The many faces of HMGB1: molecular structure-functional activity in inflammation, apoptosis, and chemotaxis.
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Sepsis and disseminated intravascular coagulationEmerging role of HMGB1 in fibrotic diseasesThe role of HMGB1 in the pathogenesis of inflammatory and autoimmune diseasesStress sounds the alarmin: The role of the danger-associated molecular pattern HMGB1 in stress-induced neuroinflammatory primingMénage à Trois in stress: DAMPs, redox and autophagyStress-induced neuroinflammatory priming: A liability factor in the etiology of psychiatric disordersThe HMGB1-RAGE Inflammatory Pathway: Implications for Brain Injury-Induced Pulmonary DysfunctionThe dendritic cell response to classic, emerging, and homeostatic danger signals. Implications for autoimmunityThioredoxin Interacting Protein (TXNIP) and Pathogenesis of Diabetic RetinopathyThe PYRIN Domain-only Protein POP1 Inhibits Inflammasome Assembly and Ameliorates Inflammatory DiseaseThe spleen contributes importantly to myocardial infarct exacerbation during post-ischemic reperfusion in mice via signaling between cardiac HMGB1 and splenic RAGE.Two high-mobility group box domains act together to underwind and kink DNA.Molecular isoforms of high-mobility group box 1 are mechanistic biomarkers for epilepsyDown-Regulation of Serum High-Mobility Group Box 1 Protein in Patients with Pulmonary Tuberculosis and Nontuberculous Mycobacterial Lung Disease.Ethanol, TLR3, and TLR4 Agonists Have Unique Innate Immune Responses in Neuron-Like SH-SY5Y and Microglia-Like BV2Disruption of the Cx43/miR21 pathway leads to osteocyte apoptosis and increased osteoclastogenesis with aging.Emerging role of HMGB1 in lung diseases: friend or foe.The HMGB1-RAGE axis mediates traumatic brain injury-induced pulmonary dysfunction in lung transplantation.NAD+ augmentation ameliorates acute pancreatitis through regulation of inflammasome signalling.Expression of High-Mobility Group Box 1 Protein (HMGB1) and Toll-Like Receptor 9 (TLR9) in Retinas of Diabetic RatsHMGN2, a new anti-tumor effector molecule of CD8⁺ T cells.Sequestering HMGB1 via DNA-conjugated beads ameliorates murine colitisHMGB1 in health and disease.The quantum of initial transformed cells potentially modulates the type of local inflammation mechanism elicited by surrounding normal epithelial tissues and systemic immune pattern for tumor arrest or progression.MD-2 is required for disulfide HMGB1-dependent TLR4 signaling.DAMP signaling is a key pathway inducing immune modulation after brain injury.The HMGB1/RAGE axis triggers neutrophil-mediated injury amplification following necrosis.Anti-HMGB1 monoclonal antibody ameliorates immunosuppression after peripheral tissue trauma: attenuated T-lymphocyte response and increased splenic CD11b (+) Gr-1 (+) myeloid-derived suppressor cells require HMGB1.Physiologic concentrations of HMGB1 have no impact on cytokine-mediated eosinophil survival or chemotaxis in response to Eotaxin-2 (CCL24).Toll-like receptor 4 (TLR4) antagonist eritoran tetrasodium attenuates liver ischemia and reperfusion injury through inhibition of high-mobility group box protein B1 (HMGB1) signaling.High systemic levels of the cytokine-inducing HMGB1 isoform secreted in severe macrophage activation syndrome.Innate immunity sensors participating in pathophysiology of joint diseases: a brief overview.The compromise of macrophage functions by hyperoxia is attenuated by ethacrynic acid via inhibition of NF-κB-mediated release of high-mobility group box-1.High mobility group box 1 contributes to anti-neutrophil cytoplasmic antibody-induced neutrophils activation through receptor for advanced glycation end products (RAGE) and Toll-like receptor 4.Intestinal Epithelial TLR-4 Activation Is Required for the Development of Acute Lung Injury after Trauma/Hemorrhagic Shock via the Release of HMGB1 from the GutExpression of the receptor for advanced glycation end products, a target for high mobility group box 1 protein, and its role in chronic recalcitrant rhinosinusitis with nasal polyps.FOXP3+ T Cells Recruited to Sites of Sterile Skeletal Muscle Injury Regulate the Fate of Satellite Cells and Guide Effective Tissue Regeneration.The Role of High Mobility Group Box 1 Protein (HMGB1) in the Immunopathology of Experimental Pulmonary Tuberculosis.HMGB1 promotes the activation of NLRP3 and caspase-8 inflammasomes via NF-κB pathway in acute glaucoma.HMGB1 Is Involved in IFN-α Production and TRAIL Expression by HIV-1-Exposed Plasmacytoid Dendritic Cells: Impact of the Crosstalk with NK Cells
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P2860
The many faces of HMGB1: molecular structure-functional activity in inflammation, apoptosis, and chemotaxis.
description
2013 nî lūn-bûn
@nan
2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@ast
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@en
type
label
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@ast
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@en
prefLabel
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@ast
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@en
P2860
P356
P1476
The many faces of HMGB1: molec ...... on, apoptosis, and chemotaxis.
@en
P2093
Daniel J Antoine
P2860
P304
P356
10.1189/JLB.1212662
P577
2013-02-27T00:00:00Z