Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth. - Wikidata - wikimedia - TriplyDB

Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth.

Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth.

http://www.wikidata.org/entity/Q33719761

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Therapeutic Value of Voltage-Gated Sodium Channel Inhibitors in Breast, Colorectal, and Prostate Cancer: A Systematic Review On the multiple roles of the voltage gated sodium channel β1 subunit in genetic diseases Sodium channel β subunits: emerging targets in channelopathies De novo gain-of-function and loss-of-function mutations of SCN8A in patients with intellectual disabilities and epilepsy Sodium channel-inhibiting drugs and cancer survival: protocol for a cohort study using the CPRD primary care database Exposure to sodium channel-inhibiting drugs and cancer survival: protocol for a cohort study using the QResearch primary care database Axon initial segment dysfunction in a mouse model of genetic epilepsy with febrile seizures plus.The sorting receptor Rer1 controls Purkinje cell function via voltage gated sodium channels.Channelrhodopsin-2 localised to the axon initial segment.Axon physiology.The sodium channel β1 subunit mediates outgrowth of neurite-like processes on breast cancer cells and promotes tumour growth and metastasis.Sodium channel SCN8A (Nav1.6): properties and de novo mutations in epileptic encephalopathy and intellectual disability.Composition, assembly, and maintenance of excitable membrane domains in myelinated axons.Independent and joint modulation of rat Nav1.6 voltage-gated sodium channels by coexpression with the auxiliary β1 and β2 subunits The sodium channel-blocking antiepileptic drug phenytoin inhibits breast tumour growth and metastasis.Na Channel β Subunits: Overachievers of the Ion Channel Family.Preferential targeting of Nav1.6 voltage-gated Na+ Channels to the axon initial segment during development Voltage-gated Na+ channel β1B: a secreted cell adhesion molecule involved in human epilepsy Regulation of Nav1.6 and Nav1.8 peripheral nerve Na+ channels by auxiliary β-subunits.The sodium channel accessory subunit Navβ1 regulates neuronal excitability through modulation of repolarizing voltage-gated K⁺ channels.Therapeutic potential for phenytoin: targeting Na(v)1.5 sodium channels to reduce migration and invasion in metastatic breast cancer.Normal sulfation levels regulate spinal cord neural precursor cell proliferation and differentiation Sodium channel-inhibiting drugs and survival of breast, colon and prostate cancer: a population-based study Voltage-gated sodium channels and metastatic disease.Blockade of voltage-gated sodium channels inhibits invasion of endocrine-resistant breast cancer cells Nav1.5 regulates breast tumor growth and metastatic dissemination in vivo Abnormal neuronal patterning occurs during early postnatal brain development of Scn1b-null mice and precedes hyperexcitability.β-Secretase: its biology as a therapeutic target in diseases β1-C121W Is Down But Not Out: Epilepsy-Associated Scn1b-C121W Results in a Deleterious Gain-of-Function.Sodium channel diversity in the vestibular ganglion: NaV1.5, NaV1.8, and tetrodotoxin-sensitive currents.Sodium channel gene family: epilepsy mutations, gene interactions and modifier effects.Axon initial segment dysfunction in epilepsy.Building and maintaining the axon initial segment.Electrophysiology and beyond: multiple roles of Na+ channel β subunits in development and disease.Signaling complexes of voltage-gated sodium and calcium channels The axon initial segment in nervous system disease and injury Neurological perspectives on voltage-gated sodium channels.Roles of ion transport in control of cell motility.Improved Methods for Fluorescence Microscopy Detection of Macromolecules at the Axon Initial Segment.Dual roles of voltage-gated sodium channels in development and cancer.

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P2860

Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth.

http://www.wikidata.org/entity/Q33719761

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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Functional reciprocity between ...... ability and neurite outgrowth.

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Proceedings of the National Academy of Sciences of the United States of America

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Barbara Ranscht

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Chunling Chen

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Fumitaka Oyama

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Haruko Miyazaki

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Jeffrey D Calhoun

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Nobuyuki Nukina

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William J Brackenbury

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P2860

Sodium channel Scn1b null mice exhibit prolonged QT and RR intervals

Sodium channel β1 subunit mutations associated with Brugada syndrome and cardiac conduction disease in humans

Mutations in sodium channel β1- and β2-subunits associated with atrial fibrillation

AnkyrinG is required for clustering of voltage-gated Na channels at axon initial segments and for normal action potential firing

Neurofascin assembles a specialized extracellular matrix at the axon initial segment

Ankyrin-G coordinates assembly of the spectrin-based membrane skeleton, voltage-gated sodium channels, and L1 CAMs at Purkinje neuron initial segments

Ataxia and abnormal cerebellar microorganization in mice with ablated contactin gene expression

Nomenclature of voltage-gated sodium channels

From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

A deletion in SCN1B is associated with febrile seizures and early-onset absence epilepsy

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2283-2288

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10.1073/PNAS.0909434107

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107

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20133873

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