza virus-induced glucoc ...... secondary bacterial infection.
@ast
Influenza virus-induced glucoc ...... secondary bacterial infection.
@en
type
label
Influenza virus-induced glucoc ...... secondary bacterial infection.
@ast
Influenza virus-induced glucoc ...... secondary bacterial infection.
@en
prefLabel
Influenza virus-induced glucoc ...... secondary bacterial infection.
@ast
Influenza virus-induced glucoc ...... secondary bacterial infection.
@en
P2860
P1433
P1476
Influenza virus-induced glucoc ...... secondary bacterial infection.
@en
P2093
Charles H Annicelli
P2860
P304
P356
10.1016/J.CHOM.2010.01.010
P577
2010-02-01T00:00:00Z