Senescence induced by RECQL4 dysfunction contributes to Rothmund-Thomson syndrome features in mice.
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Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cellsThe DNA helicase recql4 is required for normal osteoblast expansion and osteosarcoma formationSporadic premature aging in a Japanese monkey: a primate model for progeriaRECQL4 Promotes DNA End Resection in Repair of DNA Double-Strand Breaks.Camptothecin targets WRN protein: mechanism and relevance in clinical breast cancer.The structural and functional characterization of human RecQ4 reveals insights into its helicase mechanism.Recent Advances in Understanding Werner Syndrome.Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and ubiquitination in DNA double-strand break repair.RECQL4 Regulates p53 Function In Vivo During Skeletogenesis.Ribosomal Protein S3 Negatively Regulates Unwinding Activity of RecQ-like Helicase 4 through Their Physical Interaction.
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P2860
Senescence induced by RECQL4 dysfunction contributes to Rothmund-Thomson syndrome features in mice.
description
2014 nî lūn-bûn
@nan
2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@ast
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@en
type
label
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@ast
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@en
prefLabel
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@ast
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@en
P2093
P2860
P356
P1476
Senescence induced by RECQL4 d ...... son syndrome features in mice.
@en
P2093
D L Croteau
K G Becker
T Kulikowicz
P2860
P2888
P356
10.1038/CDDIS.2014.168
P577
2014-05-15T00:00:00Z
P5875
P6179
1006509332