Reversal of gene expression changes in the colorectal normal-adenoma pathway by NS398 selective COX2 inhibitor. - Wikidata - wikimedia - TriplyDB

Reversal of gene expression changes in the colorectal normal-adenoma pathway by NS398 selective COX2 inhibitor.

Reversal of gene expression changes in the colorectal normal-adenoma pathway by NS398 selective COX2 inhibitor.

http://www.wikidata.org/entity/Q33723633

about

Differential expression of miRNAs in colorectal cancer: comparison of paired tumor tissue and adjacent normal mucosa using high-throughput sequencing Claudins in intestines: Distribution and functional significance in health and diseases Discovery and validation of molecular biomarkers for colorectal adenomas and cancer with application to blood testing Asymmetric microarray data produces gene lists highly predictive of research literature on multiple cancer types.Modeling oncogenic signaling in colon tumors by multidirectional analyses of microarray data directed for maximization of analytical reliability Identification of SOCS2 and SOCS6 as biomarkers in human colorectal cancer.Transporter function and cyclic AMP turnover in normal colonic mucosa from patients with and without colorectal neoplasia.Genome-wide screening of genes regulated by DNA methylation in colon cancer development.Gene expression anti-profiles as a basis for accurate universal cancer signatures.Dysplasia-carcinoma transition specific transcripts in colonic biopsy samples.Mitosis is a source of potential markers for screening and survival and therapeutic targets in cervical cancer.Sporadic colorectal cancer development shows rejuvenescence regarding epithelial proliferation and apoptosis Promoter hypermethylation-related reduced somatostatin production promotes uncontrolled cell proliferation in colorectal cancer.Repression of KIAA1199 attenuates Wnt-signalling and decreases the proliferation of colon cancer cells.Discovery of a 29-gene panel in peripheral blood mononuclear cells for the detection of colorectal cancer and adenomas using high throughput real-time PCR.Pleiotropic functions of EAPII/TTRAP/TDP2: cancer development, chemoresistance and beyond Combined evaluation of hexokinase 2 and phosphorylated pyruvate dehydrogenase-E1α in invasive front lesions of colorectal tumors predicts cancer metabolism and patient prognosis.Comprehensive DNA Methylation Analysis Reveals a Common Ten-Gene Methylation Signature in Colorectal Adenomas and Carcinomas.Proteomic signatures of the desmoplastic invasion front reveal collagen type XII as a marker of myofibroblastic differentiation during colorectal cancer metastasis.MicroRNA Expression Profile Reveals miR-17-92 and miR-143-145 Cluster in Synchronous Colorectal Cancer Cancer develops, progresses and responds to therapies through restricted perturbation of the protein-protein interaction network.Induction of KIAA1199/CEMIP is associated with colon cancer phenotype and poor patient survival.Immunoexpression of the COX-2, p53, and caspase-3 proteins in colorectal adenoma and non-neoplastic mucosa.An integrated approach to identify causal network modules of complex diseases with application to colorectal cancer Gene expression patterns unveil a new level of molecular heterogeneity in colorectal cancer.Identification of KIAA1199 as a Biomarker for Pancreatic Intraepithelial Neoplasia Estradiol and mTORC2 cooperate to enhance prostaglandin biosynthesis and tumorigenesis in TSC2-deficient LAM cells.Colorectal adenoma and carcinoma specific miRNA profiles in biopsy and their expression in plasma specimens.Plakophilins in desmosomal adhesion and signaling.Reproducibility of parameter learning with missing observations in naive Wnt Bayesian network trained on colorectal cancer samples and doxycycline-treated cell lines.Aging related methylation influences the gene expression of key control genes in colorectal cancer and adenoma.Knockdown of CSE1L Gene in Colorectal Cancer Reduces Tumorigenesis in Vitro.Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease.Increased methylation variation in epigenetic domains across cancer types.Functional and protein‑protein interaction network analysis of colorectal cancer induced by ulcerative colitis.'Neuroinflammation' differs categorically from inflammation: transcriptomes of Alzheimer's disease, Parkinson's disease, schizophrenia and inflammatory diseases compared.Loss of Myosin Vb in colorectal cancer is a strong prognostic factor for disease recurrence.The therapeutic effect of miR-125b is enhanced by the prostaglandin endoperoxide synthase 2/cyclooxygenase 2 blockade and hampers ETS1 in the context of the microenvironment of bone metastasis.Identification of Unique miRNA Biomarkers in Colorectal Adenoma and Carcinoma Using Microarray: Evaluation of Their Putative Role in Disease Progression

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Reversal of gene expression changes in the colorectal normal-adenoma pathway by NS398 selective COX2 inhibitor.

http://www.wikidata.org/entity/Q33723633

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Reversal of gene expression ch ...... S398 selective COX2 inhibitor.

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Reversal of gene expression ch ...... S398 selective COX2 inhibitor.

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Reversal of gene expression ch ...... S398 selective COX2 inhibitor.

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Reversal of gene expression ch ...... S398 selective COX2 inhibitor.

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Reversal of gene expression ch ...... S398 selective COX2 inhibitor.

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Reversal of gene expression ch ...... S398 selective COX2 inhibitor.

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Reversal of gene expression ch ...... NS398 selective COX2 inhibitor

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P2860

Arrest of G(1)-S progression by the p53-inducible gene PC3 is Rb dependent and relies on the inhibition of cyclin D1 transcription

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Cyclooxygenase-2 selective inhibition with NS-398 suppresses proliferation and invasiveness and delays liver metastasis in colorectal cancer

COX-2 inhibition and prevention of cancer

Aspirin and NS-398 inhibit hepatocyte growth factor-induced invasiveness of human hepatoma cells

Cyclooxygenase-2 (PTGS2) inhibitors augment the rate of hexose transport in L6 myotubes in an insulin- and AMPKalpha-independent manner

Celecoxib for the prevention of sporadic colorectal adenomas

Celecoxib for the prevention of colorectal adenomatous polyps

Coxibs and cancer prevention

Non-steroidal anti-inflammatory drugs with activity against either cyclooxygenase 1 or cyclooxygenase 2 inhibit colorectal cancer in a DMH rodent model by inducing apoptosis and inhibiting cell proliferation

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765-773

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10.1038/SJ.BJC.6605515

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41089433

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1039654889

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20087348

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