Regions of the varicella-zoster virus open reading frame 63 latency-associated protein important for replication in vitro are also critical for efficient establishment of latency.
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Herpes simplex virus and varicella zoster virus, the house guests who never leaveVaricella-zoster virus T cell tropism and the pathogenesis of skin infectionIdentification of phosphorylated residues on varicella-zoster virus immediate-early protein ORF63.Mutational analysis of the varicella-zoster virus ORF62/63 intergenic regionDownregulation of varicella-zoster virus (VZV) immediate-early ORF62 transcription by VZV ORF63 correlates with virus replication in vitro and with latency.Varicella zoster virus latency.Simian varicella virus open reading frame 63/70 expression is required for efficient virus replication in cultureA sequence within the varicella-zoster virus (VZV) OriS is a negative regulator of DNA replication and is bound by a protein complex containing the VZV ORF29 proteinVaricella-Zoster virus IE63, a major viral latency protein, is required to inhibit the alpha interferon-induced antiviral responseA varicella-zoster virus mutant impaired for latency in rodents, but not impaired for replication in cell cultureRodent models of varicella-zoster virus neurotropism.Varicella-zoster virus immediate-early 63 protein interacts with human antisilencing function 1 protein and alters its ability to bind histones h3.1 and h3.3.Phosphorylation of the nuclear form of varicella-zoster virus immediate-early protein 63 by casein kinase II at serine 186.Interaction of allergy history and antibodies to specific varicella-zoster virus proteins on glioma risk.Differentiated neuroblastoma cells provide a highly efficient model for studies of productive varicella-zoster virus infection of neuronal cellsNuclear import of the varicella-zoster virus latency-associated protein ORF63 in primary neurons requires expression of the lytic protein ORF61 and occurs in a proteasome-dependent manner.Varicella-zoster virus IE63 protein phosphorylation by roscovitine-sensitive cyclin-dependent kinases modulates its cellular localization and activity.Varicella-zoster virus open reading frame 66 protein kinase is required for efficient viral growth in primary human corneal stromal fibroblast cells.Varicella-zoster virus ORF63 inhibits apoptosis of primary human neurons.Transcriptional regulation of the Herpes Simplex Virus 1alpha-gene by the viral immediate-early protein ICP22 in association with VP16.Characterization of synonymous codon usage bias in the pseudorabies virus US1 gene.VZV ORF63 protects human neuronal and keratinocyte cell lines from apoptosis and changes its localization upon apoptosis induction.
P2860
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P2860
Regions of the varicella-zoster virus open reading frame 63 latency-associated protein important for replication in vitro are also critical for efficient establishment of latency.
description
2005 nî lūn-bûn
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2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
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2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Regions of the varicella-zoste ...... ient establishment of latency.
@ast
Regions of the varicella-zoste ...... ient establishment of latency.
@en
type
label
Regions of the varicella-zoste ...... ient establishment of latency.
@ast
Regions of the varicella-zoste ...... ient establishment of latency.
@en
prefLabel
Regions of the varicella-zoste ...... ient establishment of latency.
@ast
Regions of the varicella-zoste ...... ient establishment of latency.
@en
P2093
P2860
P1433
P1476
Regions of the varicella-zoste ...... ient establishment of latency.
@en
P2093
Catherine Sadzot-Delvaux
Jeffrey I Cohen
Lesley Pesnicak
Sebastien Bontems
Tammy Krogmann
P2860
P304
P356
10.1128/JVI.79.8.5069-5077.2005
P577
2005-04-01T00:00:00Z