Aconitase and ATP synthase are targets of malondialdehyde modification and undergo an age-related decrease in activity in mouse heart mitochondria.
about
Antioxidant responses and cellular adjustments to oxidative stressOxidative stress and the homeodynamics of iron metabolismProtein carbonylation, cellular dysfunction, and disease progressionMitochondria as a source and target of lipid peroxidation products in healthy and diseased heartEffects of age and calorie restriction on tryptophan nitration, protein content, and activity of succinyl-CoA:3-ketoacid CoA transferase in rat kidney mitochondriaRedox proteomics in selected neurodegenerative disorders: from its infancy to future applications.Mechanisms of acrolein-induced myocardial dysfunction: implications for environmental and endogenous aldehyde exposure.Age-related alterations in oxidatively damaged proteins of mouse skeletal muscle mitochondrial electron transport chain complexes.Detection and characterization of peroxynitrite-induced modifications of tyrosine, tryptophan, and methionine residues by tandem mass spectrometryRedox regulation of antioxidants, autophagy, and the response to stress: implications for electrophile therapeutics.Effect of coenzyme Q10 intake on endogenous coenzyme Q content, mitochondrial electron transport chain, antioxidative defenses, and life span of mice.Aconitase is the main functional target of aging in the citric acid cycle of kidney mitochondria from mice.Mitochondria and ageing: role in heart, skeletal muscle and adipose tissue.Posttranslational modification of differentially expressed mitochondrial proteins in the retina during early experimental autoimmune uveitis.Protein kinase C-α interaction with F0F1-ATPase promotes F0F1-ATPase activity and reduces energy deficits in injured renal cells.Mitochondrial electron transport chain functions in long-lived Ames dwarf mice.Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.The redox stress hypothesis of agingEffects of age and caloric restriction on mitochondrial protein oxidative damage in mice.ATP Depletion Via Mitochondrial F1F0 Complex by Lethal Factor is an Early Event in B. Anthracis-Induced Sudden Cell Death.Cell signalling by reactive lipid species: new concepts and molecular mechanisms.Skeletal muscle mitochondria and aging: a review.Carbonylation of mitochondrial proteins in Drosophila melanogaster during agingAge-related increases in oxidatively damaged proteins of mouse kidney mitochondrial electron transport chain complexesDecreased enzyme activities of chaperones PDI and BiP in aged mouse livers.Age-related alterations in oxidatively damaged proteins of mouse heart mitochondrial electron transport chain complexes.Nitration of tryptophan 372 in succinyl-CoA:3-ketoacid CoA transferase during aging in rat heart mitochondria.Mitochondrial decay in the brains of old rats: ameliorating effect of alpha-lipoic acid and acetyl-L-carnitine.The role of mitochondria in brain aging and the effects of melatonin.4-Hydroxy-2-nonenal, a reactive product of lipid peroxidation, and neurodegenerative diseases: a toxic combination illuminated by redox proteomics studiesAconitase post-translational modification as a key in linkage between Krebs cycle, iron homeostasis, redox signaling, and metabolism of reactive oxygen species.Oxidation as an important factor of protein damage: Implications for Maillard reaction.Malondialdehyde Epitopes as Targets of Immunity and the Implications for Atherosclerosis.Proteomic study on the protective mechanism of fibroblast growth factor 21 to ischemia-reperfusion injury.Mitochondrial-Targeted Antioxidant Maintains Blood Flow, Mitochondrial Function, and Redox Balance in Old Mice Following Prolonged Limb Ischemia.Changes in mitochondrial energy utilization in young and old worker honeybees (Apis mellifera).Loss of parietal cell superoxide dismutase leads to gastric oxidative stress and increased injury susceptibility in mice.Protein thiol oxidation does not change in skeletal muscles of aging female mice.Age-associated oxidative modifications of mitochondrial α-subunit of F1 ATP synthase from mouse skeletal muscles.Effects of diabetes on oxidative and nitrosative stress in kidney mitochondria from aged rats.
P2860
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P2860
Aconitase and ATP synthase are targets of malondialdehyde modification and undergo an age-related decrease in activity in mouse heart mitochondria.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@ast
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@en
type
label
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@ast
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@en
prefLabel
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@ast
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@en
P2093
P2860
P1476
Aconitase and ATP synthase are ...... y in mouse heart mitochondria.
@en
P2093
Connie S Yarian
Igor Rebrin
Rajindar S Sohal
P2860
P304
P356
10.1016/J.BBRC.2005.02.135
P407
P577
2005-04-01T00:00:00Z