about
Missing self-recognition of Ocil/Clr-b by inhibitory NKR-P1 natural killer cell receptorsConditional Stat1 ablation reveals the importance of interferon signaling for immunity to Listeria monocytogenes infectionSelective associations with signaling proteins determine stimulatory versus costimulatory activity of NKG2DThe E3 ligase Cbl-b and TAM receptors regulate cancer metastasis via natural killer cells.Rapid pathogen-induced apoptosis: a mechanism used by dendritic cells to limit intracellular replication of Legionella pneumophila.Influenza virus-induced glucocorticoids compromise innate host defense against a secondary bacterial infection.Implications of CD94 deficiency and monoallelic NKG2A expression for natural killer cell development and repertoire formationThe role of the NKG2D immunoreceptor in immune cell activation and natural killing.Both TLR2 and TRIF contribute to interferon-β production during Listeria infectionInfluence of the microbiome on response to vaccination.A subset of natural killer cells achieves self-tolerance without expressing inhibitory receptors specific for self-MHC molecules.Bacterial Manipulation of NK Cell Regulatory Activity Increases Susceptibility to Listeria monocytogenes Infection.The Influence of Programmed Cell Death in Myeloid Cells on Host Resilience to Infection with Legionella pneumophila or Streptococcus pyogenes.The role of NKG2D signaling in inhibition of cytotoxic T-lymphocyte lysis by the Murine cytomegalovirus immunoevasin m152/gp40Expression of natural killer receptor alleles at different Ly49 loci occurs independently and is regulated by major histocompatibility complex class I molecules.Innate immune surveillance of spontaneous B cell lymphomas by natural killer cells and gammadelta T cells.CDK8-mediated STAT1-S727 phosphorylation restrains NK cell cytotoxicity and tumor surveillanceInnate immune recognition triggers secretion of lysosomal enzymes by macrophages.The combined actions of NK and T lymphocytes are necessary to reject an EGFP+ mesenchymal tumor through mechanisms dependent on NKG2D and IFN gamma.Type I Interferon Signaling Prevents IL-1β-Driven Lethal Systemic Hyperinflammation during Invasive Bacterial Infection of Soft Tissue.Regulation of NO synthesis, local inflammation, and innate immunity to pathogens by BET family proteins.Contribution of a TANK-binding kinase 1-interferon (IFN) regulatory factor 7 pathway to IFN-γ-induced gene expression.Role of tissue protection in lethal respiratory viral-bacterial coinfection.GILT is a critical host factor for Listeria monocytogenes infection.Viral and bacterial infections induce expression of multiple NK cell receptors in responding CD8(+) T cells.Surviving Deadly Lung Infections: Innate Host Tolerance Mechanisms in the Pulmonary System.Host resilience to emerging coronavirusesCoinfection With Influenza A Virus and : An Underrecognized Impact on Host Resistance and Tolerance to Pulmonary InfectionsPulmonary influenza A virus infection leads to suppression of the innate immune response to dermal injury
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P50
description
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հետազոտող
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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type
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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prefLabel
Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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Amanda M. Jamieson
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P106
P108
P1153
7101999801
P21
P31
P496
0000-0002-5781-9231